Purkinje cell dysfunction and alteration of long-term synaptic plasticity in fetal alcohol syndrome - Wikidata - awgldk - TriplyDB

Purkinje cell dysfunction and alteration of long-term synaptic plasticity in fetal alcohol syndrome

Purkinje cell dysfunction and alteration of long-term synaptic plasticity in fetal alcohol syndrome

http://www.wikidata.org/entity/Q35834342

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Phosphodiesterase inhibition increases CREB phosphorylation and restores orientation selectivity in a model of fetal alcohol spectrum disorders BK Channels in the Central Nervous System.BK channels control cerebellar Purkinje and Golgi cell rhythmicity in vivo.What the Spectrum of Microglial Functions Can Teach us About Fetal Alcohol Spectrum Disorder.Low concentrations of alcohol inhibit BDNF-dependent GABAergic plasticity via L-type Ca2+ channel inhibition in developing CA3 hippocampal pyramidal neurons.Commonality in Down and fetal alcohol syndromes.Alcohol excites cerebellar Golgi cells by inhibiting the Na+/K+ ATPase Developmental alterations in olivary climbing fiber distribution following postnatal ethanol exposure in the rat.Construction of vapor chambers used to expose mice to alcohol during the equivalent of all three trimesters of human development.Pregnenolone sulfate increases glutamate release at neonatal climbing fiber-to-Purkinje cell synapses Neurological and neuropsychological effects of low and moderate prenatal alcohol exposure.Pioglitazone blocks ethanol induction of microglial activation and immune responses in the hippocampus, cerebellum, and cerebral cortex in a mouse model of fetal alcohol spectrum disorders.Ethanol alters calcium signaling in axonal growth cones.Fetal alcohol spectrum disorders and neuroimmune changes Fetal alcohol spectrum disorders and abnormal neuronal plasticity.Sensory stimulation-dependent plasticity in the cerebellar cortex of alert mice Does moderate drinking harm the fetal brain? Insights from animal models.Functional Interactions between BKCaα-Subunit and Annexin A5: Implications in Apoptosis.Eyeblink Classical Conditioning in Alcoholism and Fetal Alcohol Spectrum Disorders.AFG3L2 supports mitochondrial protein synthesis and Purkinje cell survival.Further characterization of the effect of ethanol on voltage-gated Ca(2+) channel function in developing CA3 hippocampal pyramidal neurons Neurodevelopmental alcohol exposure elicits long-term changes to gene expression that alter distinct molecular pathways dependent on timing of exposure.Alcohol impairs long-term depression at the cerebellar parallel fiber-Purkinje cell synapse.Administration of memantine during ethanol withdrawal in neonatal rats: effects on long-term ethanol-induced motor incoordination and cerebellar Purkinje cell loss.Neonatal ethanol exposure results in dose-dependent impairments in the acquisition and timing of the conditioned eyeblink response and altered cerebellar interpositus nucleus and hippocampal CA1 unit activity in adult rats.Translational approach to behavioral learning: lessons from cerebellar plasticity.Focus on: neurotransmitter systems.Repeated intermittent alcohol exposure during the third trimester-equivalent increases expression of the GABA(A) receptor δ subunit in cerebellar granule neurons and delays motor development in rats.Oscillations, Timing, Plasticity, and Learning in the Cerebellum.Moderate Prenatal Alcohol Exposure Alters Functional Connectivity in the Adult Rat Brain.Third trimester-equivalent ethanol exposure does not alter complex spikes and climbing fiber long-term depression in cerebellar Purkinje neurons from juvenile rats.Goal-directed arm movements in children with fetal alcohol syndrome: a kinematic approach.Ethanol exposure during development reduces GABAergic/glycinergic neuron numbers and lobule volumes in the mouse cerebellar vermis.Model of very fast (> 75 Hz) network oscillations generated by electrical coupling between the proximal axons of cerebellar Purkinje cells.Plasticity in the cerebellum and primary somatosensory cortex relating to habitual and continuous slender branch climbing in laboratory mice (Mus musculus).Perinatal alcohol exposure in rat induces long-term depression of respiration after episodic hypoxia.Prenatal Ethanol Exposure Persistently Alters Endocannabinoid Signaling and Endocannabinoid-Mediated Excitatory Synaptic Plasticity in Ventral Tegmental Area Dopamine Neurons.Effects of Prenatal Alcohol Exposure (PAE): Insights into FASD using PAE Mouse Models.Increase of KCC2 in hippocampal synaptic plasticity disturbances after perinatal ethanol exposure.Aberrant NMDA-dependent LTD after perinatal ethanol exposure in young adult rat hippocampus.

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Purkinje cell dysfunction and alteration of long-term synaptic plasticity in fetal alcohol syndrome

http://www.wikidata.org/entity/Q35834342

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Purkinje cell dysfunction and ...... city in fetal alcohol syndrome

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Bertrand Bearzatto

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David Gall

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Guy Cheron

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Laurent Servais

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Raphaël Hourez

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Serge N Schiffmann

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P2860

Impaired motor coordination correlates with persistent multiple climbing fiber innervation in PKC gamma mutant mice

Calbindin in cerebellar Purkinje cells is a critical determinant of the precision of motor coordination

Increased ethanol resistance and consumption in Eps8 knockout mice correlates with altered actin dynamics

Neuronal calcium signaling

Bidirectional synaptic plasticity correlated with the magnitude of dendritic calcium transients above a threshold.

Expression of protein kinase C inhibitor blocks cerebellar long-term depression without affecting Purkinje cell excitability in alert mice.

Purkinje cell vulnerability to developmental ethanol exposure in the rat cerebellum.

Bidirectional parallel fiber plasticity in the cerebellum under climbing fiber control.

Purkinje cell degeneration, a new neurological mutation in the mouse.

Molecular regulation of voltage-gated Ca2+ channels.

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