Arsenic suppresses gene expression in promyelocytic leukemia cells partly through Sp1 oxidation.
about
From an old remedy to a magic bullet: molecular mechanisms underlying the therapeutic effects of arsenic in fighting leukemiaTranscription Regulation of the Human Telomerase Reverse Transcriptase (hTERT) GeneArsenic trioxide downregulates specificity protein (Sp) transcription factors and inhibits bladder cancer cell and tumor growthDownregulation of hTERT: an important As2O3 induced mechanism of apoptosis in myelodysplastic syndromeMultidrug resistance protein 4 (MRP4/ABCC4) regulates cAMP cellular levels and controls human leukemia cell proliferation and differentiation.Insulin regulates hypoxia-inducible factor-1α transcription by reactive oxygen species sensitive activation of Sp1 in 3T3-L1 preadipocyte.The control of reactive oxygen species production by SHP-1 in oligodendrocytesKnowledge building insights on biomarkers of arsenic toxicity to keratinocytes and melanocytesSynergy against PML-RARa: targeting transcription, proteolysis, differentiation, and self-renewal in acute promyelocytic leukemia.Post-translational control of sp-family transcription factors.Structural, chemical and biological aspects of antioxidants for strategies against metal and metalloid exposure.Arsenic trioxide for management of acute promyelocytic leukemia: current evidence on its role in front-line therapy and recurrent disease.Dithiothreitol enhanced arsenic-trioxide-induced cell apoptosis in cultured oral cancer cells via mitochondrial dysfunction and endoplasmic reticulum stress.Arsenic trioxide prevents nitric oxide production in lipopolysaccharide -stimulated RAW 264.7 by inhibiting a TRIF-dependent pathway.SUMO2 and SUMO3 transcription is differentially regulated by oxidative stress in an Sp1-dependent manner.Arsenic trioxide promotes mitochondrial DNA mutation and cell apoptosis in primary APL cells and NB4 cell line.Small-molecule inhibitor of the AP endonuclease 1/REF-1 E3330 inhibits pancreatic cancer cell growth and migration.Oxidative stress modulates PPAR gamma in vascular endothelial cellsOxidative stress and lipid peroxidation products in cancer progression and therapy.Realgar nanoparticles versus ATO arsenic compounds induce in vitro and in vivo activity against multiple myeloma.Histamine H2 Receptor in Blood Cells: A Suitable Target for the Treatment of Acute Myeloid Leukemia.Sumoylation-deficient Prdx6 repairs aberrant Sumoylation-mediated Sp1 dysregulation-dependent Prdx6 repression and cell injury in aging and oxidative stress
P2860
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P2860
Arsenic suppresses gene expression in promyelocytic leukemia cells partly through Sp1 oxidation.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Arsenic suppresses gene expres ...... partly through Sp1 oxidation.
@ast
Arsenic suppresses gene expres ...... partly through Sp1 oxidation.
@en
type
label
Arsenic suppresses gene expres ...... partly through Sp1 oxidation.
@ast
Arsenic suppresses gene expres ...... partly through Sp1 oxidation.
@en
prefLabel
Arsenic suppresses gene expres ...... partly through Sp1 oxidation.
@ast
Arsenic suppresses gene expres ...... partly through Sp1 oxidation.
@en
P2093
P2860
P50
P1433
P1476
Arsenic suppresses gene expres ...... s partly through Sp1 oxidation
@en
P2093
Hsuan-Yu Chen
Linzhao Cheng
Pan-Chyr Yang
P2860
P304
P356
10.1182/BLOOD-2005-01-0241
P407
P577
2005-03-10T00:00:00Z