Bisphenol A and its analogues activate human pregnane X receptor - Wikidata - awgldk - TriplyDB

Bisphenol A and its analogues activate human pregnane X receptor

Bisphenol A and its analogues activate human pregnane X receptor

http://www.wikidata.org/entity/Q35807873

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Perinatal bisphenol A exposure promotes dose-dependent alterations of the mouse methylome Molecular Mechanisms of Action of BPA Neurological Effects of Bisphenol A and its Analogues Structural and mechanistic insights into bisphenols action provide guidelines for risk assessment and discovery of bisphenol A substitutes EDC-2: The Endocrine Society's Second Scientific Statement on Endocrine-Disrupting Chemicals Exposure to endocrine disrupters and nuclear receptor gene expression in infertile and fertile women from different Italian areas Biotransformation of bisphenol AF to its major glucuronide metabolite reduces estrogenic activity Longitudinal Effects of Developmental Bisphenol A and Variable Diet Exposures on Epigenetic Drift in Mice The influence of phthalates and bisphenol A on the obesity development and glucose metabolism disorders.Novel functions of PXR in cardiometabolic disease.Bisphenol A promotes human prostate stem-progenitor cell self-renewal and increases in vivo carcinogenesis in human prostate epithelium.Bisphenol A increases atherosclerosis in pregnane X receptor-humanized ApoE deficient mice.Environmental Health Factors and Sexually Dimorphic Differences in Behavioral Disruptions.Bisphenol AF-induced endogenous transcription is mediated by ERα and ERK1/2 activation in human breast cancer cells.Intestinal pregnane X receptor links xenobiotic exposure and hypercholesterolemia.The molecular mechanism of bisphenol A (BPA) as an endocrine disruptor by interacting with nuclear receptors: insights from molecular dynamics (MD) simulations.Bisphenol A and Bisphenol S Induce Distinct Transcriptional Profiles in Differentiating Human Primary Preadipocytes.Exposure to Endocrine Disruptors and Nuclear Receptors Gene Expression in Infertile and Fertile Men from Italian Areas with Different Environmental Features.Weak activity of UDP-glucuronosyltransferase toward Bisphenol analogs in mouse perinatal development.Fetal liver bisphenol A concentrations and biotransformation gene expression reveal variable exposure and altered capacity for metabolism in humans.Tryptophan 299 is a conserved residue of human pregnane X receptor critical for the functional consequence of ligand binding.Perinatal bisphenol A exposure promotes hyperactivity, lean body composition, and hormonal responses across the murine life course.Pregnane X receptor mediates dyslipidemia induced by the HIV protease inhibitor amprenavir in mice.AhR-mediated effects of dioxin on neuronal acetylcholinesterase expression in vitro.Correlation between Conjugated Bisphenol A Concentrations and Efflux Transporter Expression in Human Fetal Livers.Piperine activates human pregnane X receptor to induce the expression of cytochrome P450 3A4 and multidrug resistance protein 1 Differential regulation of CYP3A4 promoter activity by a new class of natural product derivatives binding to pregnane X receptor.Fluorene-9-bisphenol is anti-oestrogenic and may cause adverse pregnancy outcomes in mice.Perinatal bisphenol A exposures increase production of pro-inflammatory mediators in bone marrow-derived mast cells of adult mice.Endocrine Disrupting Chemicals and Endometrial Cancer: An Overview of Recent Laboratory Evidence and Epidemiological Studies.Pregnane xenobiotic receptor in cancer pathogenesis and therapeutic response.Bisphenol a induces steatosis in HepaRG cells using a model of perinatal exposure.Endocrine-disrupting chemicals and uterine fibroids.Novel Epigenetic Biomarkers Mediating Bisphenol A Exposure and Metabolic Phenotypes in Female Mice.Low-Dose Bisphenol A Increases Bile Duct Proliferation in Juvenile Rats: A Possible Evidence for Risk of Liver Cancer in the Exposed Population?Accessing the molecular interactions of phthalates and their primary metabolites with the human pregnane X receptor using in silico profiling.Molecular characterization of PXR and two sulfotransferases and hepatic transcripts of PXR, two sulfotransferases and CYP3A responsive to bisphenol A in rare minnow Gobiocypris rarus.Are structural analogues to bisphenol a safe alternatives?In vitro profiling of toxicity and endocrine disrupting effects of bisphenol analogues by employing MCF-7 cells and two-hybrid yeast bioassay.Epigenetic impact of endocrine disrupting chemicals on lipid homeostasis and atherosclerosis: a pregnane X receptor-centric view.

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P2860

Bisphenol A and its analogues activate human pregnane X receptor

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Bisphenol A and its analogues activate human pregnane X receptor

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Bisphenol A and its analogues activate human pregnane X receptor

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Bisphenol A and its analogues activate human pregnane X receptor

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Bisphenol A and its analogues activate human pregnane X receptor

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Bisphenol A and its analogues activate human pregnane X receptor

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Bisphenol A and its analogues activate human pregnane X receptor

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Environmental Health Perspectives

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Bisphenol A and its analogues activate human pregnane X receptor

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P2093

Jennifer Rios-Pilier

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Jordan T Perkins

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Ni Ai

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Se-Hyung Park

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William J Welsh

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Yipeng Sui

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P2860

Association of urinary bisphenol a concentration with heart disease: evidence from NHANES 2003/06

SXR, a novel steroid and xenobiotic-sensing nuclear receptor

Chapel Hill bisphenol A expert panel consensus statement: integration of mechanisms, effects in animals and potential to impact human health at current levels of exposure

Bisphenol-A and the great divide: a review of controversies in the field of endocrine disruption

Endocrine-disrupting chemicals: an Endocrine Society scientific statement

Highly chlorinated PCBs inhibit the human xenobiotic response mediated by the steroid and xenobiotic receptor (SXR).

Rapid colorimetric assay for cellular growth and survival: Application to proliferation and cytotoxicity assays

Construction and characterization of a fully active PXR/SRC-1 tethered protein with increased stability

Endocrine disrupting chemicals, phthalic acid and nonylphenol, activate Pregnane X receptor-mediated transcription

Urinary, circulating, and tissue biomonitoring studies indicate widespread exposure to bisphenol A

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399-405

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10.1289/EHP.1104426

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3

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120

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51981066

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22214767

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3295358

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