A unique pathway of cardiac myocyte death caused by hypoxia-acidosis. - Wikidata - awgldk - TriplyDB

A unique pathway of cardiac myocyte death caused by hypoxia-acidosis.

A unique pathway of cardiac myocyte death caused by hypoxia-acidosis.

http://www.wikidata.org/entity/Q35858005

about

Defining the role of the Bcl-2 family proteins in Huntington's disease Lactic acidosis triggers starvation response with paradoxical induction of TXNIP through MondoA The effect of hypoxia-inducible factor 1-alpha on hypoxia-induced apoptosis in primary neonatal rat ventricular myocytes Upregulation of BNIP3 and translocation to mitochondria mediates cyanide-induced apoptosis in cortical cells Microarray analysis of oxidative stress regulated genes in mesencephalic dopaminergic neuronal cells: relevance to oxidative damage in Parkinson's disease Atmospheric oxygen level affects growth trajectory, cardiopulmonary allometry and metabolic rate in the American alligator (Alligator mississippiensis)Loss of BNIP3 expression is a late event in pancreatic cancer contributing to chemoresistance and worsened prognosis.Hypoxia-inducible factor-1alpha is a critical mediator of hypoxia induced apoptosis in cardiac H9c2 and kidney epithelial HK-2 cells.The genomic analysis of lactic acidosis and acidosis response in human cancers Acidosis and ischemia increase cellular Ca2+ transient alternans and repolarization alternans susceptibility in the intact rat heart.Emerging Metabolic Therapies in Pulmonary Arterial Hypertension.Gene expression and phenotypic characterization of mouse heart after chronic constant or intermittent hypoxia.Does closure of acid-sensing ion channels reduce ischemia/reperfusion injury in the rat brain?p53 directly suppresses BNIP3 expression to protect against hypoxia-induced cell death.SNPs detection in DHPS-WDR83 overlapping genes mapping on porcine chromosome 2 in a QTL region for meat pH.BNIP3 subfamily BH3-only proteins: mitochondrial stress sensors in normal and pathological functions.Acidosis, acid-sensing ion channels, and neuronal cell death.Magnetic resonance imaging detects placental hypoxia and acidosis in mouse models of perturbed pregnancies.Hypoxia and fetal heart development.Heparan sulfate/heparin promotes transthyretin fibrillization through selective binding to a basic motif in the protein.The Mitoscriptome in Aging and Disease.Overexpression of BH3-Only Protein BNIP3 Leads to Enhanced Tumor Growth.Effect of oxygen on cardiac differentiation in mouse iPS cells: role of hypoxia inducible factor-1 and Wnt/beta-catenin signaling Mitophagy and cancer.NF-κB inhibition compromises cardiac fibroblast viability under hypoxia.miR-103 Regulates Oxidative Stress by Targeting the BCL2/Adenovirus E1B 19 kDa Interacting Protein 3 in HUVECs.Differential effect of intrauterine hypoxia on caspase 3 and DNA fragmentation in fetal guinea pig hearts and brains.Inhibition of ischemic cardiomyocyte apoptosis through targeted ablation of Bnip3 restrains postinfarction remodeling in mice.Biochemical dysfunction in heart mitochondria exposed to ischaemia and reperfusion.BNIP3 promotes calcium and calpain-dependent cell death.Perfusion preservation of the donor heart: basic science to pre-clinical.Physiology and pathophysiology of Na+/H+ exchange and Na+ -K+ -2Cl- cotransport in the heart, brain, and blood.BCL2 family of apoptosis-related genes: functions and clinical implications in cancer.Multipotent stromal cells are activated to reduce apoptosis in part by upregulation and secretion of stanniocalcin-1.Bcl-2 family members and apoptosis, taken to heart.Mitochondrial membrane permeabilization and cell death during myocardial infarction: roles of calcium and reactive oxygen species.Dangerous exercise: lessons learned from dysregulated inflammatory responses to physical activity.Bnip3 functions as a mitochondrial sensor of oxidative stress during myocardial ischemia and reperfusion.Protein kinase Cepsilon: the mitochondria-mediated signaling pathway.Metabolism as means for hypoxia adaptation: metabolic profiling and flux balance analysis.

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P2860

A unique pathway of cardiac myocyte death caused by hypoxia-acidosis.

http://www.wikidata.org/entity/Q35858005

description

2004 nî lūn-bûn

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2004年の論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年论文

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2004年论文

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2004年论文

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name

A unique pathway of cardiac myocyte death caused by hypoxia-acidosis.

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A unique pathway of cardiac myocyte death caused by hypoxia-acidosis.

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type

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A unique pathway of cardiac myocyte death caused by hypoxia-acidosis.

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A unique pathway of cardiac myocyte death caused by hypoxia-acidosis.

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A unique pathway of cardiac myocyte death caused by hypoxia-acidosis.

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A unique pathway of cardiac myocyte death caused by hypoxia-acidosis.

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The Journal of Experimental Biology

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A unique pathway of cardiac myocyte death caused by hypoxia-acidosis

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Bernard J Wasserlauf

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Donna P Frazier

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Huifang Li

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John W Thompson

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Keith A Webster

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Maria-Grazia Spiga

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Nanette H Bishopric

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Regina M Graham

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3189-3200

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scholarly article

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10.1242/JEB.01109

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Pt 18

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207

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2004-08-01T00:00:00Z

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8412163

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15299040

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