The receptor for advanced glycation end products (RAGE) enhances autophagy and neutrophil extracellular traps in pancreatic cancer.
about
Reactive oxygen species and mitochondria: A nexus of cellular homeostasisOrigins, structures, and functions of circulating DNA in oncologyNeutrophils in the tumor microenvironment: trying to heal the wound that cannot heal.Chloroquine improves the response to ischemic muscle injury and increases HMGB1 after arterial ligation.At the Bench: Neutrophil extracellular traps (NETs) highlight novel aspects of innate immune system involvement in autoimmune diseases.Host-directed therapy targeting the Mycobacterium tuberculosis granuloma: a reviewPriming of neutrophils toward NETosis promotes tumor growth.Pancreatic Cancer-Induced Neutrophil Extracellular Traps: A Potential Contributor to Cancer-Associated Thrombosis.Glycative stress from advanced glycation end products (AGEs) and dicarbonyls: An emerging biological factor in cancer onset and progression.Plasma miR-185 is decreased in patients with esophageal squamous cell carcinoma and might suppress tumor migration and invasion by targeting RAGE.Autophagy and Reactive Oxygen Species Are Involved in Neutrophil Extracellular Traps Release Induced by C. albicans Morphotypes.Neutrophil Extracellular Traps Are Pathogenic in Ventilator-Induced Lung Injury and Partially Dependent on TLR4.Extracellular Influences: Molecular Subclasses and the Microenvironment in Pancreatic Cancer.Citrullinated histone H3 as a novel prognostic blood marker in patients with advanced cancer.Neutrophil extracellular traps in immunity and disease.Tissue factor-bearing microparticles and inflammation: a potential mechanism for the development of venous thromboembolism in cancer.Differences and similarities in the phenomenon of NETs formation in oral inflammation and in oral squamous cell carcinoma.Chloroquine reduces hypercoagulability in pancreatic cancer through inhibition of neutrophil extracellular traps.Autophagy in Neutrophils: From Granulopoiesis to Neutrophil Extracellular Traps
P2860
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P2860
The receptor for advanced glycation end products (RAGE) enhances autophagy and neutrophil extracellular traps in pancreatic cancer.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
The receptor for advanced glyc ...... ar traps in pancreatic cancer.
@ast
The receptor for advanced glyc ...... ar traps in pancreatic cancer.
@en
type
label
The receptor for advanced glyc ...... ar traps in pancreatic cancer.
@ast
The receptor for advanced glyc ...... ar traps in pancreatic cancer.
@en
prefLabel
The receptor for advanced glyc ...... ar traps in pancreatic cancer.
@ast
The receptor for advanced glyc ...... ar traps in pancreatic cancer.
@en
P2093
P2860
P356
P1433
P1476
The receptor for advanced glyc ...... ar traps in pancreatic cancer.
@en
P2093
A D Singhi
G C Gianfrate
L Orlichenko
N E Schapiro
P Loughran
P2860
P2888
P304
P356
10.1038/CGT.2015.21
P577
2015-04-24T00:00:00Z