Hypoxia-inducible transcription factors stabilization in the thick ascending limb protects against ischemic acute kidney injury. - Wikidata - awgldk - TriplyDB

Hypoxia-inducible transcription factors stabilization in the thick ascending limb protects against ischemic acute kidney injury.

Hypoxia-inducible transcription factors stabilization in the thick ascending limb protects against ischemic acute kidney injury.

http://www.wikidata.org/entity/Q35758445

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Hypoxia: The Force that Drives Chronic Kidney Disease Molecular mechanisms of ischemic preconditioning in the kidney Chronic hypoxia-inducible transcription factor-2 activation stably transforms juxtaglomerular renin cells into fibroblast-like cells in vivo.Deletion of von Hippel-Lindau protein converts renin-producing cells into erythropoietin-producing cells.Endothelial HIF-2 mediates protection and recovery from ischemic kidney injury.Renal cortical hexokinase and pentose phosphate pathway activation through the EGFR/Akt signaling pathway in endotoxin-induced acute kidney injury.The role of SDF-1-CXCR4/CXCR7 axis in the therapeutic effects of hypoxia-preconditioned mesenchymal stem cells for renal ischemia/reperfusion injury Muc1 is protective during kidney ischemia-reperfusion injury Innate immunity in donor procurement.Mouse model of ischemic acute kidney injury: technical notes and tricks Tubular von Hippel-Lindau knockout protects against rhabdomyolysis-induced AKI.Hypoxia-inducible factor-1α causes renal cyst expansion through calcium-activated chloride secretion.Vhl deletion in renal epithelia causes HIF-1α-dependent, HIF-2α-independent angiogenesis and constitutive diuresis.Cellular adaptive changes in AKI: mitigating renal hypoxic injury.The signaling pathway of hypoxia inducible factor and its role in renal diseases.Hypoxia as a key player in the AKI-to-CKD transition.Kidney cell-specific knockdown: anything but simple.Adaptive response to hypoxia and remote ischaemia pre-conditioning: a new hypoxia-inducible factors era in clinical medicine.Expanding roles of the hypoxia-response network in chronic kidney disease.Hypoxia and Dysregulated Angiogenesis in Kidney Disease.Novel roles for mucin 1 in the kidney.NFAT5 is protective against ischemic acute kidney injury.Podocyte-specific soluble epoxide hydrolase deficiency in mice attenuates acute kidney injury.A primary culture system of mouse thick ascending limb cells with preserved function and uromodulin processing.AKI on CKD: heightened injury, suppressed repair, and the underlying mechanisms.Leukemia kidney infiltration can cause secondary polycythemia by activating hypoxia-inducible factor (HIF) pathway.Improving mitochondrial bioenergetics under ischemic conditions increases warm ischemia tolerance in the kidney.Generation of autochthonous mouse models of clear cell renal cell carcinoma: mouse models of renal cell carcinoma.Serum uromodulin-a marker of kidney function and renal parenchymal integrity.Multiparametric magnetic resonance imaging of experimental chronic kidney disease: A quantitative correlation study with histology

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P2860

Hypoxia-inducible transcription factors stabilization in the thick ascending limb protects against ischemic acute kidney injury.

http://www.wikidata.org/entity/Q35758445

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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2011年论文

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2011年论文

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Hypoxia-inducible transcriptio ...... ischemic acute kidney injury.

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Hypoxia-inducible transcriptio ...... ischemic acute kidney injury.

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Hypoxia-inducible transcriptio ...... ischemic acute kidney injury.

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Hypoxia-inducible transcriptio ...... ischemic acute kidney injury.

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Hypoxia-inducible transcriptio ...... ischemic acute kidney injury.

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Hypoxia-inducible transcriptio ...... ischemic acute kidney injury.

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Hypoxia-inducible transcriptio ...... ischemic acute kidney injury.

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Armin Kurtz

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Bernd Klanke

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Carsten Willam

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Deepa Shukla

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Frauke Forstreuter

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Gunnar Schley

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Johannes Schödel

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Kai-Uwe Eckardt

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Kerstin Amann

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Michael S Wiesener

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P2860

The tumour suppressor protein VHL targets hypoxia-inducible factors for oxygen-dependent proteolysis

HIFalpha targeted for VHL-mediated destruction by proline hydroxylation: implications for O2 sensing

C. elegans EGL-9 and mammalian homologs define a family of dioxygenases that regulate HIF by prolyl hydroxylation

Hypoxia promotes fibrogenesis in vivo via HIF-1 stimulation of epithelial-to-mesenchymal transition

Hypoxic regulation of erythropoiesis and iron metabolism

Targeting of HIF-alpha to the von Hippel-Lindau ubiquitylation complex by O2-regulated prolyl hydroxylation

Recent advances in mechanisms regulating glucose oxidation at the level of the pyruvate dehydrogenase complex by PDKs

Molecular basis of the VHL hereditary cancer syndrome

Study of factors which modify the development of norepinephrine-induced acute renal failure in the dog

Hypoxia-inducible factor-2 (HIF-2) regulates hepatic erythropoietin in vivo

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2004-2015

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10.1681/ASN.2010121249

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11

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22

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