Normal development and function of invariant natural killer T cells in mice with isoglobotrihexosylceramide (iGb3) deficiency.
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Invariant NKT cells as novel targets for immunotherapy in solid tumorsThe role of NKT cells in tumor immunityThe transcription factor PLZF directs the effector program of the NKT cell lineageSynthetic glycolipid activators of natural killer T cells as immunotherapeutic agentsiNKT Cells and Their Potential Lipid Ligands during Viral InfectionTurned on by danger: activation of CD1d-restricted invariant natural killer T cellsThe immunoregulatory role of type I and type II NKT cells in cancer and other diseasesCrystal Structures of Mouse CD1d-iGb3 Complex and its Cognate Vα14 T Cell Receptor Suggest a Model for Dual Recognition of Foreign and Self GlycolipidsRecognition of β-linked self glycolipids mediated by natural killer T cell antigen receptorsHumans lack iGb3 due to the absence of functional iGb3-synthase: implications for NKT cell development and transplantationGlobosides but not isoglobosides can impact the development of invariant NKT cells and their interaction with dendritic cellsInvariant NKT cells reduce the immunosuppressive activity of influenza A virus-induced myeloid-derived suppressor cells in mice and humansTranscription factor Bcl11b controls selection of invariant natural killer T-cells by regulating glycolipid presentation in double-positive thymocytesRaising the roof: the preferential pharmacological stimulation of Th1 and th2 responses mediated by NKT cells.CD1d protein structure determines species-selective antigenicity of isoglobotrihexosylceramide (iGb3) to invariant NKT cellsAdenosine A2A receptors induced on iNKT and NK cells reduce pulmonary inflammation and injury in mice with sickle cell disease.V(alpha)14iNKT cells promote liver pathology during adenovirus infection by inducing CCL5 production: implications for gene therapy.NKT cells mediate pulmonary inflammation and dysfunction in murine sickle cell disease through production of IFN-gamma and CXCR3 chemokines.[Globosides as key players in the pathophysiology of Shiga toxin-associated acute kidney failure and Fabry disease].Recognition of lyso-phospholipids by human natural killer T lymphocytes.Linking inflammation to natural killer T cell activation.Stimulation of natural killer T cells by glycolipidsLysosomal alpha-galactosidase controls the generation of self lipid antigens for natural killer T cellsCD1: A Singed Cat of the Three Antigen Presentation Systems.NKT cells at the maternal-fetal interface.Reciprocal Crosstalk between Dendritic Cells and Natural Killer T Cells: Mechanisms and Therapeutic Potential.Lipid and glycolipid antigens of CD1d-restricted natural killer T cells.Activation state and intracellular trafficking contribute to the repertoire of endogenous glycosphingolipids presented by CD1d [corrected]Alpha anomers of iGb3 and Gb3 stimulate cytokine production by natural killer T cellsDietary fatty acids modulate antigen presentation to hepatic NKT cells in nonalcoholic fatty liver disease.Multi-system disorders of glycosphingolipid and ganglioside metabolismOligoclonality and innate-like features in the TCR repertoire of type II NKT cells reactive to a beta-linked self-glycolipidCytokine dependent and independent iNKT cell activationInvariant NKT cell development requires a full complement of functional CD3 zeta immunoreceptor tyrosine-based activation motifsThe effect of intracellular trafficking of CD1d on the formation of TCR repertoire of NKT cells.NKT cells in sepsis.T cell receptor CDR2 beta and CDR3 beta loops collaborate functionally to shape the iNKT cell repertoireTranscriptional control of invariant NKT cell development.Depletion of globosides and isoglobosides fully reverts the morphologic phenotype of Fabry diseaseInvariant natural killer T cells as sensors and managers of inflammation.
P2860
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P2860
Normal development and function of invariant natural killer T cells in mice with isoglobotrihexosylceramide (iGb3) deficiency.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Normal development and functio ...... sylceramide (iGb3) deficiency.
@ast
Normal development and functio ...... sylceramide (iGb3) deficiency.
@en
type
label
Normal development and functio ...... sylceramide (iGb3) deficiency.
@ast
Normal development and functio ...... sylceramide (iGb3) deficiency.
@en
prefLabel
Normal development and functio ...... sylceramide (iGb3) deficiency.
@ast
Normal development and functio ...... sylceramide (iGb3) deficiency.
@en
P2860
P50
P356
P1476
Normal development and functio ...... sylceramide (iGb3) deficiency.
@en
P2093
Bruno Luckow
Hermann-Josef Gröne
P2860
P304
P356
10.1073/PNAS.0611139104
P407
P577
2007-03-19T00:00:00Z