Corticotropin-releasing factor receptor-dependent effects of repeated stress on tau phosphorylation, solubility, and aggregation. - Wikidata - awgldk - TriplyDB

Corticotropin-releasing factor receptor-dependent effects of repeated stress on tau phosphorylation, solubility, and aggregation.

Corticotropin-releasing factor receptor-dependent effects of repeated stress on tau phosphorylation, solubility, and aggregation.

http://www.wikidata.org/entity/Q35924838

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G Protein-Coupled Receptors (GPCRs) in Alzheimer's Disease: A Focus on BACE1 Related GPCRs Functional Impact of Corticotropin-Releasing Factor Exposure on Tau Phosphorylation and Axon Transport Posttraumatic stress disorder-like induction elevates β-amyloid levels, which directly activates corticotropin-releasing factor neurons to exacerbate stress responses.Stress-induced glucocorticoid signaling remodels neurovascular coupling through impairment of cerebrovascular inwardly rectifying K+ channel function Corticotropin-releasing factor receptor-1 antagonism mitigates beta amyloid pathology and cognitive and synaptic deficits in a mouse model of Alzheimer's disease.Tau protein is essential for stress-induced brain pathology Corticotropin-releasing factor overexpression gives rise to sex differences in Alzheimer's disease-related signaling.Targeting psychologic stress signaling pathways in Alzheimer's disease.Abnormal dendritic maturation of developing cortical neurons exposed to corticotropin releasing hormone (CRH): Insights into effects of prenatal adversity?Effects of corticotrophin-releasing factor receptor 1 antagonists on amyloid-β and behavior in Tg2576 mice.Increased tau phosphorylation and aggregation in the hippocampus of mice overexpressing corticotropin-releasing factor.Stress during a critical postnatal period induces region-specific structural abnormalities and dysfunction of the prefrontal cortex via CRF1.Impact of CRFR1 Ablation on Amyloid-β Production and Accumulation in a Mouse Model of Alzheimer's Disease.The stress response neuropeptide CRF increases amyloid-β production by regulating γ-secretase activity Corticotropin-Releasing Factor Receptor-1 Antagonism Reduces Oxidative Damage in an Alzheimer’s Disease Transgenic Mouse Model.Synthesis, F-18 radiolabeling, and microPET evaluation of 3-(2,4-dichlorophenyl)-N-alkyl-N-fluoroalkyl-2,5-dimethylpyrazolo[1,5-a]pyrimidin-7-amines as ligands of the corticotropin-releasing factor type-1 (CRF1) receptor.Reproductive Stage and Modulation of Stress-Induced Tau Phosphorylation in Female Rats.Locus Coeruleus, norepinephrine and Aβ peptides in Alzheimer's disease Short-term modern life-like stress exacerbates Aβ-pathology and synapse loss in 3xTg-AD mice Dynamic O-GlcNAcylation and its roles in the cellular stress response and homeostasis.Reconsidering Animal Models of Major Depressive Disorder in the Elderly.Sex-biased cellular signaling: molecular basis for sex differences in neuropsychiatric diseases.Death by a thousand cuts in Alzheimer's disease: hypoxia--the prodrome.Caspase-6 as a novel early target in the treatment of Alzheimer's disease.Neurotoxic saboteurs: straws that break the hippo's (hippocampus) back drive cognitive impairment and Alzheimer's Disease.Trauma exposure and sleep: using a rodent model to understand sleep function in PTSD.Environmental factors in the development and progression of late-onset Alzheimer's disease."Boomerang Neuropathology" of Late-Onset Alzheimer's Disease is Shrouded in Harmful "BDDS": Breathing, Diet, Drinking, and Sleep During Aging.Corticotropin-releasing factor receptor-1 modulates biomarkers of DNA oxidation in Alzheimer's disease mice.The CRF System as a Therapeutic Target for Neuropsychiatric Disorders.Exaggerated phosphorylation of brain tau protein in CRH KO mice exposed to repeated immobilization stress.Effects of chronic noise on the corticotropin-releasing factor system in the rat hippocampus: relevance to Alzheimer's disease-like tau hyperphosphorylation.Stress induced neural reorganization: A conceptual framework linking depression and Alzheimer's disease.Early postnatal stress suppresses the developmental trajectory of hippocampal pyramidal neurons: the role of CRHR1.Corticotrophin releasing factor receptor 1 antagonists prevent chronic stress-induced behavioral changes and synapse loss in aged rats.Blockade of corticotropin-releasing hormone receptor 1 attenuates early-life stress-induced synaptic abnormalities in the neonatal hippocampus.Aging as a Precipitating Factor in Chronic Restraint Stress-Induced Tau Aggregation Pathology, and the Protective Effects of Rosmarinic Acid.Impact of the Relationship of Stress and the Immune System in the Appearance of Alzheimer's Disease.Sex differences in chronic stress responses and Alzheimer's disease.The relationship between stress and Alzheimer's disease.

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Corticotropin-releasing factor receptor-dependent effects of repeated stress on tau phosphorylation, solubility, and aggregation.

http://www.wikidata.org/entity/Q35924838

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2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年學術文章

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2012年學術文章

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2012年學術文章

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Corticotropin-releasing factor ...... , solubility, and aggregation.

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Proceedings of the National Academy of Sciences of the United States of America

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Corticotropin-releasing factor ...... , solubility, and aggregation.

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Allyson Roe Lee

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Kenner C Rice

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Michael A Staup

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Nicholas J Justice

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Paul E Sawchenko

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Robert A Rissman

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P2860

Neuropathological stageing of Alzheimer-related changes

Association of missense and 5'-splice-site mutations in tau with the inherited dementia FTDP-17

Corticotropin releasing factor receptor 1-deficient mice display decreased anxiety, impaired stress response, and aberrant neuroendocrine development

Tau-mediated neurodegeneration in Alzheimer's disease and related disorders

Neurodegeneration and defective neurotransmission in a Caenorhabditis elegans model of tauopathy.

A preparation of Alzheimer paired helical filaments that displays distinct tau proteins by polyacrylamide gel electrophoresis.

Stress-induced tau phosphorylation: functional neuroplasticity or neuronal vulnerability?

The neuroendocrinology of stress and aging: the glucocorticoid cascade hypothesis.

In vivo and in vitro characterization of antalarmin, a nonpeptide corticotropin-releasing hormone (CRH) receptor antagonist: suppression of pituitary ACTH release and peripheral inflammation.

BAC to the future: the use of bac transgenic mice for neuroscience research.

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16

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221983370

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22451915

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phosphorylation

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3341026

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