Amyloid-β oligomers induce differential gene expression in adult human brain slices.
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The role of Nox2-derived ROS in the development of cognitive impairment after sepsisAstrocyte-induced synaptogenesis is mediated by transforming growth factor β signaling through modulation of D-serine levels in cerebral cortex neuronsRyR2-Mediated Ca2+ Release and Mitochondrial ROS Generation Partake in the Synaptic Dysfunction Caused by Amyloid β Peptide Oligomers.d-serine levels in Alzheimer's disease: implications for novel biomarker developmentElucidating molecular mass and shape of a neurotoxic Aβ oligomer.Alzheimer-associated Aβ oligomers impact the central nervous system to induce peripheral metabolic deregulation.Soluble amyloid-β oligomers as synaptotoxins leading to cognitive impairment in Alzheimer's diseaseAmyloid-β(25-35) Modulates the Expression of GirK and KCNQ Channel Genes in the Hippocampus.Astaxanthin Protects Primary Hippocampal Neurons against Noxious Effects of Aβ-Oligomers.Separate enrichment analysis of pathways for up- and downregulated genes.Effect of amyloids on the vesicular machinery: implications for somatic neurotransmission.The use of human neurons for novel drug discovery in dementia research.Genetic epistasis regulates amyloid deposition in resilient aging.Amyloid beta₁₋₄₂ (Aβ₄₂) up-regulates the expression of sortilin via the p75(NTR)/RhoA signaling pathway.Different effects of soluble and aggregated amyloid β42 on gene/protein expression and enzyme activity involved in insulin and APP pathways.Do age and sex impact on the absolute cell numbers of human brain regions?Neuroprotective astrocyte-derived insulin/insulin-like growth factor 1 stimulates endocytic processing and extracellular release of neuron-bound Aβ oligomers.Autism-like behavior caused by deletion of vaccinia-related kinase 3 is improved by TrkB stimulation.Amyloid-β oligomers transiently inhibit AMP-activated kinase and cause metabolic defects in hippocampal neurons.Interaction of amyloid-β (Aβ) oligomers with neurexin 2α and neuroligin 1 mediates synapse damage and memory loss in mice.GFAP and vimentin deficiency alters gene expression in astrocytes and microglia in wild-type mice and changes the transcriptional response of reactive glia in mouse model for Alzheimer's disease.The diabetes drug liraglutide reverses cognitive impairment in mice and attenuates insulin receptor and synaptic pathology in a non-human primate model of Alzheimer's disease.The Involvement of Aβ42 and Tau in Nucleolar and Protein Synthesis Machinery Dysfunction
P2860
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P2860
Amyloid-β oligomers induce differential gene expression in adult human brain slices.
description
2012 nî lūn-bûn
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2012年の論文
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2012年論文
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2012年論文
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2012年論文
@zh-hk
2012年論文
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2012年論文
@zh-tw
2012年论文
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2012年论文
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2012年论文
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name
Amyloid-β oligomers induce differential gene expression in adult human brain slices.
@ast
Amyloid-β oligomers induce differential gene expression in adult human brain slices.
@en
type
label
Amyloid-β oligomers induce differential gene expression in adult human brain slices.
@ast
Amyloid-β oligomers induce differential gene expression in adult human brain slices.
@en
prefLabel
Amyloid-β oligomers induce differential gene expression in adult human brain slices.
@ast
Amyloid-β oligomers induce differential gene expression in adult human brain slices.
@en
P2093
P2860
P50
P356
P1476
Amyloid-β oligomers induce differential gene expression in adult human brain slices.
@en
P2093
Andrea C Paula-Lima
Cesar Torres
Fabio F Oliveira
Fernanda G De Felice
Jorge M de Souza
Leo Freitas-Correa
Leonardo M Saraiva
Louise D Mota
Samantha M Martins
Sergio T Ferreira
P2860
P304
P356
10.1074/JBC.M111.298471
P407
P577
2012-01-10T00:00:00Z