Transforming growth factor-beta-dependent and -independent pathways of induction of tubulointerstitial fibrosis in beta6(-/-) mice. - Wikidata - awgldk - TriplyDB

Transforming growth factor-beta-dependent and -independent pathways of induction of tubulointerstitial fibrosis in beta6(-/-) mice.

Transforming growth factor-beta-dependent and -independent pathways of induction of tubulointerstitial fibrosis in beta6(-/-) mice.

http://www.wikidata.org/entity/Q35792147

about

Cellular and molecular mechanisms of fibrosis Galectin-3 expression and secretion links macrophages to the promotion of renal fibrosis Emerging urinary markers of renal injury in obstructive nephropathy Molecular imaging of integrin αvβ6 expression in living subjects.Integrins in kidney disease αv integrins: key regulators of tissue fibrosis Pulmonary fibrosis: pathogenesis, etiology and regulation Characterization of integrin beta6 and thrombospondin-1 double-null mice Aldosterone and TGF-β₁ synergistically increase PAI-1 expression in hepatic stellate cells of rats Angiotensin II overcomes strain-dependent resistance of rapid CKD progression in a new remnant kidney mouse model Integrins αvβ5 and αvβ3 promote latent TGF-β1 activation by human cardiac fibroblast contraction Mechanisms of the epithelial-mesenchymal transition by TGF-beta Integrin-mediated type II TGF-β receptor tyrosine dephosphorylation controls SMAD-dependent profibrotic signaling.Sulodexide ameliorates early but not late kidney disease in models of radiation nephropathy and diabetic nephropathy.Grape seed pro-anthocyanidins ameliorates radiation-induced lung injury.TGF-beta receptor deletion in the renal collecting system exacerbates fibrosis.Mediators leading to fibrosis - how to measure and control them in tissue engineering.Ligation of protease-activated receptor 1 enhances alpha(v)beta6 integrin-dependent TGF-beta activation and promotes acute lung injury.Angiotensin type 1 receptor modulates macrophage polarization and renal injury in obesity.Induction of Tet3-dependent Epigenetic Remodeling by Low-dose Hydralazine Attenuates Progression of Chronic Kidney Disease Pharmacological inhibition of soluble epoxide hydrolase prevents renal interstitial fibrogenesis in obstructive nephropathy.Decay-accelerating factor 1 deficiency exacerbates leptospiral-induced murine chronic nephritis and renal fibrosis.Alphav beta6 integrin regulates renal fibrosis and inflammation in Alport mouse.Reducing affinity of αvβ8 interactions with latent TGFβ: dialling down fibrosis.Low-dose paclitaxel ameliorates fibrosis in the remnant kidney model by down-regulating miR-192.The αvβ1 integrin plays a critical in vivo role in tissue fibrosis.Fibrotic disease and the T(H)1/T(H)2 paradigm.Mechanisms of progression of chronic kidney disease Obstructive nephropathy: insights from genetically engineered animals.Inhibition of integrin alpha(v)beta6, an activator of latent transforming growth factor-beta, prevents radiation-induced lung fibrosis Alphavbeta6 integrin in wound healing and cancer of the oral cavity.Transforming growth factor β-1 stimulates profibrotic epithelial signaling to activate pericyte-myofibroblast transition in obstructive kidney fibrosis.Induction of an epithelial integrin alphavbeta6 in human cytomegalovirus-infected endothelial cells leads to activation of transforming growth factor-beta1 and increased collagen production.The role of Smad3 in the fibrotic phenotype in human vocal fold fibroblasts TGF-β signaling in the kidney: profibrotic and protective effects PAI-1 and kidney fibrosis.Modulation of glomerulosclerosis.Oxidative stress, plasminogen activator inhibitor 1, and lung fibrosis Periostin induces proliferation of human autosomal dominant polycystic kidney cells through alphaV-integrin receptor Quantification of angiotensin II-regulated proteins in urine of patients with polycystic and other chronic kidney diseases by selected reaction monitoring.

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P2860

Transforming growth factor-beta-dependent and -independent pathways of induction of tubulointerstitial fibrosis in beta6(-/-) mice.

http://www.wikidata.org/entity/Q35792147

description

2003 nî lūn-bûn

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

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2003年论文

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2003年论文

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2003年论文

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Transforming growth factor-bet ...... l fibrosis in beta6(-/-) mice.

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Transforming growth factor-bet ...... l fibrosis in beta6(-/-) mice.

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Transforming growth factor-bet ...... l fibrosis in beta6(-/-) mice.

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Transforming growth factor-bet ...... l fibrosis in beta6(-/-) mice.

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Transforming growth factor-bet ...... l fibrosis in beta6(-/-) mice.

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Transforming growth factor-bet ...... l fibrosis in beta6(-/-) mice.

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The American Journal of Pathology

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Transforming growth factor-bet ...... l fibrosis in beta6(-/-) mice.

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Agnes B Fogo

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Ariana Gaspert

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Dean Sheppard

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Gianluca Carlesso

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Haichun Yang

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Jeffrey M Davidson

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Li-Jun Ma

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Melissa M Barty

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P2860

Interactions between growth factors and integrins: latent forms of transforming growth factor-beta are ligands for the integrin alphavbeta1

Inactivation of the integrin beta 6 subunit gene reveals a role of epithelial integrins in regulating inflammation in the lung and skin

TGF-beta signalling from cell membrane to nucleus through SMAD proteins

The integrin alpha v beta 6 binds and activates latent TGF beta 1: a mechanism for regulating pulmonary inflammation and fibrosis

Mouse beta(6) integrin sequence, pattern of expression, and role in kidney development

Smads: transcriptional activators of TGF-beta responses

Thrombospondin-1 is a major activator of TGF-beta1 in vivo

Reduced angiotensinogen expression attenuates renal interstitial fibrosis in obstructive nephropathy in mice

Plasminogen activator inhibitor-1 expression is regulated by the angiotensin type 1 receptor in vivo.

The role of angiotensin II and plasminogen activator inhibitor-1 in progressive glomerulosclerosis.

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1868298

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