Complement receptor expression on neutrophils at an inflammatory site, the Pseudomonas-infected lung in cystic fibrosis.
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Bronchoalveolar lavage.Increased expression of the interleukin 1 receptor on blood neutrophils of humans with the sepsis syndromeState of the art: why do the lungs of patients with cystic fibrosis become infected and why can't they clear the infection?Neutrophil elastase up-regulates interleukin-8 via toll-like receptor 4.Capsular polysaccharide regulates neutrophil complement receptor interactions with type III group B streptococciSerine proteinase inhibitor therapy in alpha(1)-antitrypsin inhibitor deficiency and cystic fibrosis.Effect of nebulised recombinant DNase on neutrophil elastase load in cystic fibrosis.Anti-PcrV antibody in cystic fibrosis: a novel approach targeting Pseudomonas aeruginosa airway infection.Proteinase-activated receptor-2 up-regulation by Fcgamma-receptor activation in human neutrophilsModulation of secretory leukoprotease inhibitor gene expression in human bronchial epithelial cells by phorbol ester.Binding of Cryptococcus neoformans by human cultured macrophages. Requirements for multiple complement receptors and actin.Neutrophil elastase cleaves C3bi on opsonized pseudomonas as well as CR1 on neutrophils to create a functionally important opsonin receptor mismatch.Contribution of specific Pseudomonas aeruginosa virulence factors to pathogenesis of pneumonia in a neonatal mouse model of infectionLung infections. 3. Pseudomonas aeruginosa and other related species.Neutrophil elastase in respiratory epithelial lining fluid of individuals with cystic fibrosis induces interleukin-8 gene expression in a human bronchial epithelial cell line.Modulation of airway inflammation in cystic fibrosis. In vivo suppression of interleukin-8 levels on the respiratory epithelial surface by aerosolization of recombinant secretory leukoprotease inhibitor.Complement Effectors of Inflammation in Cystic Fibrosis Lung Fluid Correlate with Clinical Measures of DiseaseElastolytic proteases: inflammation resolution and dysregulation in chronic infective lung disease.Cystic fibrosis and other respiratory diseases of impaired mucus clearance.Proteases and cystic fibrosis.Galectin-9 signaling through TIM-3 is involved in neutrophil-mediated Gram-negative bacterial killing: an effect abrogated within the cystic fibrosis lung.The cystic fibrosis neutrophil: a specialized yet potentially defective cell.The role of proteases, endoplasmic reticulum stress and SERPINA1 heterozygosity in lung disease and α-1 anti-trypsin deficiency.Alpha-1 antitrypsin: a potent anti-inflammatory and potential novel therapeutic agent.The efficacy and safety of inhaled human α-1 antitrypsin in people with α-1 antitrypsin deficiency-related emphysema.Inflammation and its genesis in cystic fibrosis.Neutrophil Elastase Activity Is Associated with Exacerbations and Lung Function Decline in Bronchiectasis.Pseudomonas aeruginosa Sepsis Associated With Ecthyma Gangrenosum in a Previously Healthy Infant: A Case Report and Literature Review.Neutrophil Fates in Bronchiectasis and Alpha-1 Antitrypsin Deficiency.MudPIT analysis of released proteins in Pseudomonas aeruginosa laboratory and clinical strains in relation to pro-inflammatory effects.Immunomodulatory Cell Therapy to Target Cystic Fibrosis Inflammation.Age-dependent pulmonary clearance of Pseudomonas aeruginosa in a mouse model: diminished migration of polymorphonuclear leukocytes to N-formyl-methionyl-leucyl-phenylalanine.Antiproteases as therapeutics to target inflammation in cystic fibrosis.Interleukin-8 up-regulation by neutrophil elastase is mediated by MyD88/IRAK/TRAF-6 in human bronchial epithelium.The immunoevasive activities of Pseudomonas aeruginosa. Relevance for cystic fibrosis.The chemotactic activity of sputum from patients with bronchiectasis.Suppression of polymorphonuclear leucocyte chemotaxis by Pseudomonas aeruginosa elastase in vitro: a study of the mechanisms and the correlation with ring abscess in pseudomonal keratitis.The effects of bacterial products on airway cells and their function.Neutrophil-derived Oxidants and Proteinases as Immunomodulatory Mediators in Inflammation.Dampening Host Sensing and Avoiding Recognition in Pseudomonas aeruginosa Pneumonia.
P2860
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P2860
Complement receptor expression on neutrophils at an inflammatory site, the Pseudomonas-infected lung in cystic fibrosis.
description
1989 nî lūn-bûn
@nan
1989年の論文
@ja
1989年論文
@yue
1989年論文
@zh-hant
1989年論文
@zh-hk
1989年論文
@zh-mo
1989年論文
@zh-tw
1989年论文
@wuu
1989年论文
@zh
1989年论文
@zh-cn
name
Complement receptor expression ...... ected lung in cystic fibrosis.
@ast
Complement receptor expression ...... ected lung in cystic fibrosis.
@en
type
label
Complement receptor expression ...... ected lung in cystic fibrosis.
@ast
Complement receptor expression ...... ected lung in cystic fibrosis.
@en
prefLabel
Complement receptor expression ...... ected lung in cystic fibrosis.
@ast
Complement receptor expression ...... ected lung in cystic fibrosis.
@en
P2093
P2860
P356
P1476
Complement receptor expression ...... ected lung in cystic fibrosis.
@en
P2093
P2860
P304
P356
10.1172/JCI114298
P407
P577
1989-10-01T00:00:00Z