A transgenic model of central nervous system autoimmunity mediated by CD4+ and CD8+ T and B cells. - Wikidata - awgldk - TriplyDB

A transgenic model of central nervous system autoimmunity mediated by CD4+ and CD8+ T and B cells.

A transgenic model of central nervous system autoimmunity mediated by CD4+ and CD8+ T and B cells.

http://www.wikidata.org/entity/Q35785943

about

CD8(+) T-Cells as Immune Regulators of Multiple Sclerosis The Non-Obese Diabetic Mouse Strain as a Model to Study CD8(+) T Cell Function in Relapsing and Progressive Multiple Sclerosis Role of T cell-glial cell interactions in creating and amplifying central nervous system inflammation and multiple sclerosis disease symptoms Multiple sclerosis: molecular mechanisms and therapeutic opportunities Relapsing-remitting central nervous system autoimmunity mediated by GFAP-specific CD8 T cells.Myelin Oligodendrocyte Glycoprotein: Deciphering a Target in Inflammatory Demyelinating Diseases.Neuroantigen-specific autoregulatory CD8+ T cells inhibit autoimmune demyelination through modulation of dendritic cell function.Experimental Autoimmune Encephalomyelitis in Mice Meningeal Infiltration of the Spinal Cord by Non-Classically Activated B Cells is Associated with Chronic Disease Course in a Spontaneous B Cell-Dependent Model of CNS Autoimmune Disease Mincle and human B cell function Autoregulatory CD8 T cells depend on cognate antigen recognition and CD4/CD8 myelin determinants.What do effective treatments for multiple sclerosis tell us about the molecular mechanisms involved in pathogenesis?The disease-ameliorating function of autoregulatory CD8 T cells is mediated by targeting of encephalitogenic CD4 T cells in experimental autoimmune encephalomyelitis Multiple sclerosis: experimental models and reality.Using EAE to better understand principles of immune function and autoimmune pathology Bridging the gap from genetic association to functional understanding: the next generation of mouse models of multiple sclerosis.Multiple sclerosis: Prospects and promise.Autoimmunity in 2012.MicroRNA-181 Variants Regulate T Cell Phenotype in the Context of Autoimmune Neuroinflammation.Inhibition of myeloperoxidase by N-acetyl lysyltyrosylcysteine amide reduces experimental autoimmune encephalomyelitis-induced injury and promotes oligodendrocyte regeneration and neurogenesis in a murine model of progressive multiple sclerosis.

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A transgenic model of central nervous system autoimmunity mediated by CD4+ and CD8+ T and B cells.

http://www.wikidata.org/entity/Q35785943

description

2012 nî lūn-bûn

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name

A transgenic model of central ...... y CD4+ and CD8+ T and B cells.

@ast

A transgenic model of central ...... y CD4+ and CD8+ T and B cells.

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type

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A transgenic model of central ...... y CD4+ and CD8+ T and B cells.

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A transgenic model of central ...... y CD4+ and CD8+ T and B cells.

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A transgenic model of central ...... y CD4+ and CD8+ T and B cells.

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A transgenic model of central ...... y CD4+ and CD8+ T and B cells.

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P356

JIMMUNOL.1102186

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Journal of Immunology

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A transgenic model of central ...... y CD4+ and CD8+ T and B cells.

@en

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Adam Solomon

http://www.w3.org/2001/XMLSchema#string

Ana C Anderson

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Bernhard Greve

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David H Lee

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Jenna M Sullivan

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Raymond A Sobel

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Roselynn Rodriguez-Manzanet

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Rucha Chandwaskar

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Vijay K Kuchroo

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P2860

Immune regulatory CNS-reactive CD8+T cells in experimental autoimmune encephalomyelitis.

Viral infection triggers central nervous system autoimmunity via activation of CD8+ T cells expressing dual TCRs.

B cells and antibodies in CNS demyelinating disease.

Spontaneous opticospinal encephalomyelitis in a double-transgenic mouse model of autoimmune T cell/B cell cooperation.

Myelin oligodendrocyte glycoprotein-specific T and B cells cooperate to induce a Devic-like disease in mice.

Fulminant spontaneous autoimmunity of the central nervous system in mice transgenic for the myelin proteolipid protein-specific T cell receptor.

Clustering of non-major histocompatibility complex susceptibility candidate loci in human autoimmune diseases.

Clonal expansions of CD8(+) T cells dominate the T cell infiltrate in active multiple sclerosis lesions as shown by micromanipulation and single cell polymerase chain reaction

Myelin oligodendrocyte glycoprotein-specific T cell receptor transgenic mice develop spontaneous autoimmune optic neuritis.

A pathogenic role for myelin-specific CD8(+) T cells in a model for multiple sclerosis.

P304

2084-2092

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scholarly article

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10.4049/JIMMUNOL.1102186

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5

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188

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2012-01-25T00:00:00Z

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221779146

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22279107

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3288950

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