Aging accelerates endotoxin-induced thrombosis : increased responses of plasminogen activator inhibitor-1 and lipopolysaccharide signaling with aging - Wikidata - awgldk - TriplyDB

Aging accelerates endotoxin-induced thrombosis : increased responses of plasminogen activator inhibitor-1 and lipopolysaccharide signaling with aging

Aging accelerates endotoxin-induced thrombosis : increased responses of plasminogen activator inhibitor-1 and lipopolysaccharide signaling with aging

http://www.wikidata.org/entity/Q35747706

about

Microglial cell dysregulation in brain aging and neurodegeneration The effects of age on inflammatory and coagulation-fibrinolysis response in patients hospitalized for pneumonia The effects of aging on pulmonary oxidative damage, protein nitration, and extracellular superoxide dismutase down-regulation during systemic inflammation.Intestine-specific deletion of microsomal triglyceride transfer protein increases mortality in aged mice.Age-dependent vulnerability to endotoxemia is associated with reduction of anticoagulant factors activated protein C and thrombomodulin.Plasminogen activator inhibitor type 1 gene polymorphism and sepsis.Overexpression of the cell cycle inhibitor p16INK4a promotes a prothrombotic phenotype following vascular injury in mice Increased coagulation and suppressed generation of activated protein C in aged mice during intra-abdominal sepsis.Abrogation of plasminogen activator inhibitor-1-vitronectin interaction ameliorates acute kidney injury in murine endotoxemia.Plasminogen activator inhibitor 1 (PAI-1): in vitro activities and clinical relevance.Improvement of sepsis by hepatocyte growth factor, an anti-inflammatory regulator: emerging insights and therapeutic potential.The aging kidney: a review -- part I.The immunopathogenesis of sepsis in elderly patients.4G/5G plasminogen activator inhibitor-1 polymorphisms and haplotypes are associated with pneumonia CHOP deficiency results in elevated lipopolysaccharide-induced inflammation and kidney injury Gene expression profile of mouse white adipose tissue during inflammatory stress: age-dependent upregulation of major procoagulant factors Aging exacerbates microvascular endothelial damage induced by circulating factors present in the serum of septic patients.Sepsis in old age: review of human and animal studies.Proteomics reveals age-related differences in the host immune response to sepsis.Aging in rat causes hepatic hyperresposiveness to interleukin-1beta which is mediated by neutral sphingomyelinase-2.Role of TGFβ signaling in the pathogenesis of Alzheimer's disease.Protective effects of icariin on cisplatin-induced acute renal injury in mice.Drotrecogin alfa (activated) treatment of older patients with severe sepsis.Transcriptional response of human umbilical vein endothelial cells to low doses of ionizing radiation.Chronic plasminogen activator inhibitor-1 (PAI-1) overexpression dampens CD25+ lymphocyte recruitment after lipopolysaccharide endotoxemia in mouse lung.

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P2860

Aging accelerates endotoxin-induced thrombosis : increased responses of plasminogen activator inhibitor-1 and lipopolysaccharide signaling with aging

http://www.wikidata.org/entity/Q35747706

description

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Aging accelerates endotoxin-in ...... accharide signaling with aging

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Aging accelerates endotoxin-in ...... accharide signaling with aging

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The American Journal of Pathology

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Aging accelerates endotoxin-in ...... accharide signaling with aging

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David J Loskutoff

http://www.w3.org/2001/XMLSchema#string

Hidehiko Saito

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Hong Yi

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Ken-ichi Isobe

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Koji Yamamoto

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Takayoshi Shimokawa

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Tetsuhito Kojima

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P2860

MD-2, a molecule that confers lipopolysaccharide responsiveness on Toll-like receptor 4

Defective LPS Signaling in C3H/HeJ and C57BL/10ScCr Mice: Mutations in Tlr4 Gene

Cutting edge: Toll-like receptor 4 (TLR4)-deficient mice are hyporesponsive to lipopolysaccharide: evidence for TLR4 as the Lps gene product

Mouse and human CD14 (myeloid cell-specific leucine-rich glycoprotein) primary structure deduced from cDNA clones

CD14, a receptor for complexes of lipopolysaccharide (LPS) and LPS binding protein

Regulation of murine type 1 plasminogen activator inhibitor gene expression in vivo. Tissue specificity and induction by lipopolysaccharide, tumor necrosis factor-alpha, and transforming growth factor-beta

Induction of plasminogen activator inhibitor 1 gene expression in murine liver by lipopolysaccharide. Cellular localization and role of endogenous tumor necrosis factor-alpha

Cellular localization of type 1 plasminogen activator inhibitor messenger RNA and protein in murine renal tissue

Transgenic mice expressing human CD14 are hypersensitive to lipopolysaccharide.

Murine CD14 gene expression in vivo: extramyeloid synthesis and regulation by lipopolysaccharide.

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lipopolysaccharide

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