Exposure to in utero lipopolysaccharide induces inflammation in the fetal ovine skin.
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Intrauterine Candida albicans infection elicits severe inflammation in fetal sheepIntra-amniotic administration of E coli lipopolysaccharides causes sustained inflammation of the fetal skin in sheep.Lipopolysaccharide and soluble CD14 in cord blood plasma are associated with prematurity and chorioamnionitis.Fetal immune response to chorioamnionitis.Ovine fetal thymus response to lipopolysaccharide-induced chorioamnionitis and antenatal corticosteroids.Preterm birth, intrauterine infection, and fetal inflammation.Selective exposure of the fetal lung and skin/amnion (but not gastro-intestinal tract) to LPS elicits acute systemic inflammation in fetal sheep.The diagnostic performance of the Mass Restricted (MR) score in the identification of microbial invasion of the amniotic cavity or intra-amniotic inflammation is not superior to amniotic fluid interleukin-6.Oral, nasal and pharyngeal exposure to lipopolysaccharide causes a fetal inflammatory response in sheep.Pulmonary and systemic inflammatory responses to intra-amniotic IL-1α in fetal sheep.Interleukin-1 in lipopolysaccharide induced chorioamnionitis in the fetal sheep.Inflammation of the fetal ovine skin following in utero exposure to Ureaplasma parvumThe role of granulocyte colony-stimulating factor in the neutrophilia observed in the fetal inflammatory response syndrome.Effects of chorioamnionitis on the fetal lung.Intraamniotic lipopolysaccharide exposure changes cell populations and structure of the ovine fetal thymus.Chorioamnionitis-induced fetal gut injury is mediated by direct gut exposure of inflammatory mediators or by lung inflammation.Pro-inflammatory immune responses in leukocytes of premature infants exposed to maternal chorioamnionitis or funisitis.Sterile and microbial-associated intra-amniotic inflammation in preterm prelabor rupture of membranes.Chorioamnionitis - new ideas from experimental models.Developmental origins of inflammatory and immune diseases.Intra-amniotic administration of lipopolysaccharide induces spontaneous preterm labor and birth in the absence of a body temperature change.The skin microbiome: impact of modern environments on skin ecology, barrier integrity, and systemic immune programming.Age-related changes in expression and function of Toll-like receptors in human skin.Fetal skin as a pro-inflammatory organ: Evidence from a primate model of chorioamnionitis.Polymyxin B agonist capture therapy for intrauterine inflammation: proof-of-principle in a fetal ovine model.
P2860
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P2860
Exposure to in utero lipopolysaccharide induces inflammation in the fetal ovine skin.
description
2010 nî lūn-bûn
@nan
2010年の論文
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2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
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2010年论文
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name
Exposure to in utero lipopolysaccharide induces inflammation in the fetal ovine skin.
@ast
Exposure to in utero lipopolysaccharide induces inflammation in the fetal ovine skin.
@en
type
label
Exposure to in utero lipopolysaccharide induces inflammation in the fetal ovine skin.
@ast
Exposure to in utero lipopolysaccharide induces inflammation in the fetal ovine skin.
@en
prefLabel
Exposure to in utero lipopolysaccharide induces inflammation in the fetal ovine skin.
@ast
Exposure to in utero lipopolysaccharide induces inflammation in the fetal ovine skin.
@en
P2093
P2860
P356
P1476
Exposure to in utero lipopolysaccharide induces inflammation in the fetal ovine skin.
@en
P2093
Alan H Jobe
Ilias Nitsos
Masatoshi Saito
Matthew W Kemp
Suhas G Kallapur
P2860
P356
10.1177/1933719110380470
P577
2010-10-05T00:00:00Z