Existence of transdominant and potentiating mutants of UL9, the herpes simplex virus type 1 origin-binding protein, suggests that levels of UL9 protein may be regulated during infection.
about
DNA binding activity of the herpes simplex virus type 1 origin binding protein, UL9, can be modulated by sequences in the N terminus: correlation between transdominance and DNA binding.Cathepsin B mediates cleavage of herpes simplex virus type 1 origin binding protein (OBP) to yield OBPC-1, and cleavage is dependent upon viral DNA replication.Herpes simplex viruses: mechanisms of DNA replication.Direct interaction between the N- and C-terminal portions of the herpes simplex virus type 1 origin binding protein UL9 implies the formation of a head-to-tail dimer.Dominant-negative proteins in herpesviruses - from assigning gene function to intracellular immunization.Human cytomegalovirus UL84 oligomerization and heterodimerization domains act as transdominant inhibitors of oriLyt-dependent DNA replication: evidence that IE2-UL84 and UL84-UL84 interactions are required for lytic DNA replicationInitiation of lytic DNA replication in Epstein-Barr virus: search for a common family mechanism.Genome replication and progeny virion production of herpes simplex virus type 1 mutants with temperature-sensitive lesions in the origin-binding protein.Herpes simplex virus type 1 C-terminal variants of the origin binding protein (OBP), OBPC-1 and OBPC-2, cooperatively regulate viral DNA levels in vitro, and OBPC-2 affects mortality in mice.
P2860
Q34647368-B7D56367-960E-40DA-8C38-45BAF5E9E7E0Q35948032-DFA8300C-7E4F-4D0A-B68F-F64A534BE283Q36192926-1C38E35E-C4CF-495B-B905-B232C7A0076DQ36315207-6F2A90FC-CF5E-454C-B828-C79C6B26343FQ38613434-7156973C-2F18-4F78-B362-47D2B71C1BD3Q39755438-2D4C5386-314A-433F-A73D-2E528ACA4D8DQ39914140-99C3C145-8E49-4A5E-88FF-98C5AB7E44B1Q41944564-166A6AC9-0919-41CB-9163-7C7705498833Q42147484-F731E1BE-A725-4AD3-9AE7-13CDEBEB2B2D
P2860
Existence of transdominant and potentiating mutants of UL9, the herpes simplex virus type 1 origin-binding protein, suggests that levels of UL9 protein may be regulated during infection.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Existence of transdominant and ...... be regulated during infection.
@ast
Existence of transdominant and ...... be regulated during infection.
@en
type
label
Existence of transdominant and ...... be regulated during infection.
@ast
Existence of transdominant and ...... be regulated during infection.
@en
prefLabel
Existence of transdominant and ...... be regulated during infection.
@ast
Existence of transdominant and ...... be regulated during infection.
@en
P2860
P1433
P1476
Existence of transdominant and ...... be regulated during infection.
@en
P2093
Boriana Marintcheva
Sandra K Weller
P2860
P304
P356
10.1128/JVI.77.17.9639-9651.2003
P407
P577
2003-09-01T00:00:00Z