Analysis of apoptosis during hair follicle regression (catagen) - Wikidata - awgldk - TriplyDB

Analysis of apoptosis during hair follicle regression (catagen)

Analysis of apoptosis during hair follicle regression (catagen)

http://www.wikidata.org/entity/Q35764942

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Macrophages contribute to the cyclic activation of adult hair follicle stem cells Your neighbours matter - non-autonomous control of apoptosis in development and disease Mouse hair cycle expression dynamics modeled as coupled mesenchymal and epithelial oscillators 6-Gingerol inhibits hair shaft growth in cultured human hair follicles and modulates hair growth in mice A new role for neurotrophin-3: involvement in the regulation of hair follicle regression (catagen)Resting no more: re-defining telogen, the maintenance stage of the hair growth cycle Stress inhibits hair growth in mice by induction of premature catagen development and deleterious perifollicular inflammatory events via neuropeptide substance P-dependent pathways Hairless is a nuclear receptor corepressor essential for skin function.What causes alopecia areata?Conditional epidermal expression of TGFbeta 1 blocks neonatal lethality but causes a reversible hyperplasia and alopecia p53/p63/p73 in the epidermis in health and disease Estrogen leads to reversible hair cycle retardation through inducing premature catagen and maintaining telogen Stem cell dynamics in the hair follicle niche Wls is expressed in the epidermis and regulates embryonic hair follicle induction in mice Complex changes in the apoptotic and cell differentiation programs during initiation of the hair follicle response to chemotherapy Skin and hair follicle integrity is crucially dependent on beta 1 integrin expression on keratinocytes.A hot new twist to hair biology: involvement of vanilloid receptor-1 (VR1/TRPV1) signaling in human hair growth control.Human scalp hair follicles are both a target and a source of prolactin, which serves as an autocrine and/or paracrine promoter of apoptosis-driven hair follicle regression.The critical roles of serum/glucocorticoid-regulated kinase 3 (SGK3) in the hair follicle morphogenesis and homeostasis: the allelic difference provides novel insights into hair follicle biology.Neurogenic inflammation in stress-induced termination of murine hair growth is promoted by nerve growth factor.Bone morphogenetic protein signaling regulates postnatal hair follicle differentiation and cycling Involvement of the Edar signaling in the control of hair follicle involution (catagen).Post-transcriptional Regulation of Keratinocyte Progenitor Cell Expansion, Differentiation and Hair Follicle Regression by miR-22.Niche-induced cell death and epithelial phagocytosis regulate hair follicle stem cell pool.Impaired hair follicle morphogenesis and cycling with abnormal epidermal differentiation in nackt mice, a cathepsin L-deficient mutation.Prolactin and its receptor are expressed in murine hair follicle epithelium, show hair cycle-dependent expression, and induce catagen.The role of the hairless (hr) gene in the regulation of hair follicle catagen transformation.Abundant production of brain-derived neurotrophic factor by adult visceral epithelia. Implications for paracrine and target-derived Neurotrophic functions Different responses of epidermal and hair follicular cells to radiation correlate with distinct patterns of p53 and p21 induction.New roles for glial cell line-derived neurotrophic factor and neurturin: involvement in hair cycle control.Overexpression of Bcl-2 protects from ultraviolet B-induced apoptosis but promotes hair follicle regression and chemotherapy-induced alopecia.p53 Involvement in the control of murine hair follicle regression Dissecting the impact of chemotherapy on the human hair follicle: a pragmatic in vitro assay for studying the pathogenesis and potential management of hair follicle dystrophy.Hair follicle pigmentation Transcription Factor CTIP2 Maintains Hair Follicle Stem Cell Pool and Contributes to Altered Expression of LHX2 and NFATC1 Plakoglobin suppresses epithelial proliferation and hair growth in vivo A Guide to Studying Human Hair Follicle Cycling In Vivo Cyclosporine A stimulated hair growth from mouse vibrissae follicles in an organ culture model.Stress-induced neurogenic inflammation in murine skin skews dendritic cells towards maturation and migration: key role of intercellular adhesion molecule-1/leukocyte function-associated antigen interactions.Bi-compartmental communication contributes to the opposite proliferative behavior of Notch1-deficient hair follicle and epidermal keratinocytes.

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P2860

Analysis of apoptosis during hair follicle regression (catagen)

http://www.wikidata.org/entity/Q35764942

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Analysis of apoptosis during hair follicle regression (catagen)

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Analysis of apoptosis during hair follicle regression (catagen)

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Botchkarev VA

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Botchkareva NV

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P2860

Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death

Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation

NMR structure and mutagenesis of the Fas (APO-1/CD95) death domain

A death-domain-containing receptor that mediates apoptosis

Apoptosis by death factor

Bcl-2-deficient mice demonstrate fulminant lymphoid apoptosis, polycystic kidneys, and hypopigmented hair

Label-retaining cells reside in the bulge area of pilosebaceous unit: implications for follicular stem cells, hair cycle, and skin carcinogenesis

The Fas death factor

The TNF receptor superfamily of cellular and viral proteins: activation, costimulation, and death

Two tumor necrosis factor-binding proteins purified from human urine. Evidence for immunological cross-reactivity with cell surface tumor necrosis factor receptors.

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