Molecular analysis of microdissected tumors and preneoplastic intraductal lesions in pancreatic carcinoma - Wikidata - awgldk - TriplyDB

Molecular analysis of microdissected tumors and preneoplastic intraductal lesions in pancreatic carcinoma

Molecular analysis of microdissected tumors and preneoplastic intraductal lesions in pancreatic carcinoma

http://www.wikidata.org/entity/Q35745947

about

Targeted destruction of the orchestration of the pancreatic stroma and tumor cells in pancreatic cancer cases: molecular basis for therapeutic implications Challenges and pitfalls in HNPCC screening by microsatellite analysis and immunohistochemistry.Inherited predisposition to pancreatic cancer.Both p16(Ink4a) and the p19(Arf)-p53 pathway constrain progression of pancreatic adenocarcinoma in the mouse.Update on pancreatic intraepithelial neoplasia.Genetics and prevention of pancreatic cancer.Pancreatic cancer stromal biology and therapy.Two sides of the story? Smad4 loss in pancreatic cancer versus head-and-neck cancer The pathology of ductal-type pancreatic carcinomas and pancreatic intraepithelial neoplasia: insights for clinicians.Allelic loss is often the first hit in the biallelic inactivation of the p53 and DPC4 genes during pancreatic carcinogenesis Loss of heterozygosity or intragenic mutation, which comes first?Study human pancreatic cancer in mice: how close are they?Stromal biology of pancreatic cancer.Emerging molecular biology of pancreatic cancer.Beyond laser microdissection technology: follow the yellow brick road for cancer research.High prevalence of BRCA1 and BRCA2 germline mutations with loss of heterozygosity in a series of resected pancreatic adenocarcinoma and other neoplastic lesions The Mdm network and its regulation of p53 activities: a rheostat of cancer risk.Cells of origin of pancreatic neoplasms.Characterization of gene expression profiles in intraductal papillary-mucinous tumors of the pancreas.Frequency of K-ras mutations in pancreatic intraductal neoplasias associated with pancreatic ductal adenocarcinoma and chronic pancreatitis: a meta-analysis Distinguishing de novo second cancer formation from tumor recurrence: mutational fingerprinting by microdissection genotyping.Accumulation of beta-catenin protein, mutations in exon-3 of the beta-catenin gene and a loss of heterozygosity of 5q22 in solid pseudopapillary tumor of the pancreas.The intraductal carcinoma component is a significant prognostic parameter in patients with invasive ductal carcinoma of the pancreas.p53 and DPC4 alterations in the bile of patients with pancreatic carcinoma.Genetic Alterations of Periampullary and Pancreatic Ductal Adenocarcinoma: An Overview Molecular biology, models, and histopathology of chronic pancreatitis and pancreatic cancer Bcl-2 expression in pancreas development and pancreatic cancer progression

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P2860

Molecular analysis of microdissected tumors and preneoplastic intraductal lesions in pancreatic carcinoma

http://www.wikidata.org/entity/Q35745947

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2000 nî lūn-bûn

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Molecular analysis of microdis ...... esions in pancreatic carcinoma

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Molecular analysis of microdis ...... esions in pancreatic carcinoma

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The American Journal of Pathology

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Molecular analysis of microdis ...... esions in pancreatic carcinoma

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E Heinmöller

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F Hofstädter

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H Zirngibl

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W Dietmaier

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P2860

A genetic model for colorectal tumorigenesis

Frequent mutations of CDKN2 in primary pancreatic adenocarcinomas

A pancreatic cancer-specific expression profile

DPC4, a candidate tumor suppressor gene at human chromosome 18q21.1

Multiple mutation analyses in single tumor cells with improved whole genome amplification.

Precise microdissection of human prostate cancers reveals genotypic heterogeneity.

K-ras and p53 gene mutations in pancreatic cancer: ductal and nonductal tumors progress through different genetic lesions.

p53 mutations in pancreatic carcinoma and evidence of common involvement of homocopolymer tracts in DNA microdeletions.

Tumor-suppressive pathways in pancreatic carcinoma.

Poorly differentiated colonic adenocarcinoma, medullary type: clinical, phenotypic, and molecular characteristics.

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