Inflammatory responses to amyloidosis in a transgenic mouse model of Alzheimer's disease. - Wikidata - awgldk - TriplyDB

Inflammatory responses to amyloidosis in a transgenic mouse model of Alzheimer's disease.

Inflammatory responses to amyloidosis in a transgenic mouse model of Alzheimer's disease.

http://www.wikidata.org/entity/Q35841991

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Prototype Alzheimer's disease epitope vaccine induced strong Th2-type anti-Abeta antibody response with Alum to Quil A adjuvant switch Glycogen synthase kinase-3 (GSK3): inflammation, diseases, and therapeutics Using animal models to determine the significance of complement activation in Alzheimer's disease Novel Abeta peptide immunogens modulate plaque pathology and inflammation in a murine model of Alzheimer's disease.Alzheimer's disease and type 2 diabetes: multiple mechanisms contribute to interactions Retinoic acid inhibits expression of TNF-alpha and iNOS in activated rat microglia Alzheimer disease models and human neuropathology: similarities and differences Inhibition of GSK3β-mediated BACE1 expression reduces Alzheimer-associated phenotypes.A new chiral pyrrolyl α-nitronyl nitroxide radical attenuates β-amyloid deposition and rescues memory deficits in a mouse model of Alzheimer disease Proinflammatory and vasoactive effects of Abeta in the cerebrovasculature.Quantitative MRI reveals aging-associated T2 changes in mouse models of Alzheimer's disease.The complement cascade: Yin-Yang in neuroinflammation--neuro-protection and -degeneration.Amyloid precursor protein secretases as therapeutic targets for traumatic brain injury.Calcineurin triggers reactive/inflammatory processes in astrocytes and is upregulated in aging and Alzheimer's models Prominent neurodegeneration and increased plaque formation in complement-inhibited Alzheimer's mice.Genetic deletion of TNFRII gene enhances the Alzheimer-like pathology in an APP transgenic mouse model via reduction of phosphorylated IκBα.Reimagining Alzheimer's disease--an age-based hypothesis.Mildronate improves cognition and reduces amyloid-β pathology in transgenic Alzheimer's disease mice Contribution of complement activation pathways to neuropathology differs among mouse models of Alzheimer's disease.Long-term treatment of thalidomide ameliorates amyloid-like pathology through inhibition of β-secretase in a mouse model of Alzheimer's disease.Abeta42-driven cerebral amyloidosis in transgenic mice reveals early and robust pathology.Chronic neurodegeneration after traumatic brain injury: Alzheimer disease, chronic traumatic encephalopathy, or persistent neuroinflammation?Dense-core plaques in Tg2576 and PSAPP mouse models of Alzheimer's disease are centered on vessel walls Modulation of nuclear factor-kappa B activity by indomethacin influences A beta levels but not A beta precursor protein metabolism in a model of Alzheimer's disease Fyn knock-down increases Aβ, decreases phospho-tau, and worsens spatial learning in 3×Tg-AD mice The contribution of activated astrocytes to Aβ production: implications for Alzheimer's disease pathogenesis Alzheimer's-type amyloidosis in transgenic mice impairs survival of newborn neurons derived from adult hippocampal neurogenesis.Evidence for early cytotoxic aggregates in transgenic mice for human transthyretin Leu55Pro A coding variant in CR1 interacts with APOE-ε4 to influence cognitive decline.Deletion of tumor necrosis factor death receptor inhibits amyloid beta generation and prevents learning and memory deficits in Alzheimer's mice.Coxibs and Alzheimer's disease: should they stay or should they go?Astrocyte-Microglia Cross Talk through Complement Activation Modulates Amyloid Pathology in Mouse Models of Alzheimer's Disease.Glial cell dysregulation: a new perspective on Alzheimer disease.The role of interleukin-1 in neuroinflammation and Alzheimer disease: an evolving perspective.Interactions between APP secretases and inflammatory mediators.MRI and histological analysis of beta-amyloid plaques in both human Alzheimer's disease and APP/PS1 transgenic mice.Retinoic acid attenuates beta-amyloid deposition and rescues memory deficits in an Alzheimer's disease transgenic mouse model.What does complement do in Alzheimer's disease? Old molecules with new insights.Variations in the transcriptome of Alzheimer's disease reveal molecular networks involved in cardiovascular diseases.Expression of complement system components during aging and amyloid deposition in APP transgenic mice.

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Inflammatory responses to amyloidosis in a transgenic mouse model of Alzheimer's disease.

http://www.wikidata.org/entity/Q35841991

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Inflammatory responses to amyloidosis in a transgenic mouse model of Alzheimer's disease.

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Inflammatory responses to amyloidosis in a transgenic mouse model of Alzheimer's disease.

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Inflammatory responses to amyloidosis in a transgenic mouse model of Alzheimer's disease.

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Inflammatory responses to amyloidosis in a transgenic mouse model of Alzheimer's disease.

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Inflammatory responses to amyloidosis in a transgenic mouse model of Alzheimer's disease.

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Inflammatory responses to amyloidosis in a transgenic mouse model of Alzheimer's disease.

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The American Journal of Pathology

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Inflammatory responses to amyloidosis in a transgenic mouse model of Alzheimer's disease

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A J Tenner

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B Malester

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C A Lemere

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C Zehr

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J A Olschowka

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J LaFrancois

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J M Daeschner

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K Duff

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M Picciano

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Correlative Memory Deficits, Abeta Elevation, and Amyloid Plaques in Transgenic Mice

Cyclooxygenase in biology and disease.

Increased amyloid-beta42(43) in brains of mice expressing mutant presenilin 1

Cyclooxygenase-2: molecular biology, pharmacology, and neurobiology

Cyclooxygenases 1 and 2

Genetic dissection of Alzheimer's disease and related dementias: amyloid and its relationship to tau.

Microglia as mediators of inflammatory and degenerative diseases.

Interleukin-1 and the immunogenetics of Alzheimer disease.

Accelerated Alzheimer-type phenotype in transgenic mice carrying both mutant amyloid precursor protein and presenilin 1 transgenes.

Age-related amyloid beta deposition in transgenic mice overexpressing both Alzheimer mutant presenilin 1 and amyloid beta precursor protein Swedish mutant is not associated with global neuronal loss.

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Alzheimer's disease

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