Endogenous GLP-1 as a key self-defense molecule against lipotoxicity in pancreatic islets

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Anti-Inflammatory Effects of GLP-1-Based Therapies beyond Glucose Control Intra-islet glucagon-like peptide 1.

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P2860

Endogenous GLP-1 as a key self-defense molecule against lipotoxicity in pancreatic islets

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http://www.wikidata.org/entity/Q35827634

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

@yue

2015年論文

@zh-hant

2015年論文

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2015年論文

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2015年論文

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Endogenous GLP-1 as a key self-defense molecule against lipotoxicity in pancreatic islets

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Endogenous GLP-1 as a key self-defense molecule against lipotoxicity in pancreatic islets

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Item

label

Endogenous GLP-1 as a key self-defense molecule against lipotoxicity in pancreatic islets

@ast

Endogenous GLP-1 as a key self-defense molecule against lipotoxicity in pancreatic islets

@en

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Endogenous GLP-1 as a key self-defense molecule against lipotoxicity in pancreatic islets

@ast

Endogenous GLP-1 as a key self-defense molecule against lipotoxicity in pancreatic islets

@en

P1476

Endogenous GLP-1 as a key self-defense molecule against lipotoxicity in pancreatic islets

@en

P2093

Chenghu Huang

http://www.w3.org/2001/XMLSchema#string

Li Yuan

http://www.w3.org/2001/XMLSchema#string

Shuyi Cao

http://www.w3.org/2001/XMLSchema#string

P2860

A Mouse Model of Metabolic Syndrome: Insulin Resistance, Fatty Liver and Non-Alcoholic Fatty Pancreas Disease (NAFPD) in C57BL/6 Mice Fed a High Fat Diet.

GLP1-derived nonapeptide GLP1(28-36)amide protects pancreatic β-cells from glucolipotoxicity

Progressive change of intra-islet GLP-1 production during diabetes development

Divergent effects of liraglutide, exendin-4, and sitagliptin on beta-cell mass and indicators of pancreatitis in a mouse model of hyperglycaemia

Regeneration of pancreatic non-β endocrine cells in adult mice following a single diabetes-inducing dose of streptozotocin.

A local glucagon-like peptide 1 (GLP-1) system in human pancreatic islets.

The role of incretins in glucose homeostasis and diabetes treatment.

Oxidative stress and stress-activated signaling pathways: a unifying hypothesis of type 2 diabetes.

Activation of PPARβ/δ protects pancreatic β cells from palmitate-induced apoptosis by upregulating the expression of GLP-1 receptor.

Mitochondrial DNA damage via augmented oxidative stress regulates endoplasmic reticulum stress and autophagy: crosstalk, links and signaling

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173-185

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scholarly article

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10.3892/IJMM.2015.2207

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http://www.w3.org/2001/XMLSchema#string

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http://www.w3.org/2001/XMLSchema#string

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2015-05-12T00:00:00Z

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276357395

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25976560

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4494597

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Endogenous GLP-1 as a key self-defense molecule against lipotoxicity in pancreatic islets

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P2860

P2860

Endogenous GLP-1 as a key self-defense molecule against lipotoxicity in pancreatic islets

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P577

P5875

P698

P932

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P577

P5875

P698

P932