Aortic endothelium in HIV-1 infection: chronic injury, activation, and increased leukocyte adherence.
about
Human immunodeficiency virus type 1 Tat regulates endothelial cell actin cytoskeletal dynamics through PAK1 activation and oxidant productionCardiovascular disease in human immunodeficiency virus infected patients: A true or perceived risk?Role of high-sensitivity C-reactive protein measurements in HIV patientsAngiogenesis, Kaposi's Sarcoma and Kaposi's Sarcoma-Associated HerpesvirusAngiogenic effects of extracellular human immunodeficiency virus type 1 Tat protein and its role in the pathogenesis of AIDS-associated Kaposi's sarcoma.HIV Tat induces expression of ICAM-1 in HUVECs: implications for miR-221/-222 in HIV-associated cardiomyopathy.Dilation of the aortic root in children infected with human immunodeficiency virus type 1: The Prospective P2C2 HIV Multicenter Study.The contribution of HIV infection to intracranial arterial remodeling: a pilot study.HIV is an independent predictor of aortic stiffness.Expanding role of circulating adhesion molecules in assessing prognosis and treatment response in human immunodeficiency virus infectionSoluble mediators of inflammation in HIV and their implications for therapeutics and vaccine developmentSerum 25-hydroxyvitamin d levels and C-reactive protein in persons with human immunodeficiency virus infection.Human immunodeficiency virus-1 transgene expression increases pulmonary vascular resistance and exacerbates hypoxia-induced pulmonary hypertension development.Endothelial function in human immunodeficiency virus-infected antiretroviral-naive subjects before and after starting potent antiretroviral therapy: The ACTG (AIDS Clinical Trials Group) Study 5152s.Increased soluble vascular cell adhesion molecule-1 plasma levels and soluble intercellular adhesion molecule-1 during antiretroviral therapy interruption and retention of elevated soluble vascular cellular adhesion molecule-1 levels following resumEndothelial function in HIV-infected persons.The association of high-sensitivity c-reactive protein and other biomarkers with cardiovascular disease in patients treated for HIV: a nested case-control study.Monocytes from HIV-infected individuals show impaired cholesterol efflux and increased foam cell formation after transendothelial migration.Brain large artery inflammation associated with HIV and large artery remodeling.Angiocentric CD3(+) T-cell infiltrates in human immunodeficiency virus type 1-associated central nervous system disease in children.Monocytes in Kaposi's sarcoma lesions are productively infected by human herpesvirus 8.Brain large artery lymphocytic inflammation and Human Immunodeficiency Virus-related brain arterial remodeling.
P2860
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P2860
Aortic endothelium in HIV-1 infection: chronic injury, activation, and increased leukocyte adherence.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
Aortic endothelium in HIV-1 in ...... increased leukocyte adherence.
@ast
Aortic endothelium in HIV-1 in ...... increased leukocyte adherence.
@en
type
label
Aortic endothelium in HIV-1 in ...... increased leukocyte adherence.
@ast
Aortic endothelium in HIV-1 in ...... increased leukocyte adherence.
@en
prefLabel
Aortic endothelium in HIV-1 in ...... increased leukocyte adherence.
@ast
Aortic endothelium in HIV-1 in ...... increased leukocyte adherence.
@en
P2093
P2860
P1476
Aortic endothelium in HIV-1 in ...... increased leukocyte adherence.
@en
P2093
P2860
P304
P407
P577
1996-12-01T00:00:00Z