Myeloid-specific tristetraprolin deficiency in mice results in extreme lipopolysaccharide sensitivity in an otherwise minimal phenotype.
about
Hallmarks of cancer and AU-rich elementsPost-transcriptional Regulation of Immunological Responses through RiboclusteringThe control of inflammation via the phosphorylation and dephosphorylation of tristetraprolin: a tale of two phosphatasesGlobal analysis of posttranscriptional gene expression in response to sodium arseniteTreatment of inflammatory arthritis via targeting of tristetraprolin, a master regulator of pro-inflammatory gene expression.Tristetraprolin expression by keratinocytes controls local and systemic inflammation.Translational regulation of specific mRNAs controls feedback inhibition and survival during macrophage activationTNF and MAP kinase signalling pathways.Myeloid ZFP36L1 does not regulate inflammation or host defense in mouse models of acute bacterial infectionTristetraprolin mediates radiation-induced TNF-α production in lung macrophages.Targeted disruption of MCPIP1/Zc3h12a results in fatal inflammatory disease.Tristetraprolin (TTP) coordinately regulates primary and secondary cellular responses to proinflammatory stimuliDominant Suppression of Inflammation via Targeted Mutation of the mRNA Destabilizing Protein Tristetraprolin.Spontaneous retrotransposon insertion into TNF 3'UTR causes heart valve disease and chronic polyarthritis.Enhanced stability of tristetraprolin mRNA protects mice against immune-mediated inflammatory pathologies.LPS-induced production of TNF-α and IL-6 in mast cells is dependent on p38 but independent of TTPEffects of Combined Tristetraprolin/Tumor Necrosis Factor Receptor Deficiency on the Splenic TranscriptomeTristetraprolin (TTP): interactions with mRNA and proteins, and current thoughts on mechanisms of action.Tristetraprolin regulation of interleukin 23 mRNA stability prevents a spontaneous inflammatory diseaseA systems analysis identifies a feedforward inflammatory circuit leading to lethal influenza infectionTristetraprolin expression and microRNA-mediated regulation during simian immunodeficiency virus infection of the central nervous systemSuppression of IL-12 production by tristetraprolin through blocking NF-kcyB nuclear translocation.Life without TTP: apparent absence of an important anti-inflammatory protein in birdsComparative phosphoproteomics reveals components of host cell invasion and post-transcriptional regulation during Francisella infection.Post-transcriptional coordination of immunological responses by RNA-binding proteins.Post-transcriptional gene regulation by RNA-binding proteins in vascular endothelial dysfunction.Identification of quantitative trait transcripts for growth traits in the large scales of liver and muscle samples.RNA binding proteins as regulators of immune cell biology.Tristetraprolin as a Therapeutic Target in Inflammatory DiseaseRNA-binding proteins in immune regulation: a focus on CCCH zinc finger proteins.An Ancient Family of RNA-Binding Proteins: Still Important!p38 MAPK/PP2Acα/TTP pathway on the connection of TNF-α and caspases activation on hydroquinone-induced apoptosis.Beta Interferon Production Is Regulated by p38 Mitogen-Activated Protein Kinase in Macrophages via both MSK1/2- and Tristetraprolin-Dependent Pathways.The RNA-binding protein tristetraprolin schedules apoptosis of pathogen-engaged neutrophils during bacterial infection.Tristetraprolin binding site atlas in the macrophage transcriptome reveals a switch for inflammation resolutionTristetraprolin inhibits macrophage IL-27-induced activation of antitumour cytotoxic T cell responses.Gain-of-Function Mutation of Tristetraprolin Impairs Negative Feedback Control of Macrophages In Vitro yet Has Overwhelmingly Anti-Inflammatory Consequences In Vivo.Quaking Deficiency Amplifies Inflammation in Experimental Endotoxemia via the Aryl Hydrocarbon Receptor/Signal Transducer and Activator of Transcription 1-NF-κB Pathway.A knock-in tristetraprolin (TTP) zinc finger point mutation in mice: Comparison with complete TTP deficiency.Expression of the mRNA stability regulator Tristetraprolin is required for lactation maintenance in the mouse mammary gland.
P2860
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P2860
Myeloid-specific tristetraprolin deficiency in mice results in extreme lipopolysaccharide sensitivity in an otherwise minimal phenotype.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Myeloid-specific tristetraprol ...... n otherwise minimal phenotype.
@ast
Myeloid-specific tristetraprol ...... n otherwise minimal phenotype.
@en
type
label
Myeloid-specific tristetraprol ...... n otherwise minimal phenotype.
@ast
Myeloid-specific tristetraprol ...... n otherwise minimal phenotype.
@en
prefLabel
Myeloid-specific tristetraprol ...... n otherwise minimal phenotype.
@ast
Myeloid-specific tristetraprol ...... n otherwise minimal phenotype.
@en
P2093
P2860
P356
P1476
Myeloid-specific tristetraprol ...... n otherwise minimal phenotype.
@en
P2093
Deborah J Stumpo
Lian-Qun Qiu
Perry J Blackshear
P2860
P304
P356
10.4049/JIMMUNOL.1103700
P407
P577
2012-04-09T00:00:00Z