5-HT2A receptor activation is necessary for CO2-induced arousal.
about
Sudden unexpected death in epilepsy: Identifying risk and preventing mortality2014 Epilepsy Benchmarks Area IV: Limit or Prevent Adverse Consequence of Seizures and Their Treatment Across The LifespanHypercapnia-induced active expiration increases in sleep and enhances ventilation in unanaesthetized rats.Active sleep unmasks apnea and delayed arousal in infant rat pups lacking central serotonin.Furthering our understanding of SUDEP: the role of animal models.Tourette Syndrome as an Independent Risk Factor for Subsequent Sleep Disorders in Children: A Nationwide Population-Based Case-Control Study.Serotonergic agents act on 5-HT3 receptors in the brain to block seizure-induced respiratory arrest in the DBA/1 mouse model of SUDEP.Dorsal raphe serotonin neurons mediate CO2- induced arousal from sleep.Hypoglossal motoneurons are endogenously activated by serotonin during the active period of circadian cycle.Activation of serotonergic neurons in the medullary caudal raphe shortens the laryngeal chemoreflex in anaesthetized neonatal rats.Impaired central respiratory chemoreflex in an experimental genetic model of epilepsy.
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5-HT2A receptor activation is necessary for CO2-induced arousal.
description
2015 nî lūn-bûn
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2015年の論文
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2015年論文
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2015年論文
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2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
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2015年论文
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2015年论文
@zh-cn
name
5-HT2A receptor activation is necessary for CO2-induced arousal.
@ast
5-HT2A receptor activation is necessary for CO2-induced arousal.
@en
type
label
5-HT2A receptor activation is necessary for CO2-induced arousal.
@ast
5-HT2A receptor activation is necessary for CO2-induced arousal.
@en
prefLabel
5-HT2A receptor activation is necessary for CO2-induced arousal.
@ast
5-HT2A receptor activation is necessary for CO2-induced arousal.
@en
P2860
P356
P1476
5-HT2A receptor activation is necessary for CO2-induced arousal
@en
P2093
Amanda MacAskill
Haleigh R Smith
P2860
P304
P356
10.1152/JN.00213.2015
P407
P577
2015-04-29T00:00:00Z