Chronic stress enhances progression of acute lymphoblastic leukemia via β-adrenergic signaling. - Wikidata - awgldk - TriplyDB

Chronic stress enhances progression of acute lymphoblastic leukemia via β-adrenergic signaling.

Chronic stress enhances progression of acute lymphoblastic leukemia via β-adrenergic signaling.

http://www.wikidata.org/entity/Q35877948

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Neural regulation of cancer: from mechanobiology to inflammation Sympathetic nervous system regulation of the tumour microenvironment Neural Regulation of Pancreatic Cancer: A Novel Target for Intervention Chronic stress in mice remodels lymph vasculature to promote tumour cell dissemination.Lymphovascular and neural regulation of metastasis: shared tumour signalling pathways and novel therapeutic approaches.Inflammation: The Common Pathway of Stress-Related Diseases.Chronic stress accelerates pancreatic cancer growth and invasion: a critical role for beta-adrenergic signaling in the pancreatic microenvironment.Chronic stress enhances progression of periodontitis via α1-adrenergic signaling: a potential target for periodontal disease therapy Prolonged restraint stress increases IL-6, reduces IL-10, and causes persistent depressive-like behavior that is reversed by recombinant IL-10.Biobehavioral approaches to cancer progression and survival: Mechanisms and interventions Molecular pathways: beta-adrenergic signaling in cancer Chronic restraint stress decreases the repair potential from mesenchymal stem cells on liver injury by inhibiting TGF-β1 generation.β-Adrenergic receptors suppress Rap1B prenylation and promote the metastatic phenotype in breast cancer cells.β2-adrenoceptor signaling regulates invadopodia formation to enhance tumor cell invasion.Low Socioeconomic Status, Adverse Gene Expression Profiles, and Clinical Outcomes in Hematopoietic Stem Cell Transplant Recipients.The roles of beta-adrenergic receptors in tumorigenesis and the possible use of beta-adrenergic blockers for cancer treatment: possible genetic and cell-signaling mechanisms The β2-adrenoceptor activates a positive cAMP-calcium feedforward loop to drive breast cancer cell invasion.Nervous system regulation of the cancer genome.β-blockers increase response to chemotherapy via direct antitumour and anti-angiogenic mechanisms in neuroblastoma.Psychosocial factors and hematopoietic stem cell transplantation: potential biobehavioral pathways Repurposing Drugs in Oncology (ReDO)-Propranolol as an anti-cancer agent Essential role of IL-10/STAT3 in chronic stress-induced immune suppression.Infantile hemangiomas, retinopathy of prematurity and cancer: a common pathogenetic role of the β-adrenergic system.Drug repurposing in oncology--patient and health systems opportunities.The potential anticancer effect of beta-blockers and the genetic variations involved in the interindividual difference.Epinephrine Stimulates Cell Proliferation and Induces Chemoresistance in Myeloma Cells through the β-Adrenoreceptor in vitro.Cancer cells become less deformable and more invasive with activation of β-adrenergic signaling.Stress, catecholaminergic system and cancer.Proliferation of the human urothelium is induced by atypical β1 -adrenoceptors.Long-term Consequences of the Acute Neural-Inflammatory Stress Response in the Cancer Surgical Patient: New Findings and Perspectives Does the autonomic nervous system contribute to the initiation and progression of prostate cancer?Norepinephrine promotes tumor microenvironment reactivity through β3-adrenoreceptors during melanoma progression Structural Remodeling of Sympathetic Innervation in Atherosclerotic Blood Vessels: Role of Atherosclerotic Disease Progression and Chronic Social Stress.Functional involvement of β3-adrenergic receptors in melanoma growth and vascularization.Chronic stress promotes colitis by disturbing the gut microbiota and triggering immune system response.Role of host β1- and β2-adrenergic receptors in a murine model of B16 melanoma: functional involvement of β3-adrenergic receptors.Repurposing existing medications as cancer therapy: design and feasibility of a randomized pilot investigating propranolol administration in patients receiving hematopoietic cell transplantation.Neuroimmunology of Behavioral Comorbidities Associated With Cancer and Cancer Treatments.

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P2860

Chronic stress enhances progression of acute lymphoblastic leukemia via β-adrenergic signaling.

http://www.wikidata.org/entity/Q35877948

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Chronic stress enhances progre ...... ia via β-adrenergic signaling.

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Chronic stress enhances progre ...... ia via β-adrenergic signaling.

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Chronic stress enhances progre ...... ia via β-adrenergic signaling.

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Brain, Behavior, and Immunity

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Ami J Patel

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P2860

Haematopoietic stem cell release is regulated by circadian oscillations

In vivo imaging of specialized bone marrow endothelial microdomains for tumour engraftment

Parental perceptions of procedure-related distress and family adaptation in childhood leukemia.

Do stress responses promote leukemia progression? An animal study suggesting a role for epinephrine and prostaglandin-E2 through reduced NK activity.

The sympathetic nervous system induces a metastatic switch in primary breast cancer.

Signals from the sympathetic nervous system regulate hematopoietic stem cell egress from bone marrow.

Do stress-related psychosocial factors contribute to cancer incidence and survival?

Autonomic innervation and regulation of the immune system (1987-2007)

β-Adrenergic receptors (β-AR) regulate VEGF and IL-6 production by divergent pathways in high β-AR-expressing breast cancer cell lines

The influence of bio-behavioural factors on tumour biology: pathways and mechanisms.

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635-641

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