Alteration in Downstream Hypoxia Gene Signaling in Neonatal Glutathione Peroxidase Overexpressing Mouse Brain after Hypoxia-Ischemia.

about

Erythropoietin Modulates Cerebral and Serum Degradation Products from Excess Calpain Activation following Prenatal Hypoxia-Ischemia.

P2860

Q36520524-60C8E624-A4F1-4AF9-B74C-7BFB68346F0D

P2860

Alteration in Downstream Hypoxia Gene Signaling in Neonatal Glutathione Peroxidase Overexpressing Mouse Brain after Hypoxia-Ischemia.

Item

http://www.wikidata.org/entity/Q35888063

description

2015 nî lūn-bûn

@nan

2015年の論文

@ja

2015年論文

@yue

2015年論文

@zh-hant

2015年論文

@zh-hk

2015年論文

@zh-mo

2015年論文

@zh-tw

2015年论文

@wuu

2015年论文

@zh

2015年论文

@zh-cn

name

Alteration in Downstream Hypox ...... Brain after Hypoxia-Ischemia.

@ast

Alteration in Downstream Hypox ...... Brain after Hypoxia-Ischemia.

@en

type

Item

label

Alteration in Downstream Hypox ...... Brain after Hypoxia-Ischemia.

@ast

Alteration in Downstream Hypox ...... Brain after Hypoxia-Ischemia.

@en

prefLabel

Alteration in Downstream Hypox ...... Brain after Hypoxia-Ischemia.

@ast

Alteration in Downstream Hypox ...... Brain after Hypoxia-Ischemia.

@en

P1476

Alteration in Downstream Hypox ...... Brain after Hypoxia-Ischemia.

@en

P2093

Donna M Ferriero

http://www.w3.org/2001/XMLSchema#string

Matthew Lam

http://www.w3.org/2001/XMLSchema#string

R Ann Sheldon

http://www.w3.org/2001/XMLSchema#string

Raha Sadjadi

http://www.w3.org/2001/XMLSchema#string

Russell Fitzgerald

http://www.w3.org/2001/XMLSchema#string

P2860

Developmental changes in murine brain antioxidant enzymes

Increased infarct size and exacerbated apoptosis in the glutathione peroxidase-1 (Gpx-1) knockout mouse brain in response to ischemia/reperfusion injury

Necrostatin-1 attenuates mitochondrial dysfunction in neurons and astrocytes following neonatal hypoxia-ischemia.

HIF-1 alpha-deficient mice have increased brain injury after neonatal hypoxia-ischemia.

Hypoxic preconditioning protection is eliminated in HIF-1α knockout mice subjected to neonatal hypoxia-ischemia.

Selective vulnerability in the developing central nervous system.

Postnatal Erythropoietin Mitigates Impaired Cerebral Cortical Development Following Subplate Loss from Prenatal Hypoxia-Ischemia.

Perinatal brain injury and regulation of transcription.

mTOR activates hypoxia-inducible factor-1α and inhibits neuronal apoptosis in the developing rat brain during the early phase after hypoxia-ischemia.

Glutathione peroxidase overexpression causes aberrant ERK activation in neonatal mouse cortex after hypoxic preconditioning.

P304

398-406

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1159/000375369

http://www.w3.org/2001/XMLSchema#string

P433

4-5

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2015-03-17T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

273779090

http://www.w3.org/2001/XMLSchema#string

P698

25792071

http://www.w3.org/2001/XMLSchema#string

P921

hypoxia

overexpression

P932

4514555

http://www.w3.org/2001/XMLSchema#string

Alteration in Downstream Hypoxia Gene Signaling in Neonatal Glutathione Peroxidase Overexpressing Mouse Brain after Hypoxia-Ischemia.

about

P2860

P2860

Alteration in Downstream Hypoxia Gene Signaling in Neonatal Glutathione Peroxidase Overexpressing Mouse Brain after Hypoxia-Ischemia.

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P577

P5875

P698

P921

P932

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P577

P5875

P698

P921

P932