Biologically active, high levels of interleukin-22 inhibit hepatic gluconeogenesis but do not affect obesity and its metabolic consequences.
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The 2015 Ming K Jeang Award for Excellence in Cell & Bioscience.Serum levels of interleukin-22, cardiometabolic risk factors and incident type 2 diabetes: KORA F4/FF4 studyThough Active on RINm5F Insulinoma Cells and Cultured Pancreatic Islets, Recombinant IL-22 Fails to Modulate Cytotoxicity and Disease in a Protocol of Streptozotocin-Induced Experimental DiabetesHuman IL-22 binding protein isoforms act as a rheostat for IL-22 signaling.Deciphering the role of interleukin-22 in metabolic alterationsThe role of IL-22 in the resolution of sterile and nonsterile inflammation.
P2860
Biologically active, high levels of interleukin-22 inhibit hepatic gluconeogenesis but do not affect obesity and its metabolic consequences.
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2015 nî lūn-bûn
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2015年の論文
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2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
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2015年論文
@zh-tw
2015年论文
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2015年论文
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2015年论文
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name
Biologically active, high leve ...... nd its metabolic consequences.
@ast
Biologically active, high leve ...... nd its metabolic consequences.
@en
type
label
Biologically active, high leve ...... nd its metabolic consequences.
@ast
Biologically active, high leve ...... nd its metabolic consequences.
@en
prefLabel
Biologically active, high leve ...... nd its metabolic consequences.
@ast
Biologically active, high leve ...... nd its metabolic consequences.
@en
P2093
P2860
P1433
P1476
Biologically active, high leve ...... nd its metabolic consequences.
@en
P2093
Dechun Feng
George Kunos
Joseph Tam
Mingjiang Xu
Sung Hwan Ki
P2860
P2888
P356
10.1186/S13578-015-0015-0
P577
2015-05-30T00:00:00Z
P5875
P6179
1006826050