Adenovirus-mediated over-expression of the cyclin/cyclin-dependent kinase inhibitor, p21 inhibits vascular smooth muscle cell proliferation and neointima formation in the rat carotid artery model of balloon angioplasty.
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GATA-6 induces p21(Cip1) expression and G1 cell cycle arrestTroglitazone inhibits vascular smooth muscle cell growth and intimal hyperplasiaNFATc1 targets cyclin A in the regulation of vascular smooth muscle cell multiplication during restenosisContribution of the helix-loop-helix factor Id2 to regulation of vascular smooth muscle cell proliferationAccumulation of human papillomavirus type 16 E7 protein bypasses G1 arrest induced by serum deprivation and by the cell cycle inhibitor p21MAPK phosphorylation of connexin 43 promotes binding of cyclin E and smooth muscle cell proliferation.Stat3-dependent acute Rantes production in vascular smooth muscle cells modulates inflammation following arterial injury in mice.Adenovirus-mediated expression of a ribozyme to c-myb mRNA inhibits smooth muscle cell proliferation and neointima formation in vivoAberrant cell cycle reentry in human and experimental inclusion body myositis and polymyositisThe mechanisms of coronary restenosis: insights from experimental models.Deficiency of cyclin-dependent kinase inhibitors p21Cip1 and p27Kip1 accelerates atherogenesis in apolipoprotein E-deficient mice.Reactive oxygen species are downstream mediators of p53-dependent apoptosisLocal gene delivery to the vessel wall.Genetic targeting for cardiovascular therapeutics: are we near the summit or just beginning the climb?Applied gene therapy in preclinical models of vascular injury.The scaffolding protein EBP50 promotes vascular smooth muscle cell proliferation and neointima formation by regulating Skp2 and p21(cip1).Gene transfer techniques for cardiac arrhythmias.Fas ligand gene transfer to the vessel wall inhibits neointima formation and overrides the adenovirus-mediated T cell response.Gene therapy for the extension of vein graft patency: a review.Does gene therapy become pharmacotherapy?Mechanisms and therapeutic potential of microRNAs in hypertension.The therapeutic potential of targeting the cell cycle.Effects of Fluid Shear Stress on a Distinct Population of Vascular Smooth Muscle Cells.p21WAF1 Is Required for Interleukin-16-Induced Migration and Invasion of Vascular Smooth Muscle Cells via the p38MAPK/Sp-1/MMP-9 Pathway.The cell cycle: a critical therapeutic target to prevent vascular proliferative disease.p21Cip1 modulates arterial wound repair through the stromal cell-derived factor-1/CXCR4 axis in mice.Glucagon-like peptide-1 inhibits vascular smooth muscle cell dedifferentiation through mitochondrial dynamics regulation.Gene therapy to prevent occlusion of venous bypass grafts.Downregulation of cyclin-dependent kinase 2 activity and cyclin A promoter activity in vascular smooth muscle cells by p27(KIP1), an inhibitor of neointima formation in the rat carotid artery.Transcriptional targeting of replication-defective adenovirus transgene expression to smooth muscle cells in vivo.Role of c-fos and E2F in the induction of cyclin A transcription and vascular smooth muscle cell proliferation.Role of the p21 cyclin-dependent kinase inhibitor in limiting intimal cell proliferation in response to arterial injury.Regulation of vascular smooth muscle proliferation by heparin: inhibition of cyclin-dependent kinase 2 activity by p27(kip1).Vascular smooth muscle cell proliferation in restenosis.Sp1 inhibits proliferation and induces apoptosis in vascular smooth muscle cells by repressing p21WAF1/Cip1 transcription and cyclin D1-Cdk4-p21WAF1/Cip1 complex formation.The IL-24 gene protects human umbilical vein endothelial cells against H₂O₂-induced injury and may be useful as a treatment for cardiovascular disease.Potential Molecular Mechanism of Retrograde Aortic Arch Stenosis in the Hybrid Approach to Hypoplastic Left Heart Syndrome.Magnesium deficiency suppresses cell cycle progression mediated by increase in transcriptional activity of p21(Cip1) and p27(Kip1) in renal epithelial NRK-52E cells.Identification of cyclin D3 as a direct target of E2A using DamID.BMP-2 inhibits proliferation of human aortic smooth muscle cells via p21Cip1/Waf1.
P2860
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P2860
Adenovirus-mediated over-expression of the cyclin/cyclin-dependent kinase inhibitor, p21 inhibits vascular smooth muscle cell proliferation and neointima formation in the rat carotid artery model of balloon angioplasty.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
Adenovirus-mediated over-expre ...... model of balloon angioplasty.
@ast
Adenovirus-mediated over-expre ...... model of balloon angioplasty.
@en
type
label
Adenovirus-mediated over-expre ...... model of balloon angioplasty.
@ast
Adenovirus-mediated over-expre ...... model of balloon angioplasty.
@en
prefLabel
Adenovirus-mediated over-expre ...... model of balloon angioplasty.
@ast
Adenovirus-mediated over-expre ...... model of balloon angioplasty.
@en
P2093
P2860
P356
P1476
Adenovirus-mediated over-expre ...... model of balloon angioplasty.
@en
P2093
P2860
P304
P356
10.1172/JCI118281
P407
P577
1995-11-01T00:00:00Z