Relationship of sialyl-Lewis(x/a) underexpression and E-cadherin overexpression in the lymphovascular embolus of inflammatory breast carcinoma.
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Models of breast cancer: is merging human and animal models the future?Sialyl Lewis x expression in canine malignant mammary tumours: correlation with clinicopathological features and E-Cadherin expression.Molecular plasticity of E-cadherin and sialyl lewis x expression, in two comparative models of mammary tumorigenesis.Gain in cellular organization of inflammatory breast cancer: A 3D in vitro model that mimics the in vivo metastasisAldehyde dehydrogenase 1-positive cancer stem cells mediate metastasis and poor clinical outcome in inflammatory breast cancer.Synthesis and cell-selective antitumor properties of amino acid conjugated tumor-associated carbohydrate antigen-coated gold nanoparticles.Suberoylanilide hydroxamic acid blocks self-renewal and homotypic aggregation of inflammatory breast cancer spheroids.Spontaneously-forming spheroids as an in vitro cancer cell model for anticancer drug screeningThe lymphovascular embolus of inflammatory breast cancer expresses a stem cell-like phenotype.Angiogenesis, lymphangiogenesis, growth pattern, and tumor emboli in inflammatory breast cancer: a review of the current knowledge.Deficiency in surface expression of E-selectin ligand promotes lung colonization in a mouse model of breast cancer.Notch signaling mediates melanoma-endothelial cell communication and melanoma cell migration.Beyond cell-cell adhesion: Plakoglobin and the regulation of tumorigenesis and metastasisModeling and characterization of inflammatory breast cancer emboli grown in vitro.The lymphovascular embolus of inflammatory breast cancer exhibits a Notch 3 addiction.A novel sialyltransferase inhibitor AL10 suppresses invasion and metastasis of lung cancer cells by inhibiting integrin-mediated signaling.ERalpha signaling through slug regulates E-cadherin and EMT.Dynamic analysis of hepatoma spheroid formation: roles of E-cadherin and beta1-integrin.Hepatospheres: Three dimensional cell cultures resemble physiological conditions of the liver.Ex vivo treatment response of primary tumors and/or associated metastases for preclinical and clinical development of therapeutics.EGFR signaling promotes inflammation and cancer stem-like activity in inflammatory breast cancer.E-selectin and Sialyl Lewis X expression is associated with lymph node metastasis of invasive micropapillary carcinoma of the breast.TIG1 promotes the development and progression of inflammatory breast cancer through activation of Axl kinase.
P2860
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P2860
Relationship of sialyl-Lewis(x/a) underexpression and E-cadherin overexpression in the lymphovascular embolus of inflammatory breast carcinoma.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
2002年论文
@zh
2002年论文
@zh-cn
name
Relationship of sialyl-Lewis(x ...... inflammatory breast carcinoma.
@ast
Relationship of sialyl-Lewis(x ...... inflammatory breast carcinoma.
@en
type
label
Relationship of sialyl-Lewis(x ...... inflammatory breast carcinoma.
@ast
Relationship of sialyl-Lewis(x ...... inflammatory breast carcinoma.
@en
prefLabel
Relationship of sialyl-Lewis(x ...... inflammatory breast carcinoma.
@ast
Relationship of sialyl-Lewis(x ...... inflammatory breast carcinoma.
@en
P2093
P2860
P1476
Relationship of sialyl-Lewis(x ...... inflammatory breast carcinoma.
@en
P2093
James S Tomlinson
Mary L Alpaugh
Sanford H Barsky
P2860
P304
P356
10.1016/S0002-9440(10)64217-4
P407
P577
2002-08-01T00:00:00Z