CB1 Knockout Mice Unveil Sustained CB2-Mediated Antiallodynic Effects of the Mixed CB1/CB2 Agonist CP55,940 in a Mouse Model of Paclitaxel-Induced Neuropathic Pain.

CB1 Knockout Mice Unveil Sustained CB2-Mediated Antiallodynic Effects of the Mixed CB1/CB2 Agonist CP55,940 in a Mouse Model of Paclitaxel-Induced Neuropathic Pain.

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http://www.wikidata.org/entity/Q35745959
Inflammatory and Neuropathic Nociception is Preserved in GPR55 Knockout Mice.Neurotoxicity to DRG neurons varies between rodent strains treated with cisplatin and bortezomib.A Primary Cortical Input to Hippocampus Expresses a Pathway-Specific and Endocannabinoid-Dependent Form of Long-Term Potentiation.Compensatory Activation of Cannabinoid CB2 Receptor Inhibition of GABA Release in the Rostral Ventromedial Medulla in Inflammatory Pain.The Endogenous Cannabinoid System: A Budding Source of Targets for Treating Inflammatory and Neuropathic Pain.Positive Allosteric Modulation of Cannabinoid Receptor Type 1 Suppresses Pathological Pain Without Producing Tolerance or Dependence.Quantitative Analyses of Synergistic Responses between Cannabidiol and DNA-Damaging Agents on the Proliferation and Viability of Glioblastoma and Neural Progenitor Cells in Culture.Behavioral Characterization of κ Opioid Receptor Agonist Spiradoline and Cannabinoid Receptor Agonist CP55940 Mixtures in Rats.Cannabinoid 1 receptor knockout mice display cold allodynia, but enhanced recovery from spared-nerve injury-induced mechanical hypersensitivity.The monoacylglycerol lipase inhibitor KML29 with gabapentin synergistically produces analgesia in mice.Prophylactic treatment with the tricyclic antidepressant desipramine prevents development of paclitaxel-induced neuropathic pain through activation of endogenous analgesic systems.Current evidence of cannabinoid-based analgesia obtained in preclinical and human experimental settings.Regulation of cannabinoid CB2 receptor constitutive activity in vivo: repeated treatments with inverse agonists reverse the acute activation of JNK and associated apoptotic signaling in mouse brain.Monoacylglycerol lipase inhibitors reverse paclitaxel-induced nociceptive behavior and proinflammatory markers in a mouse model of chemotherapy-induced neuropathy.Targeting the Endocannabinoid System for Prevention or Treatment of Chemotherapy-Induced Neuropathic Pain: Studies in Animal Models
P2860

CB1 Knockout Mice Unveil Sustained CB2-Mediated Antiallodynic Effects of the Mixed CB1/CB2 Agonist CP55,940 in a Mouse Model of Paclitaxel-Induced Neuropathic Pain.

Item
http://www.wikidata.org/entity/Q35745959
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
CB1 Knockout Mice Unveil Susta ...... axel-Induced Neuropathic Pain.
@ast
CB1 Knockout Mice Unveil Susta ...... axel-Induced Neuropathic Pain.
@en
type
label
CB1 Knockout Mice Unveil Susta ...... axel-Induced Neuropathic Pain.
@ast
CB1 Knockout Mice Unveil Susta ...... axel-Induced Neuropathic Pain.
@en
prefLabel
CB1 Knockout Mice Unveil Susta ...... axel-Induced Neuropathic Pain.
@ast
CB1 Knockout Mice Unveil Susta ...... axel-Induced Neuropathic Pain.
@en
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P1476
P2093
P2860
P304
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CB1 Knockout Mice Unveil Susta ...... taxel-Induced Neuropathic Pain
@en
P2093
P2860
P304
P31
P356
P433
P478
P50
P577
P5875
P698
P921
P932