Loss of autophagy in hypothalamic POMC neurons impairs lipolysis. - Wikidata - awgldk - TriplyDB

Loss of autophagy in hypothalamic POMC neurons impairs lipolysis.

Loss of autophagy in hypothalamic POMC neurons impairs lipolysis.

http://www.wikidata.org/entity/Q35880397

about

The Role of Organelle Stresses in Diabetes Mellitus and Obesity: Implication for Treatment Atg7 in development and disease: panacea or Pandora's Box?Autophagy and aging Brain regulation of energy balance and body weight.Macroautophagy and cell responses related to mitochondrial dysfunction, lipid metabolism and unconventional secretion of proteins Nitric Oxide Exerts Basal and Insulin-Dependent Anorexigenic Actions in POMC Hypothalamic Neurons.Neuropeptide Y stimulates autophagy in hypothalamic neurons.Loss of Atg12, but not Atg5, in pro-opiomelanocortin neurons exacerbates diet-induced obesity.Mechanisms linking mtDNA damage and aging.Leptin and insulin pathways in POMC and AgRP neurons that modulate energy balance and glucose homeostasis Autophagy in the CNS and Periphery Coordinate Lipophagy and Lipolysis in the Brown Adipose Tissue and Liver Development of the hypothalamic melanocortin system.Autophagy - An Emerging Anti-Aging Mechanism.Autophagy in Myf5+ progenitors regulates energy and glucose homeostasis through control of brown fat and skeletal muscle development.Knockout of inositol-requiring enzyme 1α in pro-opiomelanocortin neurons decreases fat mass via increasing energy expenditure.Role of autophagy in the control of body metabolism α-MSH and Foxc2 promote fatty acid oxidation through C/EBPβ negative transcription in mice adipose tissue.Identification of autophagy as a longevity-assurance mechanism in the aging model Podospora anserina.Sugar for the brain: the role of glucose in physiological and pathological brain function Metabolic transceivers: in tune with the central melanocortin system.Role of autophagy in the progression from obesity to diabetes and in the control of energy balance.Targeting lipid sensing in the central nervous system: new therapy against the development of obesity and type 2 diabetes.Role of hypothalamic autophagy in the control of whole body energy balance.Autophagy as a crosstalk mediator of metabolic organs in regulation of energy metabolism.Autophagy--a key player in cellular and body metabolism.Autophagy in adipose tissue and the beta cell: implications for obesity and diabetes.Mitochondrial function and mitochondrial DNA maintenance with advancing age.Autophagy and amino acid metabolism in the brain: implications for epilepsy.Hypothalamic-autonomic control of energy homeostasis.Autophagy at the crossroads of catabolism and anabolism.Spatiotemporal regulation of autophagy during Caenorhabditis elegans aging.Emerging roles of autophagy in metabolism and metabolic disorders.Regulation and Functions of Autophagic Lipolysis.The pathophysiology of defective proteostasis in the hypothalamus - from obesity to ageing.Autophagy proteins regulate ERK phosphorylation.POMC Neurons: From Birth to Death.Autophagy in the control of food intake.New Roles of the Primary Cilium in Autophagy.Hypothalamic AMPK-induced autophagy increases food intake by regulating NPY and POMC expression.Current Evidence for a Role of Neuropeptides in the Regulation of Autophagy.

P2860

Q26776377-19EE582A-EDC2-4553-8189-AC547162C572 Q26781433-CFF62B20-32E7-4FF2-86F6-D791C4F3A43F Q26853625-FFBEE9F7-E3EE-49FE-AFB5-3B2B4E9C9BB2 Q26865811-B2B78C60-7987-4438-ADE8-1297CEAAAEEC Q28394794-E78E346D-8DE4-4821-9ED5-D0845FB9565C Q33631400-E5F33524-DD18-4F2B-B9AD-D48C07424860 Q35279368-0F50BE47-B57A-46AD-ACB5-1AE818575497 Q35853081-18F4AB51-C5B2-454F-99A9-A73C83488C90 Q35868104-60099ADB-9F62-44FD-85AE-8A0044934B7D Q36440547-F1979924-7B07-46BA-91A6-3C50693B4147 Q36470253-B2EE09C8-8A62-4B4A-89E9-C5A7DE4D5A7E Q36718574-5A52721E-1F09-4800-9B3A-C25320A68AB7 Q36908386-D8E1F0FA-302B-406F-B12B-DD7A7662E1FB Q37214016-7F33E8BA-5C89-46CE-9885-2C64A7E0763E Q37224690-4CB8C699-07DD-4147-A45D-11BB7F72BBF5 Q37264751-0960E4C6-15F9-49C6-AC71-4E3B08D624FA Q37397352-079AC499-8203-4BF5-8DC8-2A889117D3B3 Q37428793-9CE5F425-A94E-4783-ABF1-1631F49A3F92 Q37511476-486A9C1B-E7B6-4ECB-A912-B309898FF5D7 Q38062727-FBDDA40A-E809-4627-9FEE-B86F26AEB141 Q38078301-FC74E6BE-BF2C-48A3-A0E3-3BDE9AE2FDB5 Q38079005-F21B7CFC-5E03-422D-AC3D-557615B8D66D Q38132721-194759D7-A07A-49D7-BD89-13E7AE86B951 Q38148473-D8389DF0-03EC-42ED-8848-FFC8BEE34841 Q38198952-9B9C3039-9CBF-4DD1-8FC4-25B2297BB8B0 Q38209287-18FDD4FD-96E9-4C0A-9816-96C572427118 Q38228592-D77A4633-8683-4EC7-A7BC-604387D80866 Q38242039-D89E5DFC-556A-437C-8230-C614C17F8A32 Q38531855-15611225-20A4-4942-8653-37FF4A2432B3 Q38547568-A76E7C8B-C374-4F78-B348-4B0886363015 Q38694774-A76A40AC-64BC-4255-8BD3-C2282D3CE31C Q38779345-48E18796-9DEA-4E54-A041-C37AD596354A Q38882789-97D8CB6C-A2A2-429E-8CF0-82DFA41D2480 Q38889413-63CEB995-AA9A-4901-8562-580F71CE7BE3 Q39062345-8C4FA4C5-F1AC-4F7A-AC2D-EC5C69913DD8 Q39133316-0F70F222-033A-4863-BFA5-7308906E2B71 Q39331604-FD8F32DF-CE9B-4062-BFE1-06C5ED6FFB27 Q41621355-36B09D81-F37B-4B15-9B45-DA4CAA4AC2F9 Q41907312-DD14760F-011B-4938-A6D6-7B8BC9E69D3E Q42922923-2DD55DB3-6C36-4CD4-8EB0-0E099FAFD847

P2860

Loss of autophagy in hypothalamic POMC neurons impairs lipolysis.

http://www.wikidata.org/entity/Q35880397

description

2012 nî lūn-bûn

@nan

2012年の論文

@ja

2012年論文

@yue

2012年論文

@zh-hant

2012年論文

@zh-hk

2012年論文

@zh-mo

2012年論文

@zh-tw

2012年论文

@wuu

2012年论文

@zh

2012年论文

@zh-cn

name

Loss of autophagy in hypothalamic POMC neurons impairs lipolysis.

@ast

Loss of autophagy in hypothalamic POMC neurons impairs lipolysis.

@en

type

label

Loss of autophagy in hypothalamic POMC neurons impairs lipolysis.

@ast

Loss of autophagy in hypothalamic POMC neurons impairs lipolysis.

@en

prefLabel

Loss of autophagy in hypothalamic POMC neurons impairs lipolysis.

@ast

Loss of autophagy in hypothalamic POMC neurons impairs lipolysis.

@en

P1433

Q35880397-042A91CF-90A6-4459-8AFA-9E857845AC49

P1476

Q35880397-3BC19476-F8FF-4061-9A36-9EF696B18C6B

P2093

Q35880397-8085C96C-F38D-41D2-A452-5DF499C514F9

Q35880397-81201C89-8402-47FF-9D18-39740EAE2B6E

Q35880397-8CBF3C4B-BFD1-46E2-8FE0-B31001290CBB

Q35880397-90388F67-A640-4CA0-95C5-193AED25EA31

Q35880397-9A2C89DF-1E86-4368-A557-8545F2CB4968

Q35880397-A189B6FB-DF24-481D-909D-E39C1F746C38

Q35880397-BAB9A4B0-CA92-41C1-9F14-F1EF636D5E51

Q35880397-C3DC3E2D-7DE8-4443-8C87-4A1707D3D86F

Q35880397-F484433E-BC47-466D-AFFD-C5B3431D54A2

P2860

Q35880397-0D67FA86-1D1F-4B60-B0B7-3633B04AEFB0

Q35880397-1A7CA3CD-87DC-44EE-A8AC-44E64355CE74

Q35880397-3BC2EFC7-4B72-433F-B1E3-BBCE0E74039D

Q35880397-4851BA8E-534F-4D6D-9990-413AACC63D44

Q35880397-4C5EFA0F-4069-4D15-BF59-F3CE07658286

Q35880397-530B3592-E1F5-4B91-B8D2-CFCBDC59209A

Q35880397-58AB47C6-E3F7-4839-9F92-24403D9DC9E2

Q35880397-7A2B89DE-5306-4600-81B8-6CBFBE131D72

Q35880397-86A0228C-3D93-460A-ACA9-B7FAE63C2B43

Q35880397-94638C17-639D-492F-AFA4-505E229A52B9

P304

Q35880397-D99078BD-47B9-43BC-9434-31EDDA65DE60

P31

Q35880397-B9FC9E0F-D3FF-4219-9391-8776B8AB8A8A

P356

Q35880397-3D725B55-5D1E-4819-AB79-F71533BFED90

P433

Q35880397-A408A666-CAD3-4D8D-81E4-5EF70112EF6D

P478

Q35880397-5A477E81-841E-4396-B409-DF4FBEA4CF6D

P577

Q35880397-CAB579AA-FAE4-4695-BE09-1ECFE1CB7294

P5875

Q35880397-F1A09A46-6539-4E0F-8FF3-11111D9A73CA

P698

Q35880397-381E9B85-840E-4050-A2FD-FE948C64532B

P921

Q35880397-0919C133-8962-4289-919E-7AEC30B2A813

P932

Q35880397-21017BFA-30E4-48A1-9A2A-AE404A4D409D

P356

P1433

P1476

Loss of autophagy in hypothalamic POMC neurons impairs lipolysis.

@en

P2093

Diana Athonvarangkul

http://www.w3.org/2001/XMLSchema#string

Esperanza Arias

http://www.w3.org/2001/XMLSchema#string

Gary J Schwartz

http://www.w3.org/2001/XMLSchema#string

Hyokjoon Kwon

http://www.w3.org/2001/XMLSchema#string

Jeffrey E Pessin

http://www.w3.org/2001/XMLSchema#string

Nuria Martinez Lopez

http://www.w3.org/2001/XMLSchema#string

Rajat Singh

http://www.w3.org/2001/XMLSchema#string

Srabani Sahu

http://www.w3.org/2001/XMLSchema#string

Susmita Kaushik

http://www.w3.org/2001/XMLSchema#string

P2860

A role for NBR1 in autophagosomal degradation of ubiquitinated substrates

Homeostasis model assessment: insulin resistance and ?-cell function from fasting plasma glucose and insulin concentrations in man

Unconventional secretion of Acb1 is mediated by autophagosomes.

Hypothalamic mTOR signaling regulates food intake

SIRT1 deacetylase in POMC neurons is required for homeostatic defenses against diet-induced obesity

Autophagy-based unconventional secretory pathway for extracellular delivery of IL-1β

Unconventional secretion of Pichia pastoris Acb1 is dependent on GRASP protein, peroxisomal functions, and autophagosome formation.

A potential role for hypothalamomedullary POMC projections in leptin-induced suppression of food intake.

Identification of neuronal subpopulations that project from hypothalamus to both liver and adipose tissue polysynaptically.

Weight loss and lipolysis promote a dynamic immune response in murine adipose tissue.

P304

258-265

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1038/EMBOR.2011.260

http://www.w3.org/2001/XMLSchema#string

P433

3

http://www.w3.org/2001/XMLSchema#string

P478

13

http://www.w3.org/2001/XMLSchema#string

P577

2012-03-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

221751313

http://www.w3.org/2001/XMLSchema#string

P698

22249165

http://www.w3.org/2001/XMLSchema#string

P921

P932

3323137

http://www.w3.org/2001/XMLSchema#string