Induction of ZEB proteins by inactivation of RB protein is key determinant of mesenchymal phenotype of breast cancer. - Wikidata - awgldk - TriplyDB

Induction of ZEB proteins by inactivation of RB protein is key determinant of mesenchymal phenotype of breast cancer.

Induction of ZEB proteins by inactivation of RB protein is key determinant of mesenchymal phenotype of breast cancer.

http://www.wikidata.org/entity/Q35868272

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Mouse models for radiation-induced cancers Mouse models for efficacy testing of agents against radiation carcinogenesis—a literature review Rb suppresses collective invasion, circulation and metastasis of breast cancer cells in CD44-dependent manner Targeting the RB-E2F pathway in breast cancer miR-1279, miR-548j, miR-548m, and miR-548d-5p binding sites in CDSs of paralogous and orthologous PTPN12, MSH6, and ZEB1 Genes Multilayer control of the EMT master regulators.Expression of CD24 is associated with HER2 expression and supports HER2-Akt signaling in HER2-positive breast cancer cells.RB loss contributes to aggressive tumor phenotypes in MYC-driven triple negative breast cancer.ZEB1 Promotes Invasion in Human Fetal Neural Stem Cells and Hypoxic Glioma Neurospheres.The Retinoblastoma Tumor Suppressor Transcriptionally Represses Pak1 in Osteoblasts.Regulation of miR106b cluster through the RB pathway: mechanism and functional targets.Retinoblastoma and phosphate and tensin homolog tumor suppressors: impact on ductal carcinoma in situ progression Prognostic Value of EMT-inducing Transcription Factors (EMT-TFs) in Metastatic Breast Cancer: A Systematic Review and Meta-analysis.Direct RNA sequencing mediated identification of mRNA localized in protrusions of human MDA-MB-231 metastatic breast cancer cells KLF5 activates microRNA 200 transcription to maintain epithelial characteristics and prevent induced epithelial-mesenchymal transition in epithelial cells.Dephosphorylation of the Retinoblastoma protein (Rb) inhibits cancer cell EMT via Zeb The ZEB1 transcription factor acts in a negative feedback loop with miR200 downstream of Ras and Rb1 to regulate Bmi1 expression.Progress with palbociclib in breast cancer: latest evidence and clinical considerations.ZEB/miR-200 feedback loop: at the crossroads of signal transduction in cancer.RB1 in cancer: different mechanisms of RB1 inactivation and alterations of pRb pathway in tumorigenesis.Epithelial to mesenchymal transition inducing transcription factors and metastatic cancer.An Extracellular Matrix Molecule, Secreted by the Epithelial-Mesenchymal Transition is Associated With Lymph Node Metastasis of Thyroid Papillary Carcinoma.Concise Review: Stem Cells and Epithelial-Mesenchymal Transition in Cancer: Biological Implications and Therapeutic Targets.β-arrestin-1 mediates nicotine-induced metastasis through E2F1 target genes that modulate epithelial-mesenchymal transition.Therapeutic Targeting of Epithelial Plasticity Programs: Focus on the Epithelial-Mesenchymal Transition.Down-regulation of miR-144 promotes thyroid cancer cell invasion by targeting ZEB1 and ZEB2.THE RETINOBLASTOMA PROTEIN: A MASTER TUMOR SUPPRESSOR ACTS AS A LINK BETWEEN CELL CYCLE AND CELL ADHESION.AKT primes snail-induced EMT concomitantly with the collective migration of squamous cell carcinoma cells.A survey of zeb1, twist and claudin 1 and 4 expression during placental development and disease.Prospects for new lung cancer treatments that target EMT signaling.Retinoblastoma protein determines aggressiveness in triple-negative breast cancer.The zinc finger E-box-binding homeobox 1 (Zeb1) promotes the conversion of mouse fibroblasts into functional neurons.p16INK4A induces senescence and inhibits EMT through microRNA-141/microRNA-146b-5p-dependent repression of AUF1.Different thresholds of ZEB1 are required for Ras-mediated tumour initiation and metastasis.

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Induction of ZEB proteins by inactivation of RB protein is key determinant of mesenchymal phenotype of breast cancer.

http://www.wikidata.org/entity/Q35868272

description

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Induction of ZEB proteins by i ...... al phenotype of breast cancer.

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Induction of ZEB proteins by i ...... al phenotype of breast cancer.

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Induction of ZEB proteins by i ...... al phenotype of breast cancer.

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Induction of ZEB proteins by i ...... al phenotype of breast cancer.

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Induction of ZEB proteins by i ...... al phenotype of breast cancer.

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Induction of ZEB proteins by i ...... al phenotype of breast cancer.

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JBC.M111.313759

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Journal of Biological Chemistry

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Induction of ZEB proteins by i ...... mal phenotype of breast cancer

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Hidemi Hayashi

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Hirokazu Ohata

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Hitoshi Nakagama

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Mari Hosonaga

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Mikako Sasaki

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Shinji Kuninaka

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Takaaki M Goto

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Tatsuhiro Shibata

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Tatsuyuki Chiyoda

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Yoichi Taya

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P2860

The SLUG zinc-finger protein represses E-cadherin in breast cancer

The ZEB/miR-200 feedback loop--a motor of cellular plasticity in development and cancer?

The miR-200 family determines the epithelial phenotype of cancer cells by targeting the E-cadherin repressors ZEB1 and ZEB2

A reciprocal repression between ZEB1 and members of the miR-200 family promotes EMT and invasion in cancer cells

Epithelial-mesenchymal transitions in tumour progression

The miR-200 family and miR-205 regulate epithelial to mesenchymal transition by targeting ZEB1 and SIP1

Twist, a master regulator of morphogenesis, plays an essential role in tumor metastasis

The transcription factor snail is a repressor of E-cadherin gene expression in epithelial tumour cells

DeltaEF1 is a transcriptional repressor of E-cadherin and regulates epithelial plasticity in breast cancer cells

The transcription factor snail controls epithelial-mesenchymal transitions by repressing E-cadherin expression

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