about
Prenatal exposure to the environmental obesogen tributyltin predisposes multipotent stem cells to become adipocytesTriorganotin as a compound with potential reproductive toxicity in mammalsActivation of RXR–PPAR heterodimers by organotin environmental endocrine disruptorsInhibition of gap junctional Intercellular communication in WB-F344 rat liver epithelial cells by triphenyltin chloride through MAPK and PI3-kinase pathways.Transgenerational inheritance of prenatal obesogen exposure.The obesogenic effect of high fructose exposure during early development.Developmental effects: oestrogen-induced vaginal changes and organotin-induced adipogenesis.Postnatal development and fertility of offspring from mice exposed to triphenyltin (fentin) hydroxide during pregnancy and lactation.Effects of tributyltin chloride on developing mouse oocytes and preimplantation embryos.Childhood obesity and endocrine disrupting chemicals.Triphenyltin induced growth inhibition and antioxidative responses in the green microalga Scenedesmus quadricauda.Tributyltin impairs the reproductive cycle in female rats.Tributyltin impairs the coronary vasodilation induced by 17β-estradiol in isolated rat heart.Organotin Compounds Toxicity: Focus on Kidney.
P2860
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P2860
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Triphenyltin as a potential human endocrine disruptor.
@ast
Triphenyltin as a potential human endocrine disruptor.
@en
type
label
Triphenyltin as a potential human endocrine disruptor.
@ast
Triphenyltin as a potential human endocrine disruptor.
@en
prefLabel
Triphenyltin as a potential human endocrine disruptor.
@ast
Triphenyltin as a potential human endocrine disruptor.
@en
P2860
P1476
Triphenyltin as a potential human endocrine disruptor.
@en
P2093
John Doherty
Mari Golub
P2860
P304
P356
10.1080/10937400490452705
P577
2004-07-01T00:00:00Z