Increased tubular proliferation as an adaptive response to glomerular albuminuria - Wikidata - awgldk - TriplyDB

Increased tubular proliferation as an adaptive response to glomerular albuminuria

Increased tubular proliferation as an adaptive response to glomerular albuminuria

http://www.wikidata.org/entity/Q35804206

about

Role of Receptor Tyrosine Kinase Signaling in Renal Fibrosis Axl tyrosine kinase protects against tubulo-interstitial apoptosis and progression of renal failure in a murine model of chronic kidney disease and hyperphosphataemia.Chronic endothelin-1 infusion elevates glomerular sieving coefficient and proximal tubular albumin reuptake in the rat Axl as a mediator of cellular growth and survival Lipocalin-2 expressed in innate immune cells is an endogenous inhibitor of inflammation in murine nephrotoxic serum nephritis.A urine-concentrating defect in 11β-hydroxysteroid dehydrogenase type 2 null mice.Essential Role of X-Box Binding Protein-1 during Endoplasmic Reticulum Stress in Podocytes.Albuminuria is associated with too few glomeruli and too much testosterone Peginesatide for the treatment of renal disease-induced anemia.mTOR controls kidney epithelia in health and disease.Renal allograft fibrosis: biology and therapeutic targets.Unraveling the role of podocyte turnover in glomerular aging and injury The regenerative potential of parietal epithelial cells in adult mice.Quantifying podocyte depletion: theoretical and practical considerations.Validation of a Three-Dimensional Method for Counting and Sizing Podocytes in Whole Glomeruli.Pentraxin-2 suppresses c-Jun/AP-1 signaling to inhibit progressive fibrotic disease.A more tubulocentric view of diabetic kidney disease.Vascular Endothelial Growth Factor-A165b Restores Normal Glomerular Water Permeability in a Diphtheria-Toxin Mouse Model of Glomerular Injury.Extracellular purines' action on glomerular albumin permeability in isolated rat glomeruli: insights into the pathogenesis of albuminuria.

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P2860

Increased tubular proliferation as an adaptive response to glomerular albuminuria

http://www.wikidata.org/entity/Q35804206

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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2011年论文

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2011年论文

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name

Increased tubular proliferation as an adaptive response to glomerular albuminuria

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Increased tubular proliferation as an adaptive response to glomerular albuminuria

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type

label

Increased tubular proliferation as an adaptive response to glomerular albuminuria

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Increased tubular proliferation as an adaptive response to glomerular albuminuria

@en

prefLabel

Increased tubular proliferation as an adaptive response to glomerular albuminuria

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Increased tubular proliferation as an adaptive response to glomerular albuminuria

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Journal of the American Society of Nephrology

P1476

Increased tubular proliferation as an adaptive response to glomerular albuminuria

@en

P2093

Alan Shan

http://www.w3.org/2001/XMLSchema#string

Arnaud Marlier

http://www.w3.org/2001/XMLSchema#string

Daniel Biemesderfer

http://www.w3.org/2001/XMLSchema#string

Diane S Krause

http://www.w3.org/2001/XMLSchema#string

Hongmei Shi

http://www.w3.org/2001/XMLSchema#string

Jian-Kan Guo

http://www.w3.org/2001/XMLSchema#string

Lloyd G Cantley

http://www.w3.org/2001/XMLSchema#string

Michael Kashgarian

http://www.w3.org/2001/XMLSchema#string

Thomas A Ardito

http://www.w3.org/2001/XMLSchema#string

Zhao-Peng Du

http://www.w3.org/2001/XMLSchema#string

P2860

Active (9.6 s) and inactive (21 s) oligomers of NHE3 in microdomains of the renal brush border.

Intrarenal cells, not bone marrow-derived cells, are the major source for regeneration in postischemic kidney.

Mitochondria are the major targets in albumin-induced apoptosis in proximal tubule cells.

Restoration of tubular epithelial cells during repair of the postischemic kidney occurs independently of bone marrow-derived stem cells.

PKB and megalin determine the survival or death of renal proximal tubule cells.

Animal models of FSGS: lessons for pathogenesis and treatment.

Podocyte differentiation and hereditary proteinuria/nephrotic syndromes.

Pathways to nephron loss starting from glomerular diseases-insights from animal models.

How does the kidney filter plasma?

Intracellular stability of diphtheria toxin fragment A in the presence and absence of anti-fragment A antibody.

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429-437

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scholarly article

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10.1681/ASN.2011040396

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3

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23

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2011-12-22T00:00:00Z

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51919600

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22193389

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3294312

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