Cellular factors required for papillomavirus DNA replication.
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Two classes of human papillomavirus type 16 E1 mutants suggest pleiotropic conformational constraints affecting E1 multimerization, E2 interaction, and interaction with cellular proteinsHuman Papillomaviruses; Epithelial Tropisms, and the Development of NeoplasiaInteractions of the papovavirus DNA replication initiator proteins, bovine papillomavirus type 1 E1 and simian virus 40 large T antigen, with human replication protein AThe majority of human replication protein A remains complexed throughout the cell cycle.Recruitment of replication protein A by the papillomavirus E1 protein and modulation by single-stranded DNACellular proteins required for adeno-associated virus DNA replication in the absence of adenovirus coinfection.Cellular topoisomerase I modulates origin binding by bovine papillomavirus type 1 E1.Mismatch Repair proteins are recruited to replicating DNA through interaction with Proliferating Cell Nuclear Antigen (PCNA)Analysis of The Cancer Genome Atlas sequencing data reveals novel properties of the human papillomavirus 16 genome in head and neck squamous cell carcinoma.Competition for DNA binding sites between the short and long forms of E2 dimers underlies repression in bovine papillomavirus type 1 DNA replication control.Characterization of the DNA-binding domain of the bovine papillomavirus replication initiator E1.A C-terminal helicase domain of the human papillomavirus E1 protein binds E2 and the DNA polymerase alpha-primase p68 subunit.The replicator of the Epstein-Barr virus latent cycle origin of DNA replication, oriP, is composed of multiple functional elements.Inhibition of human papilloma virus E2 DNA binding protein by covalently linked polyamides.Genetic variation of human papillomavirus type 16 in individual clinical specimens revealed by deep sequencing.Replication and partitioning of papillomavirus genomes.Inhibition of human papillomavirus DNA replication by an E1-derived p80/UAF1-binding peptide.Evidence for a switch in the mode of human papillomavirus type 16 DNA replication during the viral life cycleTargeting human papillomavirus genome replication for antiviral drug discoveryAn interaction between human papillomavirus 16 E2 and TopBP1 is required for optimum viral DNA replication and episomal genome establishmentDNA Damage Reduces the Quality, but Not the Quantity of Human Papillomavirus 16 E1 and E2 DNA ReplicationThe E1 proteins.Human papillomavirus infection a favorable prognostic factor in laryngeal squamous cell carcinoma is associated with the expression of proliferating cell nuclear antigen.Identification of a short, hydrophilic amino acid sequence critical for origin recognition by the bovine papillomavirus E1 protein.Transcription factor YY1 represses cell-free replication from human papillomavirus origins.Tumor suppressor or oncogene? A critical role of the human papillomavirus (HPV) E2 protein in cervical cancer progression.Initial amplification of the HPV18 genome proceeds via two distinct replication mechanisms.Evidence supporting a role for TopBP1 and Brd4 in the initiation but not continuation of human papillomavirus 16 E1/E2-mediated DNA replication.Functional interaction between the bovine papillomavirus virus type 1 replicative helicase E1 and cyclin E-Cdk2.A fifteen-amino-acid peptide inhibits human papillomavirus E1-E2 interaction and human papillomavirus DNA replication in vitro.Regulation of bovine papillomavirus replicative helicase e1 by the ubiquitin-proteasome pathway.Induction of the bovine papillomavirus origin "onion skin"-type DNA replication at high E1 protein concentrations in vivo.Viral trans-factor independent replication of human papillomavirus genomes.Binding of the E1 and E2 proteins to the origin of replication of bovine papillomavirus.Bovine papillomavirus type 1 DNA replication: the transcriptional activator E2 acts in vitro as a specificity factor.Integration of Human Papillomavirus Genomes in Head and Neck Cancer: Is It Time to Consider a Paradigm Shift?Proteins of the PIAS family enhance the sumoylation of the papillomavirus E1 protein.Why Human Papillomaviruses Activate the DNA Damage Response (DDR) and How Cellular and Viral Replication Persists in the Presence of DDR Signaling.The E8--E2 gene product of human papillomavirus type 16 represses early transcription and replication but is dispensable for viral plasmid persistence in keratinocytes.An oral keratinocyte life cycle model identifies novel host genome regulation by human papillomavirus 16 relevant to HPV positive head and neck cancer.
P2860
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P2860
Cellular factors required for papillomavirus DNA replication.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
Cellular factors required for papillomavirus DNA replication.
@ast
Cellular factors required for papillomavirus DNA replication.
@en
type
label
Cellular factors required for papillomavirus DNA replication.
@ast
Cellular factors required for papillomavirus DNA replication.
@en
prefLabel
Cellular factors required for papillomavirus DNA replication.
@ast
Cellular factors required for papillomavirus DNA replication.
@en
P2093
P2860
P1433
P1476
Cellular factors required for papillomavirus DNA replication
@en
P2093
P2860
P304
P407
P577
1995-12-01T00:00:00Z