Naloxone inhibits immune cell function by suppressing superoxide production through a direct interaction with gp91phox subunit of NADPH oxidase - Wikidata - awgldk - TriplyDB

Naloxone inhibits immune cell function by suppressing superoxide production through a direct interaction with gp91phox subunit of NADPH oxidase

Naloxone inhibits immune cell function by suppressing superoxide production through a direct interaction with gp91phox subunit of NADPH oxidase

http://www.wikidata.org/entity/Q35833987

about

Type-1 angiotensin receptor signaling in central nervous system myeloid cells is pathogenic during fatal alphavirus encephalitis in mice NADPH oxidases: key modulators in aging and age-related cardiovascular diseases?The use of low-dose naltrexone (LDN) as a novel anti-inflammatory treatment for chronic pain The quest for selective nox inhibitors and therapeutics: challenges, triumphs and pitfalls Substance P enhances microglial density in the substantia nigra through neurokinin-1 receptor/NADPH oxidase-mediated chemotaxis in mice Substance P exacerbates dopaminergic neurodegeneration through neurokinin-1 receptor-independent activation of microglial NADPH oxidase NADPH-oxidase 2 activation promotes opioid-induced antinociceptive tolerance in mice.Opioid activation of toll-like receptor 4 contributes to drug reinforcement Subpicomolar diphenyleneiodonium inhibits microglial NADPH oxidase with high specificity and shows great potential as a therapeutic agent for neurodegenerative diseases.A novel role of microglial NADPH oxidase in mediating extra-synaptic function of norepinephrine in regulating brain immune homeostasis.Nox Inhibitors & Therapies: Rational Design of Peptidic and Small Molecule Inhibitors Discovery of GSK2795039, a Novel Small Molecule NADPH Oxidase 2 Inhibitor.Chronic ethanol increases systemic TLR3 agonist-induced neuroinflammation and neurodegeneration Neuroimmune Regulation of GABAergic Neurons Within the Ventral Tegmental Area During Withdrawal from Chronic Morphine.Pharmacological characterization of the opioid inactive isomers (+)-naltrexone and (+)-naloxone as antagonists of toll-like receptor 4 Effect of chronic delivery of the Toll-like receptor 4 antagonist (+)-naltrexone on incubation of heroin craving.Why is neuroimmunopharmacology crucial for the future of addiction research?Current status of NADPH oxidase research in cardiovascular pharmacology Morphinan alkaloids labeled with tritium: synthesis and applications.Targeting the Toll of Drug Abuse: The Translational Potential of Toll-Like Receptor 4.NADPH oxidases in oxidant production by microglia: activating receptors, pharmacology and association with disease.Role of microglia in ischemic focal stroke and recovery: focus on Toll-like receptors.Morphine Inhibited the Rat Neural Stem Cell Proliferation Rate by Increasing Neuro Steroid Genesis.Physiological Concentration of Prostaglandin E2 Exerts Anti-inflammatory Effects by Inhibiting Microglial Production of Superoxide Through a Novel Pathway.Post-stroke Intranasal (+)-Naloxone Delivery Reduces Microglial Activation and Improves Behavioral Recovery from Ischemic Injury.

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P2860

Naloxone inhibits immune cell function by suppressing superoxide production through a direct interaction with gp91phox subunit of NADPH oxidase

http://www.wikidata.org/entity/Q35833987

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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Naloxone inhibits immune cell ...... 1phox subunit of NADPH oxidase

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Naloxone inhibits immune cell ...... 1phox subunit of NADPH oxidase

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Naloxone inhibits immune cell ...... 1phox subunit of NADPH oxidase

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Naloxone inhibits immune cell ...... 1phox subunit of NADPH oxidase

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Naloxone inhibits immune cell ...... 1phox subunit of NADPH oxidase

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Naloxone inhibits immune cell ...... 1phox subunit of NADPH oxidase

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Journal of Neuroinflammation

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Naloxone inhibits immune cell ...... 1phox subunit of NADPH oxidase

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P2093

Belinda Wilson

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Chun-Hsien Chu

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Hui Zhou

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Huiming Gao

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Qingshan Wang

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Shih-Heng Chen

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P2860

The "toll" of opioid-induced glial activation: improving the clinical efficacy of opioids by targeting glia

Evidence that intrathecal morphine-3-glucuronide may cause pain enhancement via toll-like receptor 4/MD-2 and interleukin-1beta

The NOX family of ROS-generating NADPH oxidases: physiology and pathophysiology

Evidence that opioids may have toll-like receptor 4 and MD-2 effects

Non-stereoselective reversal of neuropathic pain by naloxone and naltrexone: involvement of toll-like receptor 4 (TLR4)

Naloxone's pentapeptide binding site on filamin A blocks Mu opioid receptor-Gs coupling and CREB activation of acute morphine

A role for proinflammatory cytokines and fractalkine in analgesia, tolerance, and subsequent pain facilitation induced by chronic intrathecal morphine

NOX enzymes and the biology of reactive oxygen

Regulation of plasminogen binding to neutrophils.

Naloxone protects rat dopaminergic neurons against inflammatory damage through inhibition of microglia activation and superoxide generation.

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32

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10.1186/1742-2094-9-32

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9

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Jau-Shyong Hong

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221838485

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1032649549

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22340895

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3305409

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