Centrosome abnormalities during a Chlamydia trachomatis infection are caused by dysregulation of the normal duplication pathway.
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Chlamydia Trachomatis Infection-Associated Risk of Cervical Cancer: A Meta-AnalysisHost Organelle Hijackers: a similar modus operandi for Toxoplasma gondii and Chlamydia trachomatis: co-infection model as a tool to investigate pathogenesisThe molecular fingerprint of high grade serous ovarian cancer reflects its fallopian tube originChlamydia trachomatis Genital Infections.A novel co-infection model with Toxoplasma and Chlamydia trachomatis highlights the importance of host cell manipulation for nutrient scavengingSpecific chlamydial inclusion membrane proteins associate with active Src family kinases in microdomains that interact with the host microtubule networkChlamydia induces anchorage independence in 3T3 cells and detrimental cytological defects in an infection model.Activation of epidermal growth factor receptor is required for Chlamydia trachomatis development.Chlamydia trachomatis infection causes mitotic spindle pole defects independently from its effects on centrosome amplificationA clinical overview of centrosome amplification in human cancers.Chlamydia trachomatis inclusions induce asymmetric cleavage furrow formation and ingression failure in host cellsChlamydial infection induces host cytokinesis failure at abscission.Fierce competition between Toxoplasma and Chlamydia for host cell structures in dually infected cells.Chlamydial intracellular survival strategies.Listeria monocytogenes induces host DNA damage and delays the host cell cycle to promote infection.A Chlamydia effector recruits CEP170 to reprogram host microtubule organization.Genital tract infection with Chlamydia trachomatis in women attended at a cervical cancer screening program in Northeastern from Brazil.Recent advances in Chlamydia subversion of host cytoskeletal and membrane trafficking pathways.Species-specific interactions of Src family tyrosine kinases regulate Chlamydia intracellular growth and traffickingShow me your license, please: deregulation of centriole duplication mechanisms that promote amplification.PLK4: a link between centriole biogenesis and cancer.
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Centrosome abnormalities during a Chlamydia trachomatis infection are caused by dysregulation of the normal duplication pathway.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
2009年论文
@zh
2009年论文
@zh-cn
name
Centrosome abnormalities durin ...... he normal duplication pathway.
@ast
Centrosome abnormalities durin ...... he normal duplication pathway.
@en
type
label
Centrosome abnormalities durin ...... he normal duplication pathway.
@ast
Centrosome abnormalities durin ...... he normal duplication pathway.
@en
prefLabel
Centrosome abnormalities durin ...... he normal duplication pathway.
@ast
Centrosome abnormalities durin ...... he normal duplication pathway.
@en
P2093
P2860
P1476
Centrosome abnormalities durin ...... he normal duplication pathway.
@en
P2093
Christine Sütterlin
Kirsten A Johnson
P2860
P304
P356
10.1111/J.1462-5822.2009.01307.X
P577
2009-03-12T00:00:00Z