Effects of C-reactive protein on K(+) channel interaction protein 2 in cardiomyocytes.

about

Effects of C-reactive protein on K(+) channel interaction protein 2 in cardiomyocytes.

Item

http://www.wikidata.org/entity/Q35826487

description

2015 nî lūn-bûn

@nan

2015年の論文

@ja

2015年論文

@yue

2015年論文

@zh-hant

2015年論文

@zh-hk

2015年論文

@zh-mo

2015年論文

@zh-tw

2015年论文

@wuu

2015年论文

@zh

2015年论文

@zh-cn

name

Effects of C-reactive protein on K(+) channel interaction protein 2 in cardiomyocytes.

@ast

Effects of C-reactive protein on K(+) channel interaction protein 2 in cardiomyocytes.

@en

type

Item

label

Effects of C-reactive protein on K(+) channel interaction protein 2 in cardiomyocytes.

@ast

Effects of C-reactive protein on K(+) channel interaction protein 2 in cardiomyocytes.

@en

prefLabel

Effects of C-reactive protein on K(+) channel interaction protein 2 in cardiomyocytes.

@ast

Effects of C-reactive protein on K(+) channel interaction protein 2 in cardiomyocytes.

@en

P1476

Effects of C-reactive protein on K(+) channel interaction protein 2 in cardiomyocytes

@en

P2093

Fei Wang

http://www.w3.org/2001/XMLSchema#string

Jing-Feng Wang

http://www.w3.org/2001/XMLSchema#string

Jing-Ting Mai

http://www.w3.org/2001/XMLSchema#string

Ming-Cheng Fang

http://www.w3.org/2001/XMLSchema#string

Nian-Sang Luo

http://www.w3.org/2001/XMLSchema#string

Wo-Liang Yuan

http://www.w3.org/2001/XMLSchema#string

Yang-Xin Chen

http://www.w3.org/2001/XMLSchema#string

Yong Xie

http://www.w3.org/2001/XMLSchema#string

Yong-Qing Lin

http://www.w3.org/2001/XMLSchema#string

P2860

A defect in the Kv channel-interacting protein 2 (KChIP2) gene leads to a complete loss of I(to) and confers susceptibility to ventricular tachycardia

Functional modulation of the transient outward current Ito by KCNE beta-subunits and regional distribution in human non-failing and failing hearts

C-reactive protein and other circulating markers of inflammation in the prediction of coronary heart disease.

Mice with cardiac overexpression of peroxisome proliferator-activated receptor γ have impaired repolarization and spontaneous fatal ventricular arrhythmias

Four kinetically distinct depolarization-activated K+ currents in adult mouse ventricular myocytes.

Mechanisms of disease: ion channel remodeling in the failing ventricle.

Role of heteromultimers in the generation of myocardial transient outward K+ currents.

Inflammation and future risk of symptomatic heart failure in patients with stable coronary artery disease.

Prognostic role of high-sensitivity C-reactive protein and B-type natriuretic peptide in implantable cardioverter-defibrillator patients.

KChIP2 attenuates cardiac hypertrophy through regulation of Ito and intracellular calcium signaling.

P304

922-931

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2015-05-15T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

26175853

http://www.w3.org/2001/XMLSchema#string

P932

4494143

http://www.w3.org/2001/XMLSchema#string