Preferential accumulation of amyloid-beta in presynaptic glutamatergic terminals (VGluT1 and VGluT2) in Alzheimer's disease cortex.
about
Cerebrospinal Fluid Particles in Alzheimer Disease and Parkinson DiseaseApoE4 induces Aβ42, tau, and neuronal pathology in the hippocampus of young targeted replacement apoE4 mice.Axonal BACE1 dynamics and targeting in hippocampal neurons: a role for Rab11 GTPaseFlow cytometry analysis of synaptosomes from post-mortem human brain reveals changes specific to Lewy body and Alzheimer's disease.Restored glial glutamate transporter EAAT2 function as a potential therapeutic approach for Alzheimer's diseasePre-synaptic C-terminal truncated tau is released from cortical synapses in Alzheimer's diseaseGlutamate system, amyloid ß peptides and tau protein: functional interrelationships and relevance to Alzheimer disease pathologyBrain regional correlation of amyloid-β with synapses and apolipoprotein E in non-demented individuals: potential mechanisms underlying regional vulnerability to amyloid-β accumulationMeta-analysis of synaptic pathology in Alzheimer's disease reveals selective molecular vesicular machinery vulnerability.A function for EHD family proteins in unidirectional retrograde dendritic transport of BACE1 and Alzheimer's disease Aβ production.Glutamate transporter EAAT2: regulation, function, and potential as a therapeutic target for neurological and psychiatric disease.Proteomic screening of glutamatergic mouse brain synaptosomes isolated by fluorescence activated sortingPreclinical to phase II amyloid beta (Aβ) peptide modulators under investigation for Alzheimer's disease.Pharmacological Rescue of Long-Term Potentiation in Alzheimer Diseased SynapsesInvolvement of the Apoer2 and Lrp1 receptors in mediating the pathological effects of ApoE4 in vivo.Human Brain-Derived Aβ Oligomers Bind to Synapses and Disrupt Synaptic Activity in a Manner That Requires APP.Par3 and aPKC regulate BACE1 endosome-to-TGN trafficking through PACS1.Pathologic Involvement of Glutamatergic Striatal Inputs From the Cortices in TAR DNA-Binding Protein 43 kDa-Related Frontotemporal Lobar Degeneration and Amyotrophic Lateral Sclerosis.Down-regulation of glutamatergic terminals (VGLUT1) driven by Aβ in Alzheimer's disease.Mitochondria in Excitatory and Inhibitory Synapses have Similar Susceptibility to Amyloid-β Peptides Modeling Alzheimer's Disease.Synaptic dysfunction in Alzheimer's disease: the effects of amyloid beta on synaptic vesicle dynamics as a novel target for therapeutic intervention.Sex Differences in Psychiatric Disease: A Focus on the Glutamate System.
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P2860
Preferential accumulation of amyloid-beta in presynaptic glutamatergic terminals (VGluT1 and VGluT2) in Alzheimer's disease cortex.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Preferential accumulation of a ...... in Alzheimer's disease cortex.
@ast
Preferential accumulation of a ...... in Alzheimer's disease cortex.
@en
type
label
Preferential accumulation of a ...... in Alzheimer's disease cortex.
@ast
Preferential accumulation of a ...... in Alzheimer's disease cortex.
@en
prefLabel
Preferential accumulation of a ...... in Alzheimer's disease cortex.
@ast
Preferential accumulation of a ...... in Alzheimer's disease cortex.
@en
P2093
P2860
P1476
Preferential accumulation of a ...... in Alzheimer's disease cortex.
@en
P2093
Carol A Miller
Karabi Nandy
Karen H Gylys
Sanh H Luu
Sophie Sokolow
P2860
P304
P356
10.1016/J.NBD.2011.08.027
P577
2011-09-03T00:00:00Z