Heritable susceptibility to severe Borrelia burgdorferi-induced arthritis is dominant and is associated with persistence of large numbers of spirochetes in tissues
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Molecular typing of Borrelia burgdorferi sensu lato: taxonomic, epidemiological, and clinical implicationsBorrelia burgdorferi binding of host complement regulator factor H is not required for efficient mammalian infectionCalprotectin, an abundant cytosolic protein from human polymorphonuclear leukocytes, inhibits the growth of Borrelia burgdorferi.Borrelia burgdorferi gene expression in vivo and spirochete pathogenicity.Assessment of decorin-binding protein A to the infectivity of Borrelia burgdorferi in the murine models of needle and tick infection.The plasminogen activation system enhances brain and heart invasion in murine relapsing fever borreliosis.Relative contributions of innate and acquired host responses to bacterial control and arthritis development in Lyme diseaseInterleukin-4 (IL-4) and IL-13 signaling pathways do not regulate Borrelia burgdorferi-induced arthritis in mice: IgG1 is not required for host control of tissue spirochetes.Comparison of protection in rabbits against host-adapted and cultivated Borrelia burgdorferi following infection-derived immunity or immunization with outer membrane vesicles or outer surface protein A.Partial pathogen protection by tick-bite sensitization and epitope recognition in peptide-immunized HLA DR3 transgenic mice.Distinct characteristics of resistance to Borrelia burgdorferi-induced arthritis in C57BL/6N mice.Modulation of murine Lyme borreliosis by interruption of the B7/CD28 T-cell costimulatory pathway.Clearance of Borrelia burgdorferi may not be required for resistance to experimental lyme arthritis.Borrelia burgdorferi stimulates the production of interleukin-10 in peripheral blood mononuclear cells from uninfected humans and rhesus monkeys.E and P selectins are not required for resistance to severe murine lyme arthritis.Activation of natural killer cells in arthritis-susceptible but not arthritis-resistant mouse strains following Borrelia burgdorferi infection.Quantitative detection of Borrelia burgdorferi by real-time PCR.PCR-Based quantification of Borrelia burgdorferi organisms in canine tissues over a 500-Day postinfection periodRapid and sensitive quantification of Borrelia burgdorferi-infected mouse tissues by continuous fluorescent monitoring of PCR.Induction of pro- and anti-inflammatory cytokines by Borrelia burgdorferi lipoproteins in monocytes is mediated by CD14.Genetic control of experimental lyme arthritis in the absence of specific immunityDual role of interleukin-10 in murine Lyme disease: regulation of arthritis severity and host defenseInterleukin-10 modulates proinflammatory cytokines in the human monocytic cell line THP-1 stimulated with Borrelia burgdorferi lipoproteins.Early induction of gamma interferon and interleukin-10 production in draining lymph nodes from mice infected with Borrelia burgdorferi.Coinfection with Borrelia burgdorferi and the agent of human granulocytic ehrlichiosis alters murine immune responses, pathogen burden, and severity of Lyme arthritis.Impact of genotypic variation of Borrelia burgdorferi sensu stricto on kinetics of dissemination and severity of disease in C3H/HeJ mice.Role of osteopontin in murine Lyme arthritis and host defense against Borrelia burgdorferi.Autocrine and exocrine regulation of interleukin-10 production in THP-1 cells stimulated with Borrelia burgdorferi lipoproteins.Quantitative detection of Borrelia burgdorferi in 2-millimeter skin samples of erythema migrans lesions: correlation of results with clinical and laboratory findings.A critical role for type I IFN in arthritis development following Borrelia burgdorferi infection of mice.Outer surface lipoproteins of Borrelia burgdorferi stimulate nitric oxide production by the cytokine-inducible pathwayInhibition of interleukin-17 prevents the development of arthritis in vaccinated mice challenged with Borrelia burgdorferi.Antigenic composition of Borrelia burgdorferi during infection of SCID mice.Cyclooxygenase 2 activity modulates the severity of murine Lyme arthritis.Murine Lyme arthritis development mediated by p38 mitogen-activated protein kinase activity.Borrelia burgdorferi-induced inflammation facilitates spirochete adaptation and variable major protein-like sequence locus recombinationInterleukin-10 anti-inflammatory response to Borrelia burgdorferi, the agent of Lyme disease: a possible role for suppressors of cytokine signaling 1 and 3.Sustained-release formulation of doxycycline hyclate for prophylaxis of tick bite infection in a murine model of Lyme borreliosisBb2Bb3 regulation of murine Lyme arthritis is distinct from Ncf1 and independent of the phagocyte nicotinamide adenine dinucleotide phosphate oxidaseNitric oxide production during murine Lyme disease: lack of involvement in host resistance or pathology.
P2860
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P2860
Heritable susceptibility to severe Borrelia burgdorferi-induced arthritis is dominant and is associated with persistence of large numbers of spirochetes in tissues
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
1994年论文
@zh
1994年论文
@zh-cn
name
Heritable susceptibility to se ...... bers of spirochetes in tissues
@ast
Heritable susceptibility to se ...... bers of spirochetes in tissues
@en
type
label
Heritable susceptibility to se ...... bers of spirochetes in tissues
@ast
Heritable susceptibility to se ...... bers of spirochetes in tissues
@en
prefLabel
Heritable susceptibility to se ...... bers of spirochetes in tissues
@ast
Heritable susceptibility to se ...... bers of spirochetes in tissues
@en
P2093
P2860
P1476
Heritable susceptibility to se ...... bers of spirochetes in tissues
@en
P2093
P2860
P304
P407
P577
1994-02-01T00:00:00Z