Plasma from septic shock patients induces loss of muscle protein. - Wikidata - awgldk - TriplyDB

Plasma from septic shock patients induces loss of muscle protein.

Plasma from septic shock patients induces loss of muscle protein.

http://www.wikidata.org/entity/Q35908213

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Prolonged Glucocorticoid Treatment in ARDS: Impact on Intensive Care Unit-Acquired Weakness Strategies to optimize respiratory muscle function in ICU patients Dexmedetomidine May Produce Extra Protective Effects on Sepsis-induced Diaphragm Injury Hypercapnia attenuates ventilator-induced diaphragm atrophy and modulates dysfunction.The consequences of sudden fluid shifts on body composition in critically ill patients The Sick and the Weak: Neuropathies/Myopathies in the Critically Ill.Muscle dysfunction in patients with lung diseases: a growing epidemic.Preserved skeletal muscle protein anabolic response to acute exercise and protein intake in well-treated rheumatoid arthritis patients Decreased Tissue COX5B Expression and Mitochondrial Dysfunction during Sepsis-Induced Kidney Injury in Rats.STAT3 in the systemic inflammation of cancer cachexia.Diaphragm weakness in mechanically ventilated critically ill patients.Impact of serum biomarkers and clinical factors on intensive care unit mortality and 6-month outcome in relatively healthy patients with severe pneumonia and acute respiratory distress syndrome.Measurement of muscle strength with handheld dynamometer in Intensive Care Unit Novel insights in ICU-acquired respiratory muscle dysfunction: implications for clinical care.Protein kinetics in human endotoxaemia and their temporal relation to metabolic, endocrine and proinflammatory cytokine responses.Levosimendan affects oxidative and inflammatory pathways in the diaphragm of ventilated endotoxemic mice.Deletion of Nlrp3 protects from inflammation-induced skeletal muscle atrophy Direct effects of TNF-α on local fuel metabolism and cytokine levels in the placebo-controlled, bilaterally infused human leg: increased insulin sensitivity, increased net protein breakdown, and increased IL-6 release.Skeletal Muscle and Lymphocyte Mitochondrial Dysfunctions in Septic Shock Trigger ICU-Acquired Weakness and Sepsis-Induced Immunoparalysis.Non-invasive ventilation abolishes the IL-6 response to exercise in muscle-wasted COPD patients: a pilot study.Assessing breathing effort in mechanical ventilation: physiology and clinical implications

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Plasma from septic shock patients induces loss of muscle protein.

http://www.wikidata.org/entity/Q35908213

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

@zh-hk

2011年論文

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2011年論文

@zh-tw

2011年论文

@wuu

2011年论文

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2011年论文

@zh-cn

name

Plasma from septic shock patients induces loss of muscle protein.

@ast

Plasma from septic shock patients induces loss of muscle protein.

@en

type

label

Plasma from septic shock patients induces loss of muscle protein.

@ast

Plasma from septic shock patients induces loss of muscle protein.

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prefLabel

Plasma from septic shock patients induces loss of muscle protein.

@ast

Plasma from septic shock patients induces loss of muscle protein.

@en

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Plasma from septic shock patients induces loss of muscle protein

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Floris Leenders

http://www.w3.org/2001/XMLSchema#string

Hieronymus W H van Hees

http://www.w3.org/2001/XMLSchema#string

Jan Zoll

http://www.w3.org/2001/XMLSchema#string

Johannes G van der Hoeven

http://www.w3.org/2001/XMLSchema#string

Leo M A Heunks

http://www.w3.org/2001/XMLSchema#string

Marianne Linkels

http://www.w3.org/2001/XMLSchema#string

P N Richard Dekhuijzen

http://www.w3.org/2001/XMLSchema#string

Rogier Donders

http://www.w3.org/2001/XMLSchema#string

P2860

The ubiquitin-proteasome proteolytic pathway: destruction for the sake of construction

Identification of ubiquitin ligases required for skeletal muscle atrophy

2001 SCCM/ESICM/ACCP/ATS/SIS International Sepsis Definitions Conference

Muscle wasting and interleukin-6-induced atrogin-I expression in the cachectic Apc ( Min/+ ) mouse

Neuromuscular blockers in early acute respiratory distress syndrome.

C2C12 cells: biophysical, biochemical, and immunocytochemical properties.

Metabolic acidosis stimulates protein degradation in rat muscle by a glucocorticoid-dependent mechanism.

Alterations of protein turnover underlying disuse atrophy in human skeletal muscle.

TNF-alpha acts via p38 MAPK to stimulate expression of the ubiquitin ligase atrogin1/MAFbx in skeletal muscle

Proteasome inhibition improves diaphragm function in congestive heart failure rats.

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10.1186/CC10475

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5

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15

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Willem-Jan M Schellekens

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2011-09-29T00:00:00Z

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51680756

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