Functional differences between microglia and monocytes after ischemic stroke.
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The Role of Monocytes in Ischemic Stroke Pathobiology: New Avenues to ExploreThe Indispensable Roles of Microglia and Astrocytes during Brain DevelopmentInflammation after Ischemic Stroke: The Role of Leukocytes and Glial CellsMacrophage-Derived Angiopoietin-Like Protein 2 Exacerbates Brain Damage by Accelerating Acute Inflammation after Ischemia-ReperfusionArterial Hypertension Aggravates Innate Immune Responses after Experimental Stroke.The Sur1-Trpm4 channel regulates NOS2 transcription in TLR4-activated microglia.Early retinal inflammatory biomarkers in the middle cerebral artery occlusion model of ischemic stroke.Ischemic stroke induces gut permeability and enhances bacterial translocation leading to sepsis in aged mice.Conditional ablation of myeloid TNF increases lesion volume after experimental stroke in mice, possibly via altered ERK1/2 signaling.Spatio-temporal activation of caspase-8 in myeloid cells upon ischemic stroke.Molecular and Cellular Responses to Interleukin-4 Treatment in a Rat Model of Transient Ischemia.Social Interaction Modulates the Neuroinflammatory Response to Global Cerebral Ischemia in Male Mice.Multiparity improves outcomes after cerebral ischemia in female mice despite features of increased metabovascular risk.Complex Roles of Microglial Cells in Ischemic Stroke Pathobiology: New Insights and Future DirectionsEmerging roles for RNA-binding proteins as effectors and regulators of cardiovascular disease.Diet-induced obesity prolongs neuroinflammation and recruits CCR2(+) monocytes to the brain following herpes simplex virus (HSV)-1 latency in mice.Role of microglia in ischemic focal stroke and recovery: focus on Toll-like receptors.Microglia and Monocyte-Derived Macrophages in StrokeEvidence that remodeling of insular cortex neurovascular unit contributes to hypertension-related sympathoexcitation.Integrated Stress Response as a Therapeutic Target for CNS Injuries.The human-specific CASP4 gene product contributes to Alzheimer-related synaptic and behavioural deficits.Interference with Protease-activated Receptor 1 Alleviates Neuronal Cell Death Induced by Lipopolysaccharide-Stimulated Microglial Cells through the PI3K/Akt Pathway.Microglia and macrophages in brain homeostasis and disease.Type I IFNs Are Required to Promote Central Nervous System Immune Surveillance through the Recruitment of Inflammatory Monocytes upon Systemic Inflammation.Deletion of the P2X4 receptor is neuroprotective acutely, but induces a depressive phenotype during recovery from ischemic stroke.The Copper bis(thiosemicarbazone) Complex CuII(atsm) Is Protective Against Cerebral Ischemia Through Modulation of the Inflammatory Milieu.CD8 signaling in microglia/macrophage M1 polarization in a rat model of cerebral ischemia.Human Umbilical Cord Mesenchymal Stem Cells Preserve Adult Newborn Neurons and Reduce Neurological Injury after Cerebral Ischemia by Reducing the Number of Hypertrophic Microglia/Macrophages.Distinguishing features of microglia- and monocyte-derived macrophages after stroke.Age-related differences in interferon regulatory factor-4 and -5 signaling in ischemic brains of mice.Regulatory T cells ameliorate intracerebral hemorrhage-induced inflammatory injury by modulating microglia/macrophage polarization through the IL-10/GSK3β/PTEN axis.Chronic Alterations in Systemic Immune Function After Traumatic Brain Injury.Enhancing endogenous capacity to repair a stroke-damaged brain: An evolving field for stroke research.Immune responses in stroke: how the immune system contributes to damage and healing after stroke and how this knowledge could be translated to better cures?Paradigm Shift to Neuroimmunomodulation for Translational Neuroprotection in Stroke.Experimental Stroke Differentially Affects Discrete Subpopulations of Splenic Macrophages.Neuroimmunology of Behavioral Comorbidities Associated With Cancer and Cancer Treatments.Neural stem cell therapy for subacute and chronic ischemic stroke.Aging alters the immunological response to ischemic stroke.Peroxiredoxin 2 activates microglia by interacting with Toll-like receptor 4 after subarachnoid hemorrhage.
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Functional differences between microglia and monocytes after ischemic stroke.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Functional differences between microglia and monocytes after ischemic stroke.
@ast
Functional differences between microglia and monocytes after ischemic stroke.
@en
type
label
Functional differences between microglia and monocytes after ischemic stroke.
@ast
Functional differences between microglia and monocytes after ischemic stroke.
@en
prefLabel
Functional differences between microglia and monocytes after ischemic stroke.
@ast
Functional differences between microglia and monocytes after ischemic stroke.
@en
P2093
P2860
P1476
Functional differences between microglia and monocytes after ischemic stroke.
@en
P2093
Anita R Patel
Evan R Jellison
Jeremy M Grenier
Joshua Crapser
Louise D McCullough
Rajkumar Verma
Rodney M Ritzel
P2860
P2888
P356
10.1186/S12974-015-0329-1
P577
2015-05-29T00:00:00Z
P5875
P6179
1053481131