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A new pathway encompassing calpain 3 and its newly identified substrate cardiac ankyrin repeat protein is involved in the regulation of the nuclear factor-κB pathway in skeletal muscleMultiple molecular interactions implicate the connectin/titin N2A region as a modulating scaffold for p94/calpain 3 activity in skeletal muscleDoes skeletal muscle have an 'epi'-memory? The role of epigenetics in nutritional programming, metabolic disease, aging and exerciseNeuromuscular Regeneration: Perspective on the Application of Mesenchymal Stem Cells and Their Secretion ProductsMechanical Signaling in the Pathophysiology of Critical Illness MyopathyRespiratory and limb muscle dysfunction in pulmonary arterial hypertension: a role for exercise training?Pharmacology of manipulating lean body massLeucine and mTORC1: a complex relationshipThe TWEAK-Fn14 pathway: a potent regulator of skeletal muscle biology in health and diseaseThe ER-bound RING finger protein 5 (RNF5/RMA1) causes degenerative myopathy in transgenic mice and is deregulated in inclusion body myositisImpaired skeletal muscle regeneration in the absence of fibrosis during hibernation in 13-lined ground squirrelsThe central role of hypothalamic inflammation in the acute illness response and cachexiaInterplay between aging and unloading on oxidative stress in fast-twitch musclesNrf2 modulates contractile and metabolic properties of skeletal muscle in streptozotocin-induced diabetic atrophyExercise training prevents oxidative stress and ubiquitin-proteasome system overactivity and reverse skeletal muscle atrophy in heart failureA histone deacetylase 4/myogenin positive feedback loop coordinates denervation-dependent gene induction and suppressionA novel glycerophosphodiester phosphodiesterase, GDE5, controls skeletal muscle development via a non-enzymatic mechanismGlucocorticoid-induced leucine zipper (GILZ) and long GILZ inhibit myogenic differentiation and mediate anti-myogenic effects of glucocorticoidsMice lacking COX10 in skeletal muscle recapitulate the phenotype of progressive mitochondrial myopathies associated with cytochrome c oxidase deficiency.Ectopic expression of eIF2Bepsilon in rat skeletal muscle rescues the sepsis-induced reduction in guanine nucleotide exchange activity and protein synthesisREDD1 is a major target of testosterone action in preventing dexamethasone-induced muscle lossRedox regulation of ischemic preconditioning is mediated by the differential activation of caveolins and their association with eNOS and GLUT-4Differential localization of autolyzed calpains 1 and 2 in slow and fast skeletal muscles in the early phase of atrophyRegulatory circuitry of TWEAK-Fn14 system and PGC-1α in skeletal muscle atrophy program.Morphological, physiological and behavioural evaluation of a 'Mice in Space' housing system.Systems analysis of transcriptional data provides insights into muscle's biological response to botulinum toxin.Drosophila eiger mutants are sensitive to extracellular pathogens.Iron accumulation with age, oxidative stress and functional decline.Overexpression of the mitochondrial T3 receptor induces skeletal muscle atrophy during aging.Genomic profiling of messenger RNAs and microRNAs reveals potential mechanisms of TWEAK-induced skeletal muscle wasting in miceRole of the proteasome in excitotoxicity-induced cleavage of glutamic acid decarboxylase in cultured hippocampal neurons.Pyropia yezoensis peptide PYP1‑5 protects against dexamethasone‑induced muscle atrophy through the downregulation of atrogin1/MAFbx and MuRF1 in mouse C2C12 myotubes.The TWEAK-Fn14 dyad is involved in age-associated pathological changes in skeletal muscleThe TWEAK-Fn14 system is a critical regulator of denervation-induced skeletal muscle atrophy in mice.PARK2/Parkin-mediated mitochondrial clearance contributes to proteasome activation during slow-twitch muscle atrophy via NFE2L1 nuclear translocation.The effect of calorie restriction on mouse skeletal muscle is sex, strain and time-dependent.Inhibition of the ubiquitin-proteasome pathway does not protect against ventilator-induced accelerated proteolysis or atrophy in the diaphragmSalmonid genomes have a remarkably expanded akirin family, coexpressed with genes from conserved pathways governing skeletal muscle growth and catabolism.DNA methyltransferase 3a and mitogen-activated protein kinase signaling regulate the expression of fibroblast growth factor-inducible 14 (Fn14) during denervation-induced skeletal muscle atrophy.Kelch-like homologue 9 mutation is associated with an early onset autosomal dominant distal myopathy
P2860
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P2860
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
The molecular basis of skeletal muscle atrophy.
@ast
The molecular basis of skeletal muscle atrophy.
@en
type
label
The molecular basis of skeletal muscle atrophy.
@ast
The molecular basis of skeletal muscle atrophy.
@en
prefLabel
The molecular basis of skeletal muscle atrophy.
@ast
The molecular basis of skeletal muscle atrophy.
@en
P1476
The molecular basis of skeletal muscle atrophy.
@en
P2093
Robert W Jackman
Susan C Kandarian
P304
P356
10.1152/AJPCELL.00579.2003
P577
2004-10-01T00:00:00Z