Mice Deficient in Proglucagon-Derived Peptides Exhibit Glucose Intolerance on a High-Fat Diet but Are Resistant to Obesity.
about
Obesity-related known and candidate SNP markers can significantly change affinity of TATA-binding protein for human gene promoters.Regulation of amino acid metabolism and alpha cell proliferation by glucagon.Candidate SNP markers of reproductive potential are predicted by a significant change in the affinity of TATA-binding protein for human gene promoters.
P2860
Mice Deficient in Proglucagon-Derived Peptides Exhibit Glucose Intolerance on a High-Fat Diet but Are Resistant to Obesity.
description
2015 nî lūn-bûn
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2015年の論文
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name
Mice Deficient in Proglucagon- ...... but Are Resistant to Obesity.
@ast
Mice Deficient in Proglucagon- ...... but Are Resistant to Obesity.
@en
type
label
Mice Deficient in Proglucagon- ...... but Are Resistant to Obesity.
@ast
Mice Deficient in Proglucagon- ...... but Are Resistant to Obesity.
@en
prefLabel
Mice Deficient in Proglucagon- ...... but Are Resistant to Obesity.
@ast
Mice Deficient in Proglucagon- ...... but Are Resistant to Obesity.
@en
P2093
P2860
P1433
P1476
Mice Deficient in Proglucagon- ...... but Are Resistant to Obesity.
@en
P2093
Keita Kinoshita
Nobuaki Ozaki
Yoshiharu Murata
Yoshiharu Oshida
Yusuke Seino
Yusuke Takagi
P2860
P304
P356
10.1371/JOURNAL.PONE.0138322
P407
P577
2015-09-17T00:00:00Z