Cardiac disease in transgenic mice expressing human immunodeficiency virus-1 nef in cells of the immune system.
about
IKK2 inhibitor alleviates kidney and wasting diseases in a murine model of human AIDSOropharyngeal Candidiasis in HIV Infection: Analysis of Impaired Mucosal Immune Response to Candida albicans in Mice Expressing the HIV-1 TransgeneIncreased cardiovascular disease risk in the HIV-positive population on ART: potential role of HIV-Nef and TatGenetic characterization of HIV type 1 Nef-induced vesicle secretionHIV protein Nef causes dyslipidemia and formation of foam cells in mouse models of atherosclerosis.The Nef-mediated AIDS-like disease of CD4C/human immunodeficiency virus transgenic mice is associated with increased Fas/FasL expression on T cells and T-cell death but is not prevented in Fas-, FasL-, tumor necrosis factor receptor 1-, or interleukDefective IL-17- and IL-22-dependent mucosal host response to Candida albicans determines susceptibility to oral candidiasis in mice expressing the HIV-1 transgene.Macrophage diversity in cardiac inflammation: a reviewOverexpression of activated murine Notch1 and Notch3 in transgenic mice blocks mammary gland development and induces mammary tumorsOverexpression of Notch1 ectodomain in myeloid cells induces vascular malformations through a paracrine pathway.Secretion modification region-derived peptide disrupts HIV-1 Nef's interaction with mortalin and blocks virus and Nef exosome release.Immunopathogenesis of oropharyngeal candidiasis in human immunodeficiency virus infection.Hallmarks of HIV-1 pathogenesis are modulated by Nef's Secretion Modification RegionOligodendrocyte-specific expression of human immunodeficiency virus type 1 Nef in transgenic mice leads to vacuolar myelopathy and alters oligodendrocyte phenotype in vitro.Altered immune response differentially enhances susceptibility to Cryptococcus neoformans and Cryptococcus gattii infection in mice expressing the HIV-1 transgene.Left ventricular dysfunction in human immunodeficiency virus infection.Erythropoietin improves functional and histological recovery of traumatized skeletal muscle tissue.Primary human immunodeficiency virus type 1 nef alleles show major differences in pathogenicity in transgenic mice.CD4+ T cells from CD4C/HIVNef transgenic mice show enhanced activation in vivo with impaired proliferation in vitro but are dispensable for the development of a severe AIDS-like organ diseaseSelective expression of human immunodeficiency virus Nef in specific immune cell populations of transgenic mice is associated with distinct AIDS-like phenotypes.Open blunt crush injury of different severity determines nature and extent of local tissue regeneration and repair.
P2860
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P2860
Cardiac disease in transgenic mice expressing human immunodeficiency virus-1 nef in cells of the immune system.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
2002年论文
@zh
2002年论文
@zh-cn
name
Cardiac disease in transgenic ...... in cells of the immune system.
@ast
Cardiac disease in transgenic ...... in cells of the immune system.
@en
type
label
Cardiac disease in transgenic ...... in cells of the immune system.
@ast
Cardiac disease in transgenic ...... in cells of the immune system.
@en
prefLabel
Cardiac disease in transgenic ...... in cells of the immune system.
@ast
Cardiac disease in transgenic ...... in cells of the immune system.
@en
P2093
P2860
P1476
Cardiac disease in transgenic ...... in cells of the immune system.
@en
P2093
Denis G Kay
Etienne Tremblay
Paul Jolicoeur
Serge Jothy
Zaher Hanna
P2860
P304
P356
10.1016/S0002-9440(10)64184-3
P407
P577
2002-07-01T00:00:00Z