Absence of nicotinic acetylcholine receptor α7 subunit amplifies inflammation and accelerates onset of fibrosis: an inflammatory kidney model.
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Endogenous Inhibitors of Kidney Inflammation.Molecular Mechanisms Regulating LPS-Induced Inflammation in the Brain.Macrophage A2A Adenosine Receptors Are Essential to Protect from Progressive Kidney InjuryActivation of the cholinergic anti-inflammatory pathway by GTS-21 attenuates cisplatin-induced acute kidney injury in mice.Zingiberis Siccatum Rhizoma, the active component of the Kampo formula Daikenchuto, induces anti-inflammatory actions through α7 nicotinic acetylcholine receptor activation.
P2860
Absence of nicotinic acetylcholine receptor α7 subunit amplifies inflammation and accelerates onset of fibrosis: an inflammatory kidney model.
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2015 nî lūn-bûn
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2015年の論文
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name
Absence of nicotinic acetylcho ...... an inflammatory kidney model.
@ast
Absence of nicotinic acetylcho ...... an inflammatory kidney model.
@en
type
label
Absence of nicotinic acetylcho ...... an inflammatory kidney model.
@ast
Absence of nicotinic acetylcho ...... an inflammatory kidney model.
@en
prefLabel
Absence of nicotinic acetylcho ...... an inflammatory kidney model.
@ast
Absence of nicotinic acetylcho ...... an inflammatory kidney model.
@en
P2093
P2860
P356
P1433
P1476
Absence of nicotinic acetylcho ...... an inflammatory kidney model.
@en
P2093
Gabriela E Garcia
Jessica Trostel
Luan D Truong
P2860
P304
P356
10.1096/FJ.14-262493
P407
P577
2015-05-18T00:00:00Z