Metabolic stages, mitochondria and calcium in hypoxic/ischemic brain damage.
about
Examination of physiological function and biochemical disorders in a rat model of prolonged asphyxia-induced cardiac arrest followed by cardio pulmonary bypass resuscitationCytoprotective effect of selective small-molecule caspase inhibitors against staurosporine-induced apoptosis.Pigment epithelium-derived factor protects retinal ganglion cells from hypoxia-induced apoptosis by preventing mitochondrial dysfunction.Lycopene protects against hypoxia/reoxygenation-induced apoptosis by preventing mitochondrial dysfunction in primary neonatal mouse cardiomyocytes.Periodic 17β-estradiol pretreatment protects rat brain from cerebral ischemic damage via estrogen receptor-β.Nelfinavir inhibits intra-mitochondrial calcium influx and protects brain against hypoxic-ischemic injury in neonatal miceHypoxia induces autophagic cell death in apoptosis-competent cells through a mechanism involving BNIP3.Mild hypoxemia during initial reperfusion alleviates the severity of secondary energy failure and protects brain in neonatal mice with hypoxic-ischemic injury.The oxygen free radicals originating from mitochondrial complex I contribute to oxidative brain injury following hypoxia-ischemia in neonatal mice.Deregulated Expression of Mitochondrial Proteins Mfn2 and Bcnl3L in Placentae from Sheep Somatic Cell Nuclear Transfer (SCNT) Conceptuses.Optical and pharmacological tools to investigate the role of mitochondria during oxidative stress and neurodegeneration.The mitochondrial permeability transition in neurologic diseaseCalcium-induced precipitate formation in brain mitochondria: composition, calcium capacity, and retentionMechanisms of impaired mitochondrial energy metabolism in acute and chronic neurodegenerative disorders.Preparation of purified perikaryal and synaptosomal mitochondrial fractions from relatively small hypothalamic brain samplesMitochondrial Proteostatic Collapse Leads to Hypoxic Injury.SOD2 in mitochondrial dysfunction and neurodegeneration.Mitochondrial calcium function and dysfunction in the central nervous system.Cell biology of ischemia/reperfusion injury.Mitochondrial dysfunction and nicotinamide dinucleotide catabolism as mechanisms of cell death and promising targets for neuroprotection.Oral contraceptives and nicotine synergistically exacerbate cerebral ischemic injury in the female brain.Mitochondrial dysfunction and NAD(+) metabolism alterations in the pathophysiology of acute brain injury.Periodic Estrogen Receptor-Beta Activation: A Novel Approach to Prevent Ischemic Brain Damage.Ischemia/Reperfusion.Wnt5a Increases the Glycolytic Rate and the Activity of the Pentose Phosphate Pathway in Cortical Neurons.Neuronal expression of the mitochondrial protein prohibitin confers profound neuroprotection in a mouse model of focal cerebral ischemia.ATP-sensitive potassium channel opener iptakalim protected against the cytotoxicity of MPP+ on SH-SY5Y cells by decreasing extracellular glutamate level.Isolation of mitochondria from the CNS.Acute tobacco smoke exposure promotes mitochondrial permeability transition in rat heart.Melatonin augments hypothermic neuroprotection in a perinatal asphyxia model.Age-dependent biphasic changes in ischemic sensitivity in the striatum of Huntington's disease R6/2 transgenic mice.Mitochondrial dynamics: cell-type and hippocampal region specific changes following global cerebral ischemia.Reverse electron transfer results in a loss of flavin from mitochondrial complex I: Potential mechanism for brain ischemia reperfusion injury.Early Effects of Prolonged Cardiac Arrest and Ischemic Postconditioning during Cardiopulmonary Resuscitation on Cardiac and Brain Mitochondrial Function in Pigs.Supra- and sub-baseline phosphocreatine recovery in developing brain after transient hypoxia-ischaemia: relation to baseline energetics, insult severity and outcome.Proteome Analysis of Hypoxic Glioblastoma Cells Reveals Sequential Metabolic Adaptation of One-Carbon Metabolic Pathways.Chronic inflammation and apoptosis propagate in ischemic cerebellum and heart of non-human primates.Synergistic inhibitory effect of nicotine plus oral contraceptive on mitochondrial complex-IV is mediated by estrogen receptor-β in female rats.Animal Toxins as Therapeutic Tools to Treat Neurodegenerative Diseases.Critical Role of Flavin and Glutathione in Complex I-Mediated Bioenergetic Failure in Brain Ischemia/Reperfusion Injury.
P2860
Q28544825-DB48F3EA-5DD3-4380-8502-9F03A4EE29DDQ30436887-F746F7F7-7605-4904-A144-760CAD4488C6Q33912451-6C7A725B-2E86-455A-8D33-E66F6EAB6DFDQ34505904-DFB1112A-8F38-426F-93EE-EB187B387285Q34673933-2225105C-9AA3-4E69-90E4-1DA9FA715F4DQ34685834-1E36F751-3FD4-4AA8-B6FF-11C7C37AF1DAQ35189872-776964DB-8B40-46B1-A6EE-ACD450CA279AQ35738335-D0394E3F-8100-464D-9FF6-281E96A0407DQ35811566-8C1DE3A1-9644-49EA-8B92-AE06C38387EFQ36246279-0EA7EA1A-D37C-48D7-B69D-D49806DBA399Q36571516-0DBC021D-CC2F-41E8-B3D0-41C1AE39DBA9Q36775895-033FA373-D51A-46D5-B7AE-6BA859482145Q36935277-8AF0B39E-E989-4878-A62E-B2A7BBE53812Q36936959-D78025DD-5AA8-4346-95B6-65D9A8BA6620Q36952616-94391357-7B3A-4757-84EE-6B157DBD67CCQ37078598-83F77BAA-4706-4442-A7AD-44041EAC7A78Q37262897-13600FA9-5306-4E43-9B16-9DE6474DE507Q37418288-FF314C91-F93B-4564-87CB-6EA8C4429DBBQ37524901-FF7D3A7F-930B-4DC5-BAFB-6294BD36C416Q37864604-D1494244-20CE-4F05-B61B-810DAB6B57A1Q38169998-6BBD0C2A-AD0E-432E-A879-CE9FFEFCCE1DQ38170006-DD142351-4F9B-47F8-8E10-9396C8FA6731Q38218301-F18A94DB-D87A-4A26-9696-27EBFC09F983Q39109413-98842519-2CA7-454F-AEE5-9BE0B12948B7Q39335491-35256655-AEF2-4ED4-9196-9A521C07C481Q40120456-D9FAECC5-E7F4-47A9-BBC9-3CBE019456DDQ40401416-015E0C1A-F1AD-4A4E-9C62-D77A13B7B143Q41993741-F0618046-C5C9-42E0-99EC-DDBAAE667699Q42496669-64451DFF-8B3A-44A4-A4F9-F240C9740A75Q44099805-E344E87A-9FE9-4F80-9520-83F42018D947Q44405624-4406A537-4E15-43F7-82ED-FF476CFDCB3FQ44460206-565C2FA0-C3D9-4DDC-B3B6-933EE55D06DEQ46306550-E8E6ACD1-EC03-44FB-87CB-625C653B1170Q46383460-1B90CBF4-48FC-45F2-B2C9-AABC2B4CC301Q46453510-DDCC0187-F23A-4875-82C3-3C8E9E9F4C3DQ47852274-85718490-4AD9-48C9-9A29-E50555EFE151Q47904195-C65D7F89-1DFF-4F19-81DD-49A9DF3BBE05Q48711766-5CC7BB45-6AEE-4AC9-AD58-512905C304B5Q51764192-C0321779-63CA-4865-B91D-47B70D114BD4Q52324493-B4294E75-29D9-4E2B-9F8F-C7417B3692E6
P2860
Metabolic stages, mitochondria and calcium in hypoxic/ischemic brain damage.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Metabolic stages, mitochondria and calcium in hypoxic/ischemic brain damage.
@ast
Metabolic stages, mitochondria and calcium in hypoxic/ischemic brain damage.
@en
type
label
Metabolic stages, mitochondria and calcium in hypoxic/ischemic brain damage.
@ast
Metabolic stages, mitochondria and calcium in hypoxic/ischemic brain damage.
@en
prefLabel
Metabolic stages, mitochondria and calcium in hypoxic/ischemic brain damage.
@ast
Metabolic stages, mitochondria and calcium in hypoxic/ischemic brain damage.
@en
P1433
P1476
Metabolic stages, mitochondria and calcium in hypoxic/ischemic brain damage.
@en
P2093
Tibor Kristián
P304
P356
10.1016/J.CECA.2004.02.016
P577
2004-09-01T00:00:00Z