Differentiation-Dependent KLF4 Expression Promotes Lytic Epstein-Barr Virus Infection in Epithelial Cells
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Identification of Novel Small Organic Compounds with Diverse Structures for the Induction of Epstein-Barr Virus (EBV) Lytic Cycle in EBV-Positive Epithelial MalignanciesMicroRNA 26a (miR-26a)/KLF4 and CREB-C/EBPβ regulate innate immune signaling, the polarization of macrophages and the trafficking of Mycobacterium tuberculosis to lysosomes during infection.Hypoxia-inducible factor-1α plays roles in Epstein-Barr virus's natural life cycle and tumorigenesis by inducing lytic infection through direct binding to the immediate-early BZLF1 gene promoter.Post-Transcriptional Regulation of KLF4 by High-Risk Human Papillomaviruses Is Necessary for the Differentiation-Dependent Viral Life Cycle.Epstein-Barr virus mRNA profiles and viral DNA methylation status in nasopharyngeal brushings from nasopharyngeal carcinoma patients reflect tumor origin5-hydroxymethylation of the EBV genome regulates the latent to lytic switch.Status of Epstein-Barr Virus Coinfection with Helicobacter pylori in Gastric Cancer.Human papillomavirus promotes Epstein-Barr virus maintenance and lytic reactivation in immortalized oral keratinocytes.The Long and Complicated Relationship between Epstein-Barr Virus and Epithelial Cells.ZIC2 is Essential for Maintenance of Latency and is a Target of an Immediate-Early Protein during KSHV Lytic Reactivation.Epstein-Barr virus: a master epigenetic manipulator.Leflunomide/teriflunomide inhibit Epstein-Barr virus (EBV)- induced lymphoproliferative disease and lytic viral replication.Differentiation-Dependent LMP1 Expression Is Required for Efficient Lytic Epstein-Barr Virus Reactivation in Epithelial Cells.No evidence for the presence of Epstein-Barr virus in squamous cell carcinoma of the mobile tongueMicroenvironmental regulation of the progression of oral potentially malignant disorders towards malignancy.Latent Membrane Protein 1 Is a Novel Determinant of Epstein-Barr Virus Genome Persistence and Reactivation.CRISPR-Cas9 Genetic Analysis of Virus-Host Interactions.Interferon regulatory factor 8 regulates caspase-1 expression to facilitate Epstein-Barr virus reactivation in response to B cell receptor stimulation and chemical induction.New Insights from Elucidating the Role of LMP1 in Nasopharyngeal Carcinoma.The Immunomodulatory Capacity of an Epstein-Barr Virus Abortive Lytic Cycle: Potential Contribution to Viral Tumorigenesis.A cancer-associated Epstein-Barr virus BZLF1 promoter variant enhances lytic infectionAir-Liquid Interface System To Understand Epstein-Barr Virus-Associated Nasopharyngeal CarcinomaAir-Liquid Interface Method To Study Epstein-Barr Virus Pathogenesis in Nasopharyngeal Epithelial CellsEstablishment and characterization of new tumor xenografts and cancer cell lines from EBV-positive nasopharyngeal carcinoma
P2860
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P2860
Differentiation-Dependent KLF4 Expression Promotes Lytic Epstein-Barr Virus Infection in Epithelial Cells
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Differentiation-Dependent KLF4 ...... Infection in Epithelial Cells
@ast
Differentiation-Dependent KLF4 ...... Infection in Epithelial Cells
@en
type
label
Differentiation-Dependent KLF4 ...... Infection in Epithelial Cells
@ast
Differentiation-Dependent KLF4 ...... Infection in Epithelial Cells
@en
prefLabel
Differentiation-Dependent KLF4 ...... Infection in Epithelial Cells
@ast
Differentiation-Dependent KLF4 ...... Infection in Epithelial Cells
@en
P2093
P2860
P1433
P1476
Differentiation-Dependent KLF4 ...... Infection in Epithelial Cells
@en
P2093
Coral K Wille
Deborah Greenspan
Dhananjay M Nawandar
Elizabeth Barlow
Eric C Johannsen
Janet E Mertz
John S Greenspan
Kathleen Makielski
P2860
P304
P356
10.1371/JOURNAL.PPAT.1005195
P577
2015-10-02T00:00:00Z