CXCR4 antagonist AMD3100 accelerates impaired wound healing in diabetic mice. - Wikidata - awgldk - TriplyDB

CXCR4 antagonist AMD3100 accelerates impaired wound healing in diabetic mice.

CXCR4 antagonist AMD3100 accelerates impaired wound healing in diabetic mice.

http://www.wikidata.org/entity/Q35748620

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Chemokines as Therapeutic Targets to Improve Healing Efficiency of Chronic Wounds Chemokine Regulation of Angiogenesis During Wound Healing The Role of Chemokines in Mesenchymal Stem Cell Homing to Wounds Targeting the molecular and cellular interactions of the bone marrow niche in immunologic disease Pharmacological mobilization of endogenous stem cells significantly promotes skin regeneration after full-thickness excision: the synergistic activity of AMD3100 and tacrolimus.SDF-1α mediates wound-promoted tumor growth in a syngeneic orthotopic mouse model of breast cancer.Role of endothelial progenitor cells and inflammatory cytokines in healing of diabetic foot ulcers Combination therapy accelerates diabetic wound closure.Intraplacental gene therapy with Ad-IGF-1 corrects naturally occurring rabbit model of intrauterine growth restriction.Dermal fibroblast expression of stromal cell-derived factor-1 (SDF-1) promotes epidermal keratinocyte proliferation in normal and diseased skin.Diabetes Limits Stem Cell Mobilization Following G-CSF but Not Plerixafor Continuous AMD3100 Treatment Worsens Renal Fibrosis through Regulation of Bone Marrow Derived Pro-Angiogenic Cells Homing and T-Cell-Related Inflammation.A cellular, molecular, and pharmacological basis for appendage regeneration in mice.Constructal thermodynamics combined with infrared experiments to evaluate temperature differences in cells A novel autologous cell-based therapy to promote diabetic wound healing.Diabetes impairs stem cell and proangiogenic cell mobilization in humans.Impaired Mobilization of Vascular Reparative Bone Marrow Cells in Streptozotocin-Induced Diabetes but not in Leptin Receptor-Deficient db/db Mice.Matrix- and plasma-derived peptides promote tissue-specific injury responses and wound healing in diabetic swine.Specific disruption of Lnk in murine endothelial progenitor cells promotes dermal wound healing via enhanced vasculogenesis, activation of myofibroblasts, and suppression of inflammatory cell recruitment.CXCR4 in epidermal keratinocytes: crosstalk within the skin.Aging Effects on Cardiac Progenitor Cell Physiology.Concise Review: Perspectives and Clinical Implications of Bone Marrow and Circulating Stem Cell Defects in Diabetes.Harnessing CXCR4 antagonists in stem cell mobilization, HIV infection, ischemic diseases, and oncology.E2F1 Hinders Skin Wound Healing by Repressing Vascular Endothelial Growth Factor (VEGF) Expression, Neovascularization, and Macrophage Recruitment.Synergistic vasculogenic effects of AMD3100 and stromal-cell-derived factor-1α in vasa nervorum of the sciatic nerve of mice with diabetic peripheral neuropathy.Tauroursodeoxycholic acid, a bile acid, promotes blood vessel repair by recruiting vasculogenic progenitor cells.Substance P enhances EPC mobilization for accelerated wound healing.Observation of an improved healing process in superficial skin wounds after irradiation with a blue-LED haemostatic device.CXCR4 antagonist delivery on decellularized skin scaffold facilitates impaired wound healing in diabetic mice by increasing expression of SDF-1 and enhancing migration of CXCR4-positive cells.Stem cell homing using local delivery of plerixafor and stromal derived growth factor-1alpha for improved bone regeneration around Ti-implants.

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P2860

CXCR4 antagonist AMD3100 accelerates impaired wound healing in diabetic mice.

http://www.wikidata.org/entity/Q35748620

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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2011年论文

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2011年论文

@zh-cn

name

CXCR4 antagonist AMD3100 accelerates impaired wound healing in diabetic mice.

@ast

CXCR4 antagonist AMD3100 accelerates impaired wound healing in diabetic mice.

@en

type

label

CXCR4 antagonist AMD3100 accelerates impaired wound healing in diabetic mice.

@ast

CXCR4 antagonist AMD3100 accelerates impaired wound healing in diabetic mice.

@en

prefLabel

CXCR4 antagonist AMD3100 accelerates impaired wound healing in diabetic mice.

@ast

CXCR4 antagonist AMD3100 accelerates impaired wound healing in diabetic mice.

@en

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Journal of Investigative Dermatology

P1476

CXCR4 antagonist AMD3100 accelerates impaired wound healing in diabetic mice

@en

P2093

Aiko Ito

http://www.w3.org/2001/XMLSchema#string

Christine Kamide

http://www.w3.org/2001/XMLSchema#string

Douglas W Losordo

http://www.w3.org/2001/XMLSchema#string

Gangjian Qin

http://www.w3.org/2001/XMLSchema#string

Haruki Sekiguchi

http://www.w3.org/2001/XMLSchema#string

Hideya Takenaka

http://www.w3.org/2001/XMLSchema#string

Hiromichi Hamada

http://www.w3.org/2001/XMLSchema#string

John Kenny

http://www.w3.org/2001/XMLSchema#string

Kentaro Jujo

http://www.w3.org/2001/XMLSchema#string

Marie-Ange Renault

http://www.w3.org/2001/XMLSchema#string

P2860

The lymphocyte chemoattractant SDF-1 is a ligand for LESTR/fusin and blocks HIV-1 entry

Benefits of targeting both pericytes and endothelial cells in the tumor vasculature with kinase inhibitors

Plerixafor: AMD 3100, AMD3100, JM 3100, SDZ SID 791

Isolation of putative progenitor endothelial cells for angiogenesis

Stromal fibroblasts present in invasive human breast carcinomas promote tumor growth and angiogenesis through elevated SDF-1/CXCL12 secretion

Cutaneous wound healing

Angiogenesis and vasculogenesis as therapeutic strategies for postnatal neovascularization

CXCR4 blockade augments bone marrow progenitor cell recruitment to the neovasculature and reduces mortality after myocardial infarction

Rapid mobilization of murine and human hematopoietic stem and progenitor cells with AMD3100, a CXCR4 antagonist

Cellular and molecular basis of wound healing in diabetes.

P2888

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711-720

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scholarly article

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10.1038/JID.2011.356

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P407

P433

3 Pt 1

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P478

132

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P577

2011-11-03T00:00:00Z

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51765722

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P698

22048734

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3276738

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