Monoclonal antibody-mediated CD200 receptor signaling suppresses macrophage activation and tissue damage in experimental autoimmune uveoretinitis - Wikidata - awgldk - TriplyDB

Monoclonal antibody-mediated CD200 receptor signaling suppresses macrophage activation and tissue damage in experimental autoimmune uveoretinitis

Monoclonal antibody-mediated CD200 receptor signaling suppresses macrophage activation and tissue damage in experimental autoimmune uveoretinitis

http://www.wikidata.org/entity/Q35916057

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Ocular Immune Privilege and Transplantation Myeloid cells expressing VEGF and arginase-1 following uptake of damaged retinal pigment epithelium suggests potential mechanism that drives the onset of choroidal angiogenesis in mice The role of lipoprotein-associated phospholipase A2 in a murine model of experimental autoimmune uveoretinitis Current concepts and future directions in the pathogenesis and treatment of non-infectious intraocular inflammation.CD200R1 agonist attenuates mechanisms of chronic disease in a murine model of multiple sclerosis.The immunosuppressive surface ligand CD200 augments the metastatic capacity of squamous cell carcinoma.Neurobiology of microglial action in CNS injuries: receptor-mediated signaling mechanisms and functional roles Road to fulfilment: taming the immune response to restore vision.Autoimmune and autoinflammatory mechanisms in uveitis.Tumor expression of CD200 inhibits IL-10 production by tumor-associated myeloid cells and prevents tumor immune evasion of CTL therapy.Agonistic CD200R1 DNA Aptamers Are Potent Immunosuppressants That Prolong Allogeneic Skin Graft Survival Mechanisms of immune privilege in the posterior eye.CD200R signaling inhibits pro-angiogenic gene expression by macrophages and suppresses choroidal neovascularization.Leishmania promotes its own virulence by inducing expression of the host immune inhibitory ligand CD200 CD200-CD200R dysfunction exacerbates microglial activation and dopaminergic neurodegeneration in a rat model of Parkinson's disease The classification of microglial activation phenotypes on neurodegeneration and regeneration in Alzheimer's disease brain.Interplay between innate and adaptive immunity in the development of non-infectious uveitis The phenotype of circulating follicular-helper T cells in patients with rheumatoid arthritis defines CD200 as a potential therapeutic target.HMGB1 Facilitated Macrophage Reprogramming towards a Proinflammatory M1-like Phenotype in Experimental Autoimmune Myocarditis Development Role of the retinal vascular endothelial cell in ocular disease Rationale for anti-CD200 immunotherapy in B-CLL and other hematologic malignancies: new concepts in blocking immune suppression.Therapeutic dosing of fingolimod (FTY720) prevents cell infiltration, rapidly suppresses ocular inflammation, and maintains the blood-ocular barrier.Updates on the risk markers and outcomes of severe juvenile idiopathic arthritis-associated uveitis.Understanding the neurobiology of CD200 and the CD200 receptor: a therapeutic target for controlling inflammation in human brains?Decreased expression of CD200 and CD200 receptor in Alzheimer's disease: a potential mechanism leading to chronic inflammation.Programmed cell removal: a new obstacle in the road to developing cancer.Gene therapy for noninfectious uveitis.Tackling the physiological barriers for successful mesenchymal stem cell transplantation into the central nervous system.Addressing the Inflammatory Response to Clinically Relevant Polymers by Manipulating the Host Response Using ITIM Domain-Containing Receptors.Autoimmune uveitis: clinical, pathogenetic, and therapeutic features.Doyne lecture 2016: intraocular health and the many faces of inflammation.Inhibition of CD200R1 expression by C/EBP β in reactive microglial cells.Tumor-derived vaccines containing CD200 inhibit immune activation: implications for immunotherapy.There might be a role for CD200 in the pathogenesis of autoimmune and inflammatory skin disorders.Novel CD200 homologues iSEC1 and iSEC2 are gastrointestinal secretory cell-specific ligands of inhibitory receptor CD200R.Tickling the CD200 receptor: A remedy for those irritating macrophages.Myogenic properties of human mesenchymal stem cells derived from three different sources.Pharmacological Tools to Activate Microglia and their Possible use to Study Neural Network Patho-physiology.TNFR1 signalling is a critical checkpoint for developing macrophages that control of T-cell proliferation.Systemic and local anti-C5 therapy reduces the disease severity in experimental autoimmune uveoretinitis.

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Monoclonal antibody-mediated CD200 receptor signaling suppresses macrophage activation and tissue damage in experimental autoimmune uveoretinitis

http://www.wikidata.org/entity/Q35916057

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Monoclonal antibody-mediated C ...... ental autoimmune uveoretinitis

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Monoclonal antibody-mediated C ...... ental autoimmune uveoretinitis

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Monoclonal antibody-mediated C ...... ental autoimmune uveoretinitis

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The American Journal of Pathology

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Monoclonal antibody-mediated C ...... ental autoimmune uveoretinitis

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Andrew D Dick

http://www.w3.org/2001/XMLSchema#string

Ben J E Raveney

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Claudia J Calder

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David A Copland

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Holly Cherwinski

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Jonathon D Sedgwick

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Joseph Phillips

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Lindsay B Nicholson

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P2860

Alternative activation of macrophages

Lymphoid/neuronal cell surface OX2 glycoprotein recognizes a novel receptor on macrophages implicated in the control of their function

Down-regulation of the macrophage lineage through interaction with OX2 (CD200)

Distribution of OX2 antigen and OX2 receptor within retina.

Interphotoreceptor retinoid binding protein peptide-induced uveitis in B10.RIII mice: characterization of disease parameters and immunomodulation.

CD200 and membrane protein interactions in the control of myeloid cells.

Pattern recognition receptors: doubling up for the innate immune response.

Control of myeloid activity during retinal inflammation.

Enhanced tolerance to autoimmune uveitis in CD200-deficient mice correlates with a pronounced Th2 switch in response to antigen challenge

Regulation of myeloid cell function through the CD200 receptor.

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