Fragmentation of the Golgi apparatus of motor neurons in amyotrophic lateral sclerosis
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Proteomic profiling of cerebrospinal fluid identifies biomarkers for amyotrophic lateral sclerosisGolgi Fragmentation in ALS Motor Neurons. New Mechanisms Targeting Microtubules, Tethers, and Transport VesiclesWho needs microtubules? Myogenic reorganization of MTOC, Golgi complex and ER exit sites persists despite lack of normal microtubule tracksTBCE Mutations Cause Early-Onset Progressive Encephalopathy with Distal Spinal Muscular AtrophyRecurrent Muscle Weakness with Rhabdomyolysis, Metabolic Crises, and Cardiac Arrhythmia Due to Bi-allelic TANGO2 MutationsDistinctive features of degenerating Purkinje cells in spinocerebellar ataxia type 31.Immunoreactivity of valosin-containing protein in sporadic amyotrophic lateral sclerosis and in a case of its novel mutantFragmentation of the Golgi apparatus induced by the overexpression of wild-type and mutant human tau forms in neurons.Highly immunoreactive IgG antibodies directed against a set of twenty human proteins in the sera of patients with amyotrophic lateral sclerosis identified by protein array.In Alzheimer's disease the Golgi apparatus of a population of neurons without neurofibrillary tangles is fragmented and atrophic.Rous-Whipple Award Lecture. Contributions to the physiology and pathology of the Golgi apparatusFragmentation of the Golgi apparatus of motor neurons in amyotrophic lateral sclerosis (ALS). Clinical studies in ALS of Guam and experimental studies in deafferented neurons and in beta,beta'-iminodipropionitrile axonopathy.Golgi fragmentation in amyotrophic lateral sclerosis, an overview of possible triggers and consequences.Dominant spinal muscular atrophy is caused by mutations in BICD2, an important golgin protein.Aggravated decrease in the activity of nucleus basalis neurons in Alzheimer's disease is apolipoprotein E-type dependent.Editorial: Golgi Pathology in Neurodegenerative Diseases.Stathmin 1/2-triggered microtubule loss mediates Golgi fragmentation in mutant SOD1 motor neurons.Characterization of early pathogenesis in the SOD1(G93A) mouse model of ALS: part II, results and discussionThe Golgi apparatus of spinal cord motor neurons in transgenic mice expressing mutant Cu,Zn superoxide dismutase becomes fragmented in early, preclinical stages of the diseaseLoss of the golgin GM130 causes Golgi disruption, Purkinje neuron loss, and ataxia in mice.Morphometric alterations of Golgi apparatus in Alzheimer's disease are related to tau hyperphosphorylation.Golgi fragmentation precedes neuromuscular denervation and is associated with endosome abnormalities in SOD1-ALS mouse motor neurons.Changes in the Golgi Apparatus of Neocortical and Hippocampal Neurons in the Hibernating Hamster.Clinical Significance of TDP-43 Neuropathology in Amyotrophic Lateral Sclerosis.Control of CNS functions by RNA-binding proteins in neurological diseases
P2860
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P2860
Fragmentation of the Golgi apparatus of motor neurons in amyotrophic lateral sclerosis
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
Fragmentation of the Golgi apparatus of motor neurons in amyotrophic lateral sclerosis
@ast
Fragmentation of the Golgi apparatus of motor neurons in amyotrophic lateral sclerosis
@en
type
label
Fragmentation of the Golgi apparatus of motor neurons in amyotrophic lateral sclerosis
@ast
Fragmentation of the Golgi apparatus of motor neurons in amyotrophic lateral sclerosis
@en
prefLabel
Fragmentation of the Golgi apparatus of motor neurons in amyotrophic lateral sclerosis
@ast
Fragmentation of the Golgi apparatus of motor neurons in amyotrophic lateral sclerosis
@en
P2093
P2860
P1476
Fragmentation of the Golgi apparatus of motor neurons in amyotrophic lateral sclerosis
@en
P2093
J O Gonatas
N K Gonatas
W F Hickey
Z Mourelatos
P2860
P304
P407
P577
1992-03-01T00:00:00Z