Experimental autoimmune myocarditis in A/J mice is an interleukin-4-dependent disease with a Th2 phenotype. - Wikidata - awgldk - TriplyDB

Experimental autoimmune myocarditis in A/J mice is an interleukin-4-dependent disease with a Th2 phenotype.

Experimental autoimmune myocarditis in A/J mice is an interleukin-4-dependent disease with a Th2 phenotype.

http://www.wikidata.org/entity/Q35746595

about

Learning from myocarditis: mimicry, chaos and black holes Mutations in the 5' NTR and the Non-Structural Protein 3A of the Coxsackievirus B3 Selectively Attenuate Myocarditogenicity Eosinophils in Autoimmune Diseases.Complete Freund's adjuvant induces experimental autoimmune myocarditis by enhancing IL-6 production during initiation of the immune response Cardiac troponins and autoimmunity: their role in the pathogenesis of myocarditis and of heart failure.Impaired up-regulation of CD25 on CD4+ T cells in IFN-gamma knockout mice is associated with progression of myocarditis to heart failure Cardiac myosin and the TH1/TH2 paradigm in autoimmune myocarditis Women and autoimmune diseases.The transition from viral to autoimmune myocarditis.Low-dose mercury heightens early innate response to coxsackievirus infection in female mice.Quantitative analysis of myocardial inflammation by flow cytometry in murine autoimmune myocarditis: correlation with cardiac function.Interferon-gamma protects against chronic viral myocarditis by reducing mast cell degranulation, fibrosis, and the profibrotic cytokines transforming growth factor-beta 1, interleukin-1 beta, and interleukin-4 in the heart.Therapeutic administration of the chemokine CXCL1/KC abrogates autoimmune inflammatory heart disease TH1-TH2: a procrustean paradigm.Th2 regulation of viral myocarditis in mice: different roles for TLR3 versus TRIF in progression to chronic disease.T cell subsets and their signature cytokines in autoimmune and inflammatory diseases.Neutrophils sustain pathogenic CD8+ T cell responses in the heart.Viruses as adjuvants for autoimmunity: evidence from Coxsackievirus-induced myocarditis.Interferon-gamma induces chronic active myocarditis and cardiomyopathy in transgenic mice.Update on coxsackievirus B3 myocarditis.The proteoglycan biglycan enhances antigen-specific T cell activation potentially via MyD88 and TRIF pathways and triggers autoimmune perimyocarditis T-bet negatively regulates autoimmune myocarditis by suppressing local production of interleukin 17 Localization of CD8 T cell epitope within cardiac myosin heavy chain-α334-352 that induces autoimmune myocarditis in A/J mice Sex-defined T-cell responses to cardiac self determine differential outcomes of murine dilated cardiomyopathy Interleukin-13 protects against experimental autoimmune myocarditis by regulating macrophage differentiation.TLR3 deficiency induces chronic inflammatory cardiomyopathy in resistant mice following coxsackievirus B3 infection: role for IL-4.Spontaneous autoimmune myocarditis and cardiomyopathy in HLA-DQ8.NODAbo transgenic mice.STAT3 activity is necessary and sufficient for the development of immune-mediated myocarditis in mice and promotes progression to dilated cardiomyopathy.Autoimmunological features in inflammatory cardiomyopathy.Autoimmune myocarditis, valvulitis, and cardiomyopathy.Experimental priming of encephalitogenic Th1/Th17 cells requires pertussis toxin-driven IL-1β production by myeloid cells Cardiac myosin-Th17 responses promote heart failure in human myocarditis.Autoimmune cardiomyopathy and heart block develop spontaneously in HLA-DQ8 transgenic IAbeta knockout NOD mice L.E.A.P.S. heteroconjugate is able to prevent and treat experimental autoimmune myocarditis by altering trafficking of autoaggressive cells to the heart.Identification of cardiac troponin I sequence motifs leading to heart failure by induction of myocardial inflammation and fibrosis.IL-33 independently induces eosinophilic pericarditis and cardiac dilation: ST2 improves cardiac function Alternatively activated macrophages in infection and autoimmunity.Pathogenic IL-23 signaling is required to initiate GM-CSF-driven autoimmune myocarditis in mice.Fatal eosinophilic myocarditis develops in the absence of IFN-γ and IL-17A.Myocarditis: infection versus autoimmunity.

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P2860

Experimental autoimmune myocarditis in A/J mice is an interleukin-4-dependent disease with a Th2 phenotype.

http://www.wikidata.org/entity/Q35746595

description

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The American Journal of Pathology

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B H Schofield

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M Afanasyeva

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M J Zilliox

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P2860

Interleukin 4 induces cultured monocytes/macrophages to form giant multinucleated cells

Interaction of stat6 and NF-kappaB: direct association and synergistic activation of interleukin-4-induced transcription

The IL-4 receptor: signaling mechanisms and biologic functions

Murine IgG1 complexes trigger immune effector functions predominantly via Fc gamma RIII (CD16)

Cellular responses to interferon-gamma

Lethal autoimmune myocarditis in interferon-gamma receptor-deficient mice: enhanced disease severity by impaired inducible nitric oxide synthase induction.

Development of dendritic cells and their use in tumor therapy.

Therapeutic strategies for allergic diseases.

Activation of eotaxin gene transcription by NF-kappa B and STAT6 in human airway epithelial cells.

Regulation of antigen-specific CD8+ T cell homeostasis by perforin and interferon-gamma.

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